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Apolipoprotein H has been implicated in a variety of physiologic pathways including lipoprotein metabolism, coagulation, and the production of antiphospholipid autoantibodies. Additionally we are shipping APOH Antibodies (145) and APOH Proteins (34) and many more products for this protein.
Showing 10 out of 68 products:
Human APOH ELISA Kit for Sandwich ELISA - ABIN414966
Lackner, Beck, Eichmeir, Gemeiner, Hummel, Pullirsch, Razzazi-Fazeli, Seifner, Miller: Contamination of therapeutic human immunoglobulin preparations with apolipoprotein H (?2-glycoprotein I). in Electrophoresis 2014
TLR2 (show TLR2 ELISA Kits) increase the stimulating effect of beta2GP1/anti-beta2GP1 complex on the expression of TNF-alpha (show TNF ELISA Kits) in mouse peritoneal macrophages
The Fifth Domain of Beta 2 Glycoprotein I Protects from Natural IgM Mediated Cardiac Ischaemia Reperfusion Injury.
These results indicate that both NF-kappaB (show NFKB1 ELISA Kits) and c-Jun/AP-1 (show JUN ELISA Kits) are involved in regulating anti-beta2GPI-induced expression of prothrombotic and proinflammatory molecules in vivo.
Phospholipid scramblase 1 regulates phosphatidylserine exposure in response to oxygen stress, leading to beta2-glycoprotein I and IgM binding and lipid-mediated, inflammatory responses.
beta2-GPI-D-I-tDCs may offer a novel approach for developing therapy for APS (show SH2B2 ELISA Kits) patients
Deletion of beta2 GPI accelerates and potentiates the autoimmune phenotype in male BXSB.Yaa mice
Interaction of beta2-glycoprotein I with lipopolysaccharide leads to Toll-like receptor 4 (TLR4 (show TLR4 ELISA Kits))-dependent activation of macrophages
Data are consistent with pathogenic mechanisms in antiphospholipid syndrome involving both neutralization of beta(2)-glycoprotein I (beta(2)GPI) function and beta(2)GPI-immunoglobulin complex formation.
beta-2-glycoprotein 1 plays an important physiologic role in the recognition of dying cells
certain subpopulations of antibodies raised against tetanus toxoid and cross-reactive with beta(2)GPI, because of the molecular mimicry mechanism, could have pathologic potential
Prevalence and pathogenicity of IgA aB2GPI antibodies may vary from a population of ESRD patients to another.
The association of IL28B (show IL28B ELISA Kits) and APOH single nucleotide polymorphisms (SNPs) with sustained virological response and of ITPA (show ITPA ELISA Kits) SNPs with anemia related phenotypes.
We identified APOH as an obesity-resistance gene that interacts with FTO (show FTO ELISA Kits) rs9939609 to more than double the occurrence of thinness.
Trp316Ser variant (rs1801690) near the apolipoprotein H (beta2-glycoprotein-I) gene was associated with serum lipid parameters in Chinese volunteers. Suggest that there may be have racial/ethnic-and/or gender-specificity.
Plasma apolipoprotein H limits HCV replication and associates with response to NS3 protease inhibitor-based therapy.
The renoprotective and antifibrosis effects of beta2GPI and reduced beta2GPI on diabetic nephropathy were closely associated with suppressing the activation of the TGF-beta1 (show TGFB1 ELISA Kits)-p38 MAPK (show MAPK14 ELISA Kits) pathway.
Apo H is up-regulated in aspirin-tolerant asthma (ATA) compared to aspirin-exacerbated respiratory disease (AERD) and normal controls, suggesting that Apo H may be involved in different pathogenesis of ATA from AERD
Apolipoprotein H expression is associated with IL28B (show IL28B ELISA Kits) Single Nucleotide Polymorphism and viral clearance in hepatitis C virus infection.
The interaction between beta2GPI and TLR4 (show TLR4 ELISA Kits) is confirmed by the reduction of anti-beta2GPI antibody binding and by the up-regulation of E-selectin (show SELE ELISA Kits) or ICAM-1 (show ICAM1 ELISA Kits) by TLR4 (show TLR4 ELISA Kits) silencing in HUVEC.
APOH is a new candidate gene associated with thrombosis
Apolipoprotein H has been implicated in a variety of physiologic pathways including lipoprotein metabolism, coagulation, and the production of antiphospholipid autoantibodies. APOH may be a required cofactor for anionic phospholipid binding by the antiphospholipid autoantibodies found in sera of many patients with lupus and primary antiphospholipid syndrome, but it does not seem to be required for the reactivity of antiphospholipid autoantibodies associated with infections.
, apolipoprotein H (beta-2-glycoprotein I)
, APC inhibitor
, activated protein C-binding protein
, beta-2-glycoprotein 1
, beta-2-glycoprotein I
, anticardiolipin cofactor