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The protein encoded by CTNNBIP1 binds CTNNB1 and prevents interaction between CTNNB1 and TCF family members. Additionally we are shipping CTNNBIP1 Antibodies (47) and CTNNBIP1 Proteins (15) and many more products for this protein.
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miR (show MLXIP ELISA Kits)-215-5p is a negative regulator of adipocyte differentiation through post-transcriptional regulation of FNDC3B (show FNDC3B ELISA Kits) and CTNNBIP1 during early adipogenesis
miR (show MLXIP ELISA Kits)-29b plays a pivotal role in fetal mouse neurogenesis by regulating ICAT-mediated Wnt (show WNT2 ELISA Kits)/beta-catenin (show CTNNB1 ELISA Kits) signaling.
Mechanistic studies revealed that CTNNBIP1 suppresses Wnt (show WNT2 ELISA Kits)/beta-catenin (show CTNNB1 ELISA Kits) signaling and that miR (show MLXIP ELISA Kits)-215 promotes beta-catenin (show CTNNB1 ELISA Kits) activation and upregulates alpha-SMA (show SMN1 ELISA Kits) and fibronectin (show FN1 ELISA Kits) expression in TGF-beta1 (show TGFB1 ELISA Kits)-treated MMCs by targeting CTNNBIP1
Overexpression of Icat induces G(2) arrest and cell death in tumor cell mutants for adenomatous polyposis coli (show APC ELISA Kits), beta-catenin (show CTNNB1 ELISA Kits), or Axin (show AXIN1 ELISA Kits).
ICAT plays an important role in the anteriorization of neural cells by inhibiting the posteriorizing activity of Wnt (show WNT2 ELISA Kits) signaling
These results suggest that the loss of ICAT gene function causes the arrest of UB branching and the apoptotic death of MM cells, resulting in renal agenesis.
Inhibition of beta-catenin (show CTNNB1 ELISA Kits) signaling in articular chondrocytes causes increased cell apoptosis and articular cartilage destruction in Col2a1 (show COL2A1 ELISA Kits)-ICAT- transgenic mice.
Overexpression of ICAT promoted Caski cells' proliferation, arrested the cell cycle in the S phase and enhanced cell migration. Conclusion Overexpression of ICAT can promote the proliferation and migration of Caski cervical cancer cells.
Somatic mutation of beta-catenin (CTNNB1 (show CTNNB1 ELISA Kits)) is known to be crucial for Wilms tumor development in up to 15% of cases.
CTNNBIP1 expression correlated with longer overall survival in LAC (show LCT ELISA Kits) patients. This study reveals that miR (show MLXIP ELISA Kits)-214 plays a critical role in CSLC self-renewal and stemness by targeting CTNNBIP1.
Data show that microRNA miR (show MLXIP ELISA Kits)-215 activates beta-catenin (show CTNNB1 ELISA Kits) pathways by decreasing catenin beta interacting protein 1 (CTNNBIP1)expression in gliomas.
Simultaneous silencing of beta-catenin (show CTNNB1 ELISA Kits) and STAT3 (show STAT3 ELISA Kits) synergistically induces apoptosis and inhibits cell proliferation in HepG2 liver cancer cells.
Finally, pro-incubation with idebenone inhibited mitochondrial dysfunction induced by oxLDL through the mitochondrial-dependent apoptotic pathway and GSK3beta (show GSK3b ELISA Kits)/beta-catenin (show CTNNB1 ELISA Kits) signalling pathways.
A potent beta-catenin (show CTNNB1 ELISA Kits) inhibitor, ICAT/CTNNBIP1 was a direct target of miR (show MLXIP ELISA Kits)-424-5p.
Particulate matter (PM10) downregulates E-Cadherin (show CDH1 ELISA Kits)/beta-Catenin (show CTNNB1 ELISA Kits) expression.
A statistically significant lapatinib- and gefitinib-induced repression of cyclin D1 (show CCND1 ELISA Kits), MMP9 (show MMP9 ELISA Kits) and beta-catenin (show CTNNB1 ELISA Kits) in CERV196 cells.
Nuclear YAP (show YAP1 ELISA Kits) and cytoplasmic beta-catenin (show CTNNB1 ELISA Kits) play important roles in carcinogenesis.
The protein encoded by this gene binds CTNNB1 and prevents interaction between CTNNB1 and TCF family members. The encoded protein is a negative regulator of the Wnt signaling pathway. Two transcript variants encoding the same protein have been found for this gene.
, beta-catenin-interacting protein 1
, catenin, beta interacting protein 1 a
, catenin beta interacting protein 1 b
, catenin, beta interacting protein 1
, inhibitor of beta-catenin and Tcf-4
, beta-catenin-interacting protein ICAT