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APOB product is the main apolipoprotein of chylomicrons and low density lipoproteins. Additionally we are shipping APOB Antibodies (357) and APOB Proteins (26) and many more products for this protein.
Showing 10 out of 119 products:
Mouse (Murine) APOB ELISA Kit for Sandwich ELISA - ABIN1874344
Hollie, Hui: Group 1B phospholipase A? deficiency protects against diet-induced hyperlipidemia in mice. in Journal of lipid research 2011
Show all 4 references for ABIN1874344
Rat (Rattus) APOB ELISA Kit for Sandwich ELISA - ABIN416402
Shulman, Cohen, Soto-Gutierrez, Yagi, Wang, Goldwasser, Lee-Parsons, Benny-Ratsaby, Yarmush, Nahmias: Enhancement of naringenin bioavailability by complexation with hydroxypropyl-?-cyclodextrin. [corrected]. in PLoS ONE 2011
Show all 3 references for ABIN416402
Human APOB ELISA Kit for Sandwich ELISA - ABIN415222
Pramfalk, Larsson, Härdfeldt, Eriksson, Parini: Culturing of HepG2 cells with human serum improve their functionality and suitability in studies of lipid metabolism. in Biochimica et biophysica acta 2015
Multiple novel LDLR (show LDLR ELISA Kits) and ApoB mutations have been identified in a-United Kingdom-based cohort with familial hypercholesterolemia.
Data suggest that LDL-C/apoB ratio (ratio of plasma level of total cholesterol to plasma level of apolipoprotein B) is an important biomarker in overall evaluation of atherogenicity in obese, overweight, and normal-weight men; this study of qualitative changes in lipid/lipoprotein profile for early diagnosis of dyslipidemias leading to atherosclerosis was conducted in Russia.
Mutations in APOB is associated with hypobetalipoproteinemia.
LDLR (show LDLR ELISA Kits) A(+)A(+) genotype, ApoB X(+) allele and ApoE (show APOE ELISA Kits) E4 allele increased the risk of premature coronary artery disease by 1.8, 2.1 and 12.1 respectively.
C7673T polymorphism in apoB gene is significantly associated with increased ischemic stroke risk in the Chinese population.
Apo (show C9orf3 ELISA Kits)-B correlation with both LDL-C and non-HDL (show HSD11B1 ELISA Kits)-C is reduced in individuals with high TG levels and in particular for those with diabetes or MetS.
Report increased intestinal cholesterol absorption and elevated serum cholesterol in families with primary hypercholesterolemia without mutations in APOB.
associations between apolipoprotein B, A1, and the B/A1 ratio and nonalcoholic fatty liver disease in both normal-weight and overweight Korean population
Data indicate that serum apolipoproteins ApoB, ApoA-1 (show APOA1 ELISA Kits) and ApoB/A-1 ratio levels are independently associated with coronary heart disease (CHD (show CHDH ELISA Kits)) risk in Korean men.
Low plasma concentration of PTX3 (show PITX3 ELISA Kits) in early pregnancy is associated with subsequent development of gestational diabetes and with an enhanced risk for CVD as estimated by an elevated apoB/apoA (show APOA ELISA Kits) ratio at 5 years postpartum.
A transposable element insertion in APOB causes cholesterol deficiency in Holstein cattle
Nonesterified fatty acids significantly inhibit the expression of ApoB100, ApoE (show APOE ELISA Kits), MTP (show MTTP ELISA Kits), and LDLR (show LDLR ELISA Kits), thereby decreasing the synthesis and assembly of VLDL and inducing TG accumulation in bovine hepatocytes.
after calving the apolipoprotein B(100) mRNA synthesis was lower, whereas microsomal triglyceride transfer protein (MTP (show MTTP ELISA Kits)) and apolipoprotein E (show APOE ELISA Kits) messenger RNA abundance were higher in the liver.
The effect of hypercholesterolemia induced immune response and inflammation on progression of atherosclerosis in ApoB(tm25gy) LDLr (show LDLR ELISA Kits)(tm1Her) mice, expressing only ApoB100 and deficient in the low density lipoprotein receptor (show LDLR ELISA Kits).
ApoB-containing lipoproteins contribute to augmentation of AngII-induced abdominal aortic aneurysms in male mice.
Immunization with human apolipoprotein B100 (ApoB) resulted in four-fold increased accumulation of effector T cells in ApoB-containing matrigel
PCSK9 (show PCSK9 ELISA Kits) markedly increases intestinal triglyceride-rich apoB production through mechanisms mediated in part by transcriptional effects on apoB.
Mice that produce apoB100 in the RPE and liver secrete lipoproteins into Bruch's membrane, but not to the extent that distinct features of AMD (show AMD1 ELISA Kits) develop
The aim of this study was to characterize the ocular morphology of low-density lipoprotein receptor-deficient apolipoprotein B-100-only mice, with IGF-II overexpression.
Our data establish the role of APOB gene in severe gut (show GUSB ELISA Kits) dysmotility.
Cardiac lipotoxicity may originate from direct inhibition of myocardial ApoB lipoprotein and subsequent decreased lipid export, caused by supraphysiological levels of catecholamines.
ApoB-100 transgenic animals present i) elevated serum and cerebral levels of triglycerides and ApoB-100, ii) increased cerebral tau phosphorylation at phosphosites Ser (show SIGLEC1 ELISA Kits)(199), Ser (show SIGLEC1 ELISA Kits)(199/202), Ser (show SIGLEC1 ELISA Kits)(396) and Ser (show SIGLEC1 ELISA Kits)(404).
model in which increased hepatic sortilin (show SORT1 ELISA Kits) binds intracellular APOB-containing particles in the Golgi apparatus as well as extracellular LDL at the plasma membrane and traffics them to the lysosome for degradation
found association between genotypes for LDLR (show LDLR ELISA Kits) and APOB polymorphisms and serum lipid levels, but none of them seem to be the causal mutation but probably represent closely linked polymorphisms
This gene product is the main apolipoprotein of chylomicrons and low density lipoproteins. It occurs in plasma as two main isoforms, apoB-48 and apoB-100: the former is synthesized exclusively in the gut and the latter in the liver. The intestinal and the hepatic forms of apoB are encoded by a single gene from a single, very long mRNA. The two isoforms share a common N-terminal sequence. The shorter apoB-48 protein is produced after RNA editing of the apoB-100 transcript at residue 2180 (CAA->UAA), resulting in the creation of a stop codon, and early translation termination. Mutations in this gene or its regulatory region cause hypobetalipoproteinemia, normotriglyceridemic hypobetalipoproteinemia, and hypercholesterolemia due to ligand-defective apoB, diseases affecting plasma cholesterol and apoB levels.
, apolipoprotein B (including Ag(x) antigen)
, apolipoprotein B-100
, apolipoprotein B48
, mutant Apo B 100
, apolipoprotein B PI
, apolipoprotein B-48
, apolipoprotein B