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The protein encoded by CCR7 is a member of the G protein-coupled receptor family. Additionally we are shipping CCR7 Kits (25) and CCR7 Proteins (5) and many more products for this protein.
Showing 10 out of 276 products:
Human Monoclonal CCR7 Primary Antibody for FACS, ICC - ABIN4899110
Lee, Fertig, Jin, Sukumar, Pandey, Popel: Breast cancer cells condition lymphatic endothelial cells within pre-metastatic niches to promote metastasis. in Nature communications 2014
Show all 46 references for ABIN4899110
Human Monoclonal CCR7 Primary Antibody for FACS - ABIN4895618
Kaur, Sanford, Garry, Lang, Klumpp, Watanabe, Bronson, Lifson, Rosati, Pavlakis, Felber, Knipe, Desrosiers: Ability of herpes simplex virus vectors to boost immune responses to DNA vectors and to protect against challenge by simian immunodeficiency virus. in Virology 2006
Show all 34 references for ABIN4895618
Human Monoclonal CCR7 Primary Antibody for FACS - ABIN4895613
Santra, Tomaras, Warrier, Nicely, Liao, Pollara, Liu, Alam, Zhang, Cocklin, Shen, Duffy, Xia, Schutte, Pemble Iv, Dennison, Li, Chao, Vidnovic, Evans, Klein, Kumar, Robinson, Landucci, Forthal et al.: Human Non-neutralizing HIV-1 Envelope Monoclonal Antibodies Limit the Number of Founder Viruses during SHIV Mucosal Infection in Rhesus Macaques. ... in PLoS pathogens 2015
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Human Monoclonal CCR7 Primary Antibody for FACS - ABIN4895609
Iyer, Gangadhara, Victor, Gomez, Basu, Hong, Labranche, Montefiori, Villinger, Moss, Amara: Codelivery of Envelope Protein in Alum with MVA Vaccine Induces CXCR3-Biased CXCR5+ and CXCR5- CD4 T Cell Responses in Rhesus Macaques. in Journal of immunology (Baltimore, Md. : 1950) 2015
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Human Monoclonal CCR7 Primary Antibody for FACS - ABIN4895616
Li, Richert-Spuhler, Evans, Gillis, Connole, Estes, Keele, Klatt, Reeves: Hypercytotoxicity and rapid loss of NKp44+ innate lymphoid cells during acute SIV infection. in PLoS pathogens 2014
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Human Monoclonal CCR7 Primary Antibody for FACS - ABIN1105995
Zanin-Zhorov, Hershkoviz, Hecht, Cahalon, Lider: Fibronectin-associated Fas ligand rapidly induces opposing and time-dependent effects on the activation and apoptosis of T cells. in Journal of immunology (Baltimore, Md. : 1950) 2003
Human Polyclonal CCR7 Primary Antibody for FACS, IF - ABIN651190
Berndt, Haverkampf, Reith, Keil, Niggemann, Zänker, Dittmar: Fusion of CCL21 Non-Migratory Active Breast Epithelial and Breast Cancer Cells Give Rise to CCL21 Migratory Active Tumor Hybrid Cell Lines. in PLoS ONE 2013
Human Monoclonal CCR7 Primary Antibody for FACS - ABIN4895611
Chimal-Ramírez, Espinoza-Sánchez, Chávez-Sánchez, Arriaga-Pizano, Fuentes-Pananá: Monocyte Differentiation towards Protumor Activity Does Not Correlate with M1 or M2 Phenotypes. in Journal of immunology research 2016
Human Polyclonal CCR7 Primary Antibody for IF, ELISA - ABIN1535569
Schweickart, Raport, Godiska, Byers, Eddy, Shows, Gray: Cloning of human and mouse EBI1, a lymphoid-specific G-protein-coupled receptor encoded on human chromosome 17q12-q21.2. in Genomics 1995
Human Polyclonal CCR7 Primary Antibody for FACS, WB - ABIN1742100
Sánchez-Sánchez, Riol-Blanco, Rodríguez-Fernández: The multiple personalities of the chemokine receptor CCR7 in dendritic cells. in Journal of immunology (Baltimore, Md. : 1950) 2006
Ccr7 functions during axis formation as a GPCR (show GPRC6A Antibodies) to inhibit beta-catenin (show CTNNB1 Antibodies), likely by promoting Ca(2 (show CA2 Antibodies)+) transients throughout the blastula.
