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Strongly activates transcription when bound to HCFC1. Additionally we are shipping CREB/ATF BZIP Transcription Factor Antibodies (53) and many more products for this protein.
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Zhangfei/CREBZF stabilizes p53 (show TP53 Proteins) and co-localizes with it in cellular nuclei; the bLZip domain of Zhangfei is required for its profound effects on cell growth and interaction with p53 (show TP53 Proteins).
Zhangfei had a suppressive effect on most unfolded protein response genes.
Results indicate that Ursodeoxycholic acid (UDCA) activates SMILE gene expression, which leads to repression of liver X receptor alpha (LXRalpha (show NR1H3 Proteins))-mediated hepatic lipogenic enzyme gene expression.
Our objective was to determine the molecular mechanisms by which Zhangfei influences medulloblastoma cell lines
The transcription factor CREBZF is a novel positive regulator of p53 (show TP53 Proteins).
CREBZF, a novel Smad8 (show SMAD9 Proteins)-binding protein.
a microarray study of SMILE knock-down and PMA activation in HeLa cells was compared to earlier analysis based on microarray data of kidney allograft tolerance and rejection to determine possible new genes and gene networks involved in kidney transplantation
the curcumin/LKB1 (show STK11 Proteins)/AMPK (show PRKAA1 Proteins)/SMILE/PGC1alpha pathway differentially regulates ER stress-mediated gene transcription
SMILE is involved in the endoplasmic reticulum stress response, by modulating proteasome activity and XBP-1 (show XBP1 Proteins) transcript expression. This function of SMILE may influence immune cell behavior in the context of transplantation.
These results indicate that SMILE isoforms regulate the inhibition of estrogen receptor (show ESR1 Proteins) transactivation by SHP (show LAMC1 Proteins) in a cell-type-specific manner and act as a novel transcriptional co-regulator in ER signalling.
SMILE is an insulin (show INS Proteins)-inducible corepressor that suppresses hepatic gluconeogenesis.
Small heterodimer partner (show NR0B2 Proteins)-interacting leucine zipper protein (show ROGDI Proteins) inhibits adipogenesis by regulating peroxisome proliferator-activated receptor gamma (show PPARG Proteins) activity.
Taken together, these findings demonstrate that SMILE negatively regulates osteocalcin (show BGLAP Proteins) via a direct interaction with RUNX2 (show RUNX2 Proteins)
CREBZF is involved in implantation and is up-regulated by oestrogen.
Smile is a novel gene specifying both bronchial smooth muscle and lung alveolar myofibroblast lineages, contributing to our understanding of the biological control of the development of these cells.
Strongly activates transcription when bound to HCFC1. Suppresses the expression of HSV proteins in cells infected with the virus in a HCFC1-dependent manner. Also suppresses the HCFC1- dependent transcriptional activation by CREB3 and reduces the amount of CREB3 in the cell. Able to down-regulate expression of some cellular genes in CREBZF-expressing cells (By similarity).
CREB/ATF bZIP transcription factor
, HCF-binding transcription factor Zhangfei
, SHP-interacting leucine zipper protein
, host cell factor-binding transcription factor Zhangfei
, tyrosine kinase-associated leucine zipper protein LAZip
, tyrosine kinase-associated leucine zipper protein LAZipII