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Adducins are a family of cytoskeleton proteins encoded by three genes (alpha, beta, gamma). Additionally we are shipping alpha Adducin Proteins (4) and alpha Adducin Kits (1) and many more products for this protein.
Showing 10 out of 176 products:
Human Monoclonal alpha Adducin Primary Antibody for IF, IHC - ABIN658927
Schuur, van Swieten, Schol-Gelok, Ikram, Vernooij, Liu, Isaacs, de Boer, de Koning, Niessen, Vrooman, Oostra, van der Lugt, Breteler, van Duijn: Genetic risk factors for cerebral small-vessel disease in hypertensive patients from a genetically isolated population. in Journal of neurology, neurosurgery, and psychiatry 2010
Show all 5 references for ABIN658927
Human Monoclonal alpha Adducin Primary Antibody for IF, IHC - ABIN658928
Bailey, Xie, Do, Montpetit, Diaz, Mohan, Keavney, Yusuf, Gerstein, Engert, Anand: Variation at the NFATC2 locus increases the risk of thiazolidinedione-induced edema in the Diabetes REduction Assessment with ramipril and rosiglitazone Medication (DREAM) study. in Diabetes Care 2010
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Human Monoclonal alpha Adducin Primary Antibody for WB - ABIN393590
Irvin, Lynch, Kabagambe, Tiwari, Barzilay, Eckfeldt, Boerwinkle, Davis, Ford, Arnett: Pharmacogenetic association of hypertension candidate genes with fasting glucose in the GenHAT Study. in Journal of hypertension 2010
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Human Polyclonal alpha Adducin Primary Antibody for IF, IHC (p) - ABIN196918
Pariser, Herradon, Ezquerra, Perez-Pinera, Deuel: Pleiotrophin regulates serine phosphorylation and the cellular distribution of beta-adducin through activation of protein kinase C. in Proceedings of the National Academy of Sciences of the United States of America 2005
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Human Polyclonal alpha Adducin Primary Antibody for IF, IHC (p) - ABIN197406
Barkalow, Italiano, Chou, Matsuoka, Bennett, Hartwig: Alpha-adducin dissociates from F-actin and spectrin during platelet activation. in The Journal of cell biology 2003
Show all 2 references for ABIN197406
This indicates that ADD1 G460W polymorphism could be an important factor in the pathophysiology of tinnitus.
Study shows that ADD1-rs4963 conferred susceptibility to colorectal cancer (CRC (show CALR Antibodies)) suggesting an association between ADD1 and CRC (show CALR Antibodies) risk.
The T allele of ADD1 is associated with essential hypertension in Asians.
study of potential effects of interaction between DNA methylation (show HELLS Antibodies) of ADD1 promoter and ADD1 tagSNPs and environmental factors on essential hypertension (EH); results indicate ADD1 SNP rs4961 has a protective role in development of EH; interactions between alcohol consumption and DNA methylation (show HELLS Antibodies) of ADD1 gene promoter have a significant role in modifying EH susceptibility
There were significant differences between the control group and pediatric hypertensive group in terms of ACE (show ACE Antibodies) I/D (P<0.05) and AGT (show AGXT Antibodies) M235T (P<0.05) polymorphisms, but there were no differences in ADD Gly460Trp (P>0.05) polymorphism.
A significant association was found between ADD1 gene G614T polymorphism and essential hypertension in Chinese patients. Further studies need to be done to confirm these findings in a large sample.
When alpha-adducin complexes with sodium potassium ATPase (show DNAH8 Antibodies) in astrocytes, non-cell autonomous neurodegeneration is triggered.
Phosphorylation of ADD1 at Ser12 and Ser355 by cyclin-dependent kinase 1 (show CDK1 Antibodies) enables ADD1 to bind to myosin-X (Myo10 (show MYO10 Antibodies)).
Fnnctional polymorphism in the phosphorylation site of ADD1 (rs4963) may influence the susceptibility of non-cardia gastric cancer.
concentration of AST was inversely correlated with ADD1 CpG2 (show SYNE1 Antibodies)-5 methylation levels in female controls. These observations may bring new hints to elaborate the pathogenesis of essential hypertension
adducin activity is not essential for actin ring assembly and periodicity but is necessary to control the diameter of both actin rings and axons and actin filament growth within rings.
observations suggest that MALAT1 promotes hepatic steatosis and insulin (show INS Antibodies) resistance by increasing nuclear SREBP-1c (show SREBF1 Antibodies) protein stability.
Advanced glycation end products overproduction in mice liver and skeletal muscle is associated to increased lipogenesis due to the activation of SREBP-1c (show SREBF1 Antibodies).
Srebp-1 (show SREBF1 Antibodies) interacts with c-Myc (show MYC Antibodies), facilitates its binding to downstream pluripotent targets.
SREBP1a or -1c could be acting as transcriptional repressors for rarg2 and when srebf1a expression is down-regulated, its repressing activity disappears.
SREBP-2 (show SREBF2 Antibodies) is critical for survival and limb patterning during development
The two cereal dietary fibers potently decreased protein expressions of sterol regulatory element binding protein-1 (show SREBF1 Antibodies) and key factors involved in lipogenesis
SREBP-1 (show SREBF1 Antibodies) acts as a positive regulator of Sfrp2 (show SFRP2 Antibodies) transcription in chondrogenic cells.
regulatory mechanism by which insulin (show INS Antibodies) inhibits cardiac UCP3 (show UCP3 Antibodies) expression through activation of the lipogenic factor SREBP-1 (show SREBF1 Antibodies).
Taken together, our results suggest that an NCE-box within the clusterin (show CLU Antibodies) promoter is necessary for insulin (show INS Antibodies)-stimulated hepatic expression of clusterin (show CLU Antibodies) via SREBP-1c (show SREBF1 Antibodies).
Adducins are a family of cytoskeleton proteins encoded by three genes (alpha, beta, gamma). Adducin is a heterodimeric protein that consists of related subunits, which are produced from distinct genes but share a similar structure. Alpha- and beta-adducin include a protease-resistant N-terminal region and a protease-sensitive, hydrophilic C-terminal region. Alpha- and gamma-adducins are ubiquitously expressed. In contrast, beta-adducin is expressed at high levels in brain and hematopoietic tissues. Adducin binds with high affinity to Ca(2+)/calmodulin and is a substrate for protein kinases A and C. Alternative splicing results in multiple variants encoding distinct isoforms\; however, not all variants have been fully described.
adducin 1 (alpha)
, adducin 2 (beta)
, erythrocyte adducin alpha subunit
, erythrocyte adducin subunit alpha
, adipocyte determination- and differentiation-dependent factor 1
, sterol regulatory element-binding protein 1