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Adducins are a family of cytoskeleton proteins encoded by three genes (alpha, beta, gamma). Additionally we are shipping alpha Adducin Antibodies (173) and alpha Adducin Proteins (4) and many more products for this protein.
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This indicates that ADD1 G460W polymorphism could be an important factor in the pathophysiology of tinnitus.
Study shows that ADD1-rs4963 conferred susceptibility to colorectal cancer (CRC (show CALR ELISA Kits)) suggesting an association between ADD1 and CRC (show CALR ELISA Kits) risk.
The T allele of ADD1 is associated with essential hypertension in Asians.
study of potential effects of interaction between DNA methylation (show HELLS ELISA Kits) of ADD1 promoter and ADD1 tagSNPs and environmental factors on essential hypertension (EH); results indicate ADD1 SNP rs4961 has a protective role in development of EH; interactions between alcohol consumption and DNA methylation (show HELLS ELISA Kits) of ADD1 gene promoter have a significant role in modifying EH susceptibility
There were significant differences between the control group and pediatric hypertensive group in terms of ACE (show ACE ELISA Kits) I/D (P<0.05) and AGT (show AGXT ELISA Kits) M235T (P<0.05) polymorphisms, but there were no differences in ADD Gly460Trp (P>0.05) polymorphism.
A significant association was found between ADD1 gene G614T polymorphism and essential hypertension in Chinese patients. Further studies need to be done to confirm these findings in a large sample.
When alpha-adducin complexes with sodium potassium ATPase (show DNAH8 ELISA Kits) in astrocytes, non-cell autonomous neurodegeneration is triggered.
Phosphorylation of ADD1 at Ser12 and Ser355 by cyclin-dependent kinase 1 (show CDK1 ELISA Kits) enables ADD1 to bind to myosin-X (Myo10 (show MYO10 ELISA Kits)).
Fnnctional polymorphism in the phosphorylation site of ADD1 (rs4963) may influence the susceptibility of non-cardia gastric cancer.
concentration of AST was inversely correlated with ADD1 CpG2 (show SYNE1 ELISA Kits)-5 methylation levels in female controls. These observations may bring new hints to elaborate the pathogenesis of essential hypertension
The two cereal dietary fibers potently decreased protein expressions of sterol regulatory element binding protein-1 (show SREBF1 ELISA Kits) and key factors involved in lipogenesis
SREBP-1 (show SREBF1 ELISA Kits) acts as a positive regulator of Sfrp2 (show SFRP2 ELISA Kits) transcription in chondrogenic cells.
regulatory mechanism by which insulin (show INS ELISA Kits) inhibits cardiac UCP3 (show UCP3 ELISA Kits) expression through activation of the lipogenic factor SREBP-1 (show SREBF1 ELISA Kits).
Taken together, our results suggest that an NCE-box within the clusterin (show CLU ELISA Kits) promoter is necessary for insulin (show INS ELISA Kits)-stimulated hepatic expression of clusterin (show CLU ELISA Kits) via SREBP-1c (show SREBF1 ELISA Kits).
both Smad3 (show SMAD3 ELISA Kits) and SREBP-1a activation cooperatively regulate TGFbeta (show TGFB1 ELISA Kits) transcriptional responses.
Endoplasmic reticulum stress and SREBP-1 (show SREBF1 ELISA Kits)-dependent effects were induced in glomeruli of angiotensin II-infused mice.
Data show that knock-in (KI) mice in which a human miR (show MLXIP ELISA Kits)-33b is inserted within sterol regulatory element-binding protein 1 (Srebf1 (show SREBF1 ELISA Kits)) intron had reduced HDL (show HSD11B1 ELISA Kits) cholesterol (HDL (show HSD11B1 ELISA Kits)-C).
CREB regulated transcription coactivator 2 (CRTC2 (show CRTC2 ELISA Kits)) functions as a mediator of mTOR (show FRAP1 ELISA Kits) signalling to modulate COPII-dependent SREBP1 (show SREBF1 ELISA Kits) processing
Thyrotropin increases hepatic triglyceride content through upregulation of SREBP-1c (show SREBF1 ELISA Kits) activity.
Hepatic genes expression profiles demonstrated that LBP (show LBP ELISA Kits) can activate the phosphorylation of AMPK (show PRKAA1 ELISA Kits), suppress nuclear expression of SREBP-1c (show SREBF1 ELISA Kits), and decrease protein and mRNA expression of lipogenic genes in vivo or in vitro
Adducins are a family of cytoskeleton proteins encoded by three genes (alpha, beta, gamma). Adducin is a heterodimeric protein that consists of related subunits, which are produced from distinct genes but share a similar structure. Alpha- and beta-adducin include a protease-resistant N-terminal region and a protease-sensitive, hydrophilic C-terminal region. Alpha- and gamma-adducins are ubiquitously expressed. In contrast, beta-adducin is expressed at high levels in brain and hematopoietic tissues. Adducin binds with high affinity to Ca(2+)/calmodulin and is a substrate for protein kinases A and C. Alternative splicing results in multiple variants encoding distinct isoforms\; however, not all variants have been fully described.
adducin 1 (alpha)
, adducin 2 (beta)
, erythrocyte adducin alpha subunit
, erythrocyte adducin subunit alpha
, adipocyte determination- and differentiation-dependent factor 1
, sterol regulatory element-binding protein 1