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plays a role in inhibition of nerve growth factor-induced neuronal outgrowth and regulation of neurogenesis [RGD, Feb 2006].. Additionally we are shipping ATF5 Kits (9) and ATF5 Proteins (4) and many more products for this protein.
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Human Polyclonal ATF5 Primary Antibody for EIA, WB - ABIN950552
Wei, Ge, Zhou, Chen, Cui, Liu, Yang, Wu, Gu, Jiang: Identification and characterization of the promoter of human ATF5 gene. in Journal of biochemistry 2010
Show all 5 references for ABIN950552
Cow (Bovine) Polyclonal ATF5 Primary Antibody for WB - ABIN2787491
Zhou, Palam, Jiang, Narasimhan, Staschke, Wek: Phosphorylation of eIF2 directs ATF5 translational control in response to diverse stress conditions. in The Journal of biological chemistry 2008
Show all 2 references for ABIN2787491
Human Polyclonal ATF5 Primary Antibody for WB - ABIN452440
Peters, Liang, Li, Kannan, Peng, Taub, Diamond: ATF-7, a novel bZIP protein, interacts with the PRL-1 protein-tyrosine phosphatase. in The Journal of biological chemistry 2001
Human Polyclonal ATF5 Primary Antibody for ELISA, WB - ABIN1534206
Yamada, Ohira, Horie, Ando, Takayasu, Suzuki, Sugano, Hirata, Goto, Matsunaga, Hiyama, Hayashi, Ando, Suita, Kaneko, Sasaki, Hashizume, Ohnuma, Nakagawara et al.: Expression profiling and differential screening between hepatoblastomas and the corresponding normal livers: identification of high expression of the PLK1 oncogene as a poor-prognostic indicator of ... in Oncogene 2004
This study provides the first evidence that the methylation level of ATF5 decreased, and its mRNA expression was evidently up-regulated in glioma.
These results suggest that the hepatic functions of the human iPS (show SLC27A4 Antibodies)-HLCs (show HLCS Antibodies) could be enhanced by ATF5, c/EBPalpha (show CEBPA Antibodies), and PROX1 (show PROX1 Antibodies) transduction.
Activating transcription factor 5 enhances radioresistance and malignancy in cancer.
Data show that ATF5 is an essential structural protein that is required for the interaction between the mother centriole and the pericentriolar material.
Low expression level of ATF5 in hepatocellular carcinoma indicated aggressive tumor behavior and predicted a worse clinical outcome.
Report a global loss of 5hmC identified three new genes (ECM1 (show ECM1 Antibodies), ATF5, and EOMES (show EOMES Antibodies)) with potential anti-cancer functions that may promote the understanding of the molecular mechanisms of hepatocellular carcinoma development and progression.
the TAK1 (show MAP3K7 Antibodies)-NLK (show NLK Antibodies) pathway is a novel regulator of basal or IL-1beta (show IL1B Antibodies)-triggered C/EBP (show CEBPA Antibodies) activation though stabilization of ATF5
ATF5 promotes the proliferation of HSV-1 via a potential mechanism by which ATF5 enhances the transcription of viral genes during the course of an HSV-1 infection
N-terminal hydrophobic amino acids play an important role in the regulation of ATF5 protein expression in IL-1beta (show IL1B Antibodies)-mediated immune response and that ATF5 is a negative regulator for IL-1beta (show IL1B Antibodies)-induced expression of SAA1 (show SAA1 Antibodies) and SAA2 (show SAA1 Antibodies) in HepG2 cells.
The 5'-untranslated region regulates ATF5 mRNA stability via nonsense-mediated mRNA decay in response to environmental stress.
ATF5 is one of the transcription factors crucial for the vomeronasal sensory formation.
Atf5 is required for mouse olfactory bulb development via interneuron.
Data indicate that downregulation of ATF5 inhibits adipogenesis through C/EBPalpha (show CEBPA Antibodies) by impairing the interaction with p300 (show NOTCH1 Antibodies)-C/EBPbeta (show CEBPB Antibodies).
Both ATF5 and CHOP (show DDIT3 Antibodies) have proapoptotic functions in mouse embryonic fibroblasts.
Adult neurons express ATF5; its levels increase upon endoplasmic reticulum stress as a neuroprotective mechanism.
ATF5 is required for terminal differentiation and survival of olfactory sensory neurons.
BBF2H7 (show CREB3L2 Antibodies)-ATF5-MCL1 (show MCL1 Antibodies) pathway specifically suppressed ER stress-induced apoptosis in chondrocytes.
These findings indicate a reciprocal interaction between ATF5 and Shh (show SHH Antibodies) in which Shh (show SHH Antibodies) stimulates ATF5 expression and in which ATF5 contributes to Shh (show SHH Antibodies)-stimulated cerebellar granule neuron progenitor cell expansion.
Data show that transcription factor ATF5 is expressed in the postnatal brain.
essential in malignant glioma genesis and ATF5-mediated survival pathway identified
plays a role in inhibition of nerve growth factor-induced neuronal outgrowth and regulation of neurogenesis
activating transcription factor 5
, cAMP-dependent transcription factor ATF-5
, cyclic AMP-dependent transcription factor ATF-5
, transcription factor ATFx
, BZIP protein ATF7
, NRIF3-associated protein
, activating transcription factor 5-alpha/beta
, activating transcription factor 7
, activating transcription factor X
, transcription factor-like protein ODA-10