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Strongly activates transcription when bound to HCFC1. Additionally we are shipping CREB/ATF BZIP Transcription Factor Proteins (3) and many more products for this protein.
Showing 10 out of 51 products:
Cow (Bovine) Polyclonal CREBZF Primary Antibody for WB - ABIN2774980
Misra, Rapin, Akhova, Bainbridge, Korchinski: Zhangfei is a potent and specific inhibitor of the host cell factor-binding transcription factor Luman. in The Journal of biological chemistry 2005
Human Polyclonal CREBZF Primary Antibody for EIA, WB - ABIN783399
Hogan, Cockram, Lu: Cooperative interaction of Zhangfei and ATF4 in transactivation of the cyclic AMP response element. in FEBS letters 2006
Zhangfei/CREBZF stabilizes p53 (show TP53 Antibodies) and co-localizes with it in cellular nuclei; the bLZip domain of Zhangfei is required for its profound effects on cell growth and interaction with p53 (show TP53 Antibodies).
Zhangfei had a suppressive effect on most unfolded protein response genes.
Results indicate that Ursodeoxycholic acid (UDCA) activates SMILE gene expression, which leads to repression of liver X receptor alpha (LXRalpha (show NR1H3 Antibodies))-mediated hepatic lipogenic enzyme gene expression.
Our objective was to determine the molecular mechanisms by which Zhangfei influences medulloblastoma cell lines
The transcription factor CREBZF is a novel positive regulator of p53 (show TP53 Antibodies).
CREBZF, a novel Smad8 (show SMAD9 Antibodies)-binding protein.
a microarray study of SMILE knock-down and PMA activation in HeLa cells was compared to earlier analysis based on microarray data of kidney allograft tolerance and rejection to determine possible new genes and gene networks involved in kidney transplantation
the curcumin/LKB1 (show STK11 Antibodies)/AMPK (show PRKAA1 Antibodies)/SMILE/PGC1alpha pathway differentially regulates ER stress-mediated gene transcription
SMILE is involved in the endoplasmic reticulum stress response, by modulating proteasome activity and XBP-1 (show XBP1 Antibodies) transcript expression. This function of SMILE may influence immune cell behavior in the context of transplantation.
These results indicate that SMILE isoforms regulate the inhibition of estrogen receptor (show ESR1 Antibodies) transactivation by SHP (show LAMC1 Antibodies) in a cell-type-specific manner and act as a novel transcriptional co-regulator in ER signalling.
SMILE is an insulin (show INS Antibodies)-inducible corepressor that suppresses hepatic gluconeogenesis.
Small heterodimer partner (show NR0B2 Antibodies)-interacting leucine zipper protein (show ROGDI Antibodies) inhibits adipogenesis by regulating peroxisome proliferator-activated receptor gamma (show PPARG Antibodies) activity.
Taken together, these findings demonstrate that SMILE negatively regulates osteocalcin (show BGLAP Antibodies) via a direct interaction with RUNX2 (show RUNX2 Antibodies)
CREBZF is involved in implantation and is up-regulated by oestrogen.
Smile is a novel gene specifying both bronchial smooth muscle and lung alveolar myofibroblast lineages, contributing to our understanding of the biological control of the development of these cells.
Strongly activates transcription when bound to HCFC1. Suppresses the expression of HSV proteins in cells infected with the virus in a HCFC1-dependent manner. Also suppresses the HCFC1- dependent transcriptional activation by CREB3 and reduces the amount of CREB3 in the cell. Able to down-regulate expression of some cellular genes in CREBZF-expressing cells (By similarity).
CREB/ATF bZIP transcription factor
, HCF-binding transcription factor Zhangfei
, SHP-interacting leucine zipper protein
, host cell factor-binding transcription factor Zhangfei
, tyrosine kinase-associated leucine zipper protein LAZip
, tyrosine kinase-associated leucine zipper protein LAZipII