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CD180 is a cell surface molecule consisting of extracellular leucine-rich repeats (LRR) and a short cytoplasmic tail. Additionally we are shipping CD180 Antibodies (134) and CD180 Proteins (7) and many more products for this protein.
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The findings of association in different populations with different SNPs support a conclusion that variation close to the ANKRA2 (show ANKRA2 ELISA Kits)/CD180 locus analysed in this study contributes to differential response to pathogen exposure.
The role of TLR2 (show TLR2 ELISA Kits), TLR4 (show TLR4 ELISA Kits) and RP105/MD1 (show LY86 ELISA Kits) in the immunoregulatory effect of acidic exopolysaccharides from Lactobacillus plantarum N14 (show CLPTM1 ELISA Kits), is reported.
These data unveil a novel innate immune signaling axis that orchestrates key cytokine responses of macrophages and provide molecular insight into the functions of RP105 (show GPR83 ELISA Kits) as an innate immune receptor for mycobacteria.
RP105 (show GPR83 ELISA Kits) regulates monocyte-driven arteriogenesis in a murine hind limb ischemia model.
RP105 deficiency results in reduced early atherosclerotic plaque development with a marked decrease in lesional macrophage content, which may be due to disturbed migration of RP105 deficient monocytes resulting from CCR2 downregulation
Deficiency of the endogenous TLR4 (show TLR4 ELISA Kits) inhibitor, RP105 (show GPR83 ELISA Kits), leads to pronounced cardiac dilation after myocardial infarction.
Data indicate that RP105 (show GPR83 ELISA Kits) and TLR4 (show TLR4 ELISA Kits) are both expressed by vascular smooth muscle cells (VSMC).
RP105 (show GPR83 ELISA Kits) deficiency on circulating cells results in an intriguing unexpected TLR-associated mechanisms that decrease atherosclerotic lesion formation with alterations on proinflammatory B2 B cells.
The function of TLR2 (show TLR2 ELISA Kits) and TLR4 (show TLR4 ELISA Kits) in response to TLR ligands could be associated with each other by RP105 (show GPR83 ELISA Kits).
Data show that both RP105 (show GPR83 ELISA Kits)/MD-1 (show LY86 ELISA Kits) and TLR4 (show TLR4 ELISA Kits)/MD-2 (show LY96 ELISA Kits) are expressed in marginal zone (MZ) B cells.
The RP105 (show GPR83 ELISA Kits)/MD-1 (show LY86 ELISA Kits) complex is a major mediator of adipose tissue inflammation independent of TLR4 (show TLR4 ELISA Kits) signaling and may represent a novel therapeutic target for obesity-associated metabolic disorders.
Modulation of B cell proliferation by RP105 (show GPR83 ELISA Kits) is not a function of B cell-intrinsic expression of RP105 (show GPR83 ELISA Kits).
By associating with PIM (show PIM1 ELISA Kits)-1L, CD180 can thus obtain autonomous signaling capabilities, and this complex is then channeling inflammatory signals into B cell survival programs
Since MEC1 (show ATR ELISA Kits) cells are derived from a CLL patient with mutated IGVH genes (M-CLL) negative correlation between CD180 and CD32 (show FCGR2B ELISA Kits) expression on cycling MEC1 (show ATR ELISA Kits) cells could be limited to M-CLL.
we address of monocytes functional status through assessment of the patterns of expression of Fcgamma receptors CD64 (show FCGR1A ELISA Kits), CD32 (show FCGR2B ELISA Kits), CD16 (show CD16 ELISA Kits) and CD180 receptor on monocytes from CLL patients and healthy individuals using specific mAbs and flow cytometry.
Data indicate that TLR9 (show TLR9 ELISA Kits)-signaling as a crucial factor for turning retinoic acid (RA) into a strong stimulator of RP105-mediated B-cell proliferation.
IL-4 (show IL4 ELISA Kits) failed to up-regulate expression of RP105 at the cell surface. In conclusion, the anti-inflammatory actions of IL-4 (show IL4 ELISA Kits) occur independently of IL-10 (show IL10 ELISA Kits), RP105, and the kinase activity of RIPK2 (show RIPK2 ELISA Kits)
Both mouse and human RP105/MD-1 (show LY86 ELISA Kits) exhibit dimerization of the 1:1 RP105/MD-1 (show LY86 ELISA Kits) complex, demonstrating a novel organization.
Lower mRNA expression of LY64 was detected in gingival tissue of chronic periodontitis patients compared to healthy controls.
Data show that CD180, CD284 (show TLR4 ELISA Kits) and CD14 (show NDUFA2 ELISA Kits) expression is higher on normal B cells than on CD19 (show CD19 ELISA Kits)+ B-cell chronic lymphocytic leukaemia cells.
RP105 cross-linkaage enhanced B-lymphocyte (show AKAP17A ELISA Kits) proliferation, TLR9 (show TLR9 ELISA Kits) expression, and growth.
RP105 regulated TLR4 (show TLR4 ELISA Kits) signaling in dendritic cells
CD180 is a cell surface molecule consisting of extracellular leucine-rich repeats (LRR) and a short cytoplasmic tail. The extracellular LRR is associated with a molecule called MD-1 and form the cell surface receptor complex, RP105/MD-1. It belongs to the family of pathogen receptors, Toll-like receptors (TLR). RP105/MD1, by working in concert with TLR4, controls B cell recognition and signaling of lipopolysaccharide (LPS), a membrane constituent of Gram-negative bacteria.
, lymphocyte antigen 78
, CD180 molecule
, radio protective 105
, lymphocyte antigen 64-like protein
, radioprotective 105 kDa protein
, lymphocyte antigen 64
, lymphocyte antigen-64, radioprotective, 105kDa