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Adam19 encodes a member of the ADAM (a disintegrin and metalloprotease domain) family. Additionally we are shipping ADAM19 Antibodies (65) and ADAM19 Kits (17) and many more products for this protein.
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TGF-beta (show TGFB1 Proteins) stimulation of renal cells results in a significant up-regulation of Adams 10, 17, 12, and 19
Thus, we found that electrical stimulation accelerated NMJ formation, possibly through activation of ADAM19/neuregulin/ErbB (show EGFR Proteins) signaling in NG108-15 cells.
Meltrin beta-mini is a new isoform that induces neuron outgrowth in neuronal cells
Meltrin beta expressed in cardiac neural crest cells is required for ventricular septum formation of the heart.
This study identifies a novel sheddase, ADAM17 (show ADAM17 Proteins), for Kitl1 and Kitl2, and demonstrates that ADAM19 can reduce ADAM17 (show ADAM17 Proteins)-dependent phorbol-ester-stimulated Kitl1 ectodomain shedding.
meltrin-beta functions as a modulator of juxtacrine signaling from axons that activate the Akt (show AKT1 Proteins) pathway and the Krox-20 (show EGR2 Proteins) expression, which is the prerequisite for Schwann cell differentiation
ADAM19 is a protective factor for human prostate cancer. Further, this study suggests that upregulation of ADAM19 expression could be of therapeutic potential in human prostate cancer.
these findings suggested that miR (show MLXIP Proteins)-145 functions as a tumor suppressor in RB by directly targeting ADAM19 in retinoblastoma cells
miR (show MLXIP Proteins)-30c inhibited colon cancer cells via targeting ADAM19.
four suggestive loci (PAX3 (show PAX3 Proteins), CCRN4L (show CCRN4L Proteins), PIGQ (show PIGQ Proteins), and ADAM19)determinants of disease progression in Alzheimer's disease.
ADAM19 rs1422795 and HTR4 (show HTR4 Proteins) rs11168048 are associated with pulmonary function.
MiR (show MLXIP Proteins)-153 has a role in inhibiting migration and invasion of human non-small-cell lung cancer by targeting ADAM19
ADAM19 was upregulated in patients with ulcerative colitis and, to a lesser extent, in patients with Crohn's disease compared with normal controls. In contrast, ADAM9 (show ADAM9 Proteins) and ADAM10 (show ADAM10 Proteins) expression did not differ between patients with IBD and controls.
Prorenin receptor is mainly localized in the subcellular organelles, such as the endoplasmic reticulum and Golgi apparatus, and the prorenin receptor is cleaved by ADAM19 in the Golgi, resulting in two fragments.
Single nucleotide polymorphisms in ADAM19 gene is associated with lymphoma.
These findings suggest that ADAM19 autolysis is activated by lipopolysaccharide and that ADAM19 promotes the secretion of CRIP2 (show CRIP2 Proteins).
A reduction of ADAM19 protein in gastrula stage embryos results in a decrease of Brachyury (show TBX1 Proteins) expression in the notochord concomitant with an increase in the dorsal markers, Goosecoid and Chordin (show CHRD Proteins).
This gene encodes a member of the ADAM (a disintegrin and metalloprotease domain) family. Members of this family are membrane-anchored proteins structurally related to snake venom disintegrins, and have been implicated in a variety of biological processes involving cell-cell and cell-matrix interactions, including fertilization, muscle development, and neurogenesis. This member is a type I transmembrane protein and serves as a marker for dendritic cell differentiation. It has also been demonstrated to be an active metalloproteinase, which may be involved in normal physiological and pathological processes such as cells migration, cell adhesion, cell-cell and cell-matrix interactions, and signal transduction.
, a disintegrin and metalloproteinase domain 19 (meltrin beta)
, disintegrin and metalloproteinase domain-containing protein 19
, meltrin beta
, metalloprotease and disintegrin dendritic antigen marker
, metalloprotease-disintegrin meltrin beta
, ADAM metallopeptidase domain 19 (meltrin beta)
, SRY-box containing gene 30
, a disintegrin and metallopeptidase domain 19 (meltrin beta)