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CD200R1 encodes a receptor for the OX-2 membrane glycoprotein. Additionally we are shipping CD200R1 Antibodies (285) and CD200R1 Proteins (21) and many more products for this protein.
Showing 3 out of 9 products:
Human CD200R1 ELISA Kit for Sandwich ELISA - ABIN1117913
Holmannova, Kolackova, Mandak, Kunes, Holubcova, Krejsek, Vlaskova, Andrys: Inhibitory CD200R and proapoptotic CD95/CD95L molecules on innate immunity cells are modulated by cardiac surgery. in Perfusion 2014
CD200R signaling inhibits pro-angiogenic gene expression by macrophages and suppresses choroidal neovascularization.
Data show that neutrophil infiltration of tissues was markedly decreased in CD200 antigen (show CD200 ELISA Kits) (tg) transgenic animals and increased in CD200 receptor 1 CD200R1(-/-) knockout mice.
we show a causal role for the CD200 (show CD200 ELISA Kits)-CD200R inhibitory axis in arteriogenesis in a murine hindlimb ischemia model.
CD200 (show CD200 ELISA Kits)-CD200R1 interaction in glial cells is involved in the neuroprotective action of PPAR-gamma (show PPARG ELISA Kits) agonists.
[review] Influenza-infected CD200R1-deficient mice show less bacterial load and exhibit decreased pathogenesis and mortality than wild-type animals following streptococcal pneumonia superinfection.
Anti-CD200R2 (show CD200R1L ELISA Kits), anti-IL-9 (show IL9 ELISA Kits), anti-IL-35, or anti-TGF-beta (show TGFB1 ELISA Kits) abolishes increased graft survival and Treg induction induced in cromolyn-treated CD200R1KO.CD200tg mice, and suggest combined role of mast cells and Tregs.
AML1 (show RUNX1 ELISA Kits)-ETO (show RUNX1T1 ELISA Kits) upregulated CD200R1 in lineage(-) cells, which was characteristic of AML1 (show RUNX1 ELISA Kits)-ETO (show RUNX1T1 ELISA Kits)-positive leukemia in mice.
a role for both graft and host CD200 (show CD200 ELISA Kits)/CD200R expression in increased allograft survival.
The main sites of CD200/CD200 (show CD200 ELISA Kits) receptor (CD200R) interaction in neonates and blood vessels, until adulthood, highlight the possible role of the CD200 (show CD200 ELISA Kits)/CD200R system in microglial development and renewal.
These data demonstrate an unanticipated and novel requirement for CD200R1 in "licensing" pro-inflammatory functions of TLR2 (show TLR2 ELISA Kits) and in limiting viral replication that are supported by ex vivo and in vivo evidence.
Constitutively expressed Siglec-9 (show SIGLEC9 ELISA Kits) inhibits LPS (show IRF6 ELISA Kits)-induced CCR7 (show CCR7 ELISA Kits), but enhances IL-4 (show IL4 ELISA Kits)-induced CD200R expression in human macrophages.
Inflammatory Bowel Disease patients showed a significant decrease in the percentage of plasmacytoid dendritic cells and myeloid dendritic cells expressing CD200R1 compared to that of the control group.
CD200 (show CD200 ELISA Kits)-CD200R1 signaling may be required for human pregnancy success.
This is the first report of CD200R1 expression by human epithelial tumor cells, and specifically, early-stage human breast cancer cells. It is also the first report of CD200R1 expression by term placental villous trophoblasts.
data show that the expression of CD200R, CD95 (show FAS ELISA Kits) and CD95L (show FASL ELISA Kits) was influenced by cardiac surgery and imply the role of these membrane molecules in cell regulation-inhibition and apoptosis following cardiac surgery.
The authors showed that the viral orthologues e127 from rat cytomegalovirus and K14 (show KRT14 ELISA Kits) from human herpesvirus 8 do not bind the activating CD200R-like proteins from their respective species, although they do bind the inhibitory receptors.
The proportion of CD200R1+ cells in PBMCs, peripheral CD14 (show NDUFA2 ELISA Kits)+ cells and CD4 (show CD4 ELISA Kits)+ T cells was significantly lower in rheumatoid arthritis patients compared to controls.
We demonstrate a significant correlation between CD200R1 positive cells and disease severity in rheumatoid arthritis (RA) patients, thus indicating the relevance of the CD200 (show CD200 ELISA Kits)/CD200R1 signaling pathway potential involvement in the pathogenesis of RA.
The mechanism underlying ESA (show FLOT2 ELISA Kits) might be associated with enhanced expression of CD200 (show CD200 ELISA Kits) and CD200R in the trophoblast, leading to an upregulation of the immune response during the first trimester of pregnancy.
The expression of CD200R as well as B7-H4 (show VTCN1 ELISA Kits) co-stimulatory molecules on CD14 (show NDUFA2 ELISA Kits)(+) cells were significantly higher in cord blood when compared with peripheral blood.
This gene encodes a receptor for the OX-2 membrane glycoprotein. Both the receptor and substrate are cell surface glycoproteins containing two immunoglobulin-like domains. This receptor is restricted to the surfaces of myeloid lineage cells and the receptor-substrate interaction may function as a myeloid downregulatory signal. Mouse studies of a related gene suggest that this interaction may control myeloid function in a tissue-specific manner. Alternative splicing of this gene results in multiple transcript variants.
CD200 receptor 1
, CD200 receptor 1-like
, CD200 cell surface glycoprotein receptor
, Orexin receptor 2
, antigen identified by monoclonal antibody MRC OX-2 receptor
, cell surface glycoprotein CD200 receptor 1
, cell surface glycoprotein OX2 receptor 1
, MOX2 receptor
, cell surface glycoprotein receptor CD200
, OX102 antigen
, OX2 receptor