Results suggest that baicalin exerts an inhibitory effect on airway inflammation, and this effect may be associated with the inhibition of CCR7 and its ligands, CCL19 (show CCL19 Antibodies) and CCL21 (show CCL21 Antibodies), as well as on the nuclear factor-Kappa B (NF-kappaB (show NFKB1 Antibodies)) pathway in a mouse model of asthma.
This study reveals a role for CCR7 in limiting Treg recirculation back to the thymus and enables separation of the mechanisms controlling Treg production and thymic recirculation.
data reveal that CCR7 plays multifaceted roles in regulating collecting vessel permeability and fibrosis, with one of the key players being IRF4 (show IRF4 Antibodies)-dependent DCs.
the graft site microenvironment plays a critical role in alloimmunity by determining DC trafficking through the CCR7-CCL19 (show CCL19 Antibodies)/21 axis.
Comprehensive Survey of miRNA-mRNA Interactions Reveals That Ccr7 and Cd247 (CD3 zeta (show CD247 Antibodies)) are Posttranscriptionally Controlled in Pancreas Infiltrating T Lymphocytes of Non-Obese Diabetic (NOD) Mice
CD11b (show ITGAM Antibodies) and CXCR4 (show CXCR4 Antibodies) but not CCR7 have roles in neutrophil migration modulating lymph node lymphocyte proliferation in bacterial infections
The results highlight a novel role of CCR7 in regulating effector CD8 (show CD8A Antibodies) T cell migration in the spleen and demonstrate differential requirement of CCR7 for primary and secondary CD8 (show CD8A Antibodies) T cell responses to infection.
mucosal draining lymph nodes express CCR7, which has a role in trafficking of RORgamma innate lymphoid cells
results demonstrate a contribution of CCR7 to STAP-2 (show STAP2 Antibodies)-dependent enhancement of BCR-ABL (show ABL1 Antibodies)-mediated cell growth in Ba/F3 cells
CCR7 increases the secretion of nitric oxide and modulates the T cell immune response.
we conclude that CCR7 gene locus harbours a polymorphism that modifies risk of MI in patients with Coronary artery disease (CAD (show CAD Antibodies)). Replication of this association could be sought in a prospective cohort of initially healthy individuals
this study shows that plasmacytoid dendritic cells from rheumatoid arthritis patients have high expression levels of CCR7
This study suggests that NRP1 (show NELL1 Antibodies) expression and LVD are independent factors that are likely to predict the risk of LN metastasis in squamous cell carcinoma (SCC (show CYP11A1 Antibodies))of the tongue, whereas the expression of VEGFC (show VEGFC Antibodies), VEGFR3 (show FLT4 Antibodies), CCR7, and SEMA3E (show SEMA3E Antibodies) are nonindependent predictive factors
High CCR7 expression is associated with gastric cancer.
CCR7 pathway up-regulates Twist expression via ERK (show EPHB2 Antibodies) and PI3K (show PIK3CA Antibodies)/AKT (show AKT1 Antibodies) signaling to manage the epithelial-mesenchymal transition of pancreatic ductal adenocarcinoma
Constitutively expressed Siglec-9 (show SIGLEC9 Antibodies) inhibits LPS (show IRF6 Antibodies)-induced CCR7, but enhances IL-4 (show IL4 Antibodies)-induced CD200R (show CD200R1 Antibodies) expression in human macrophages.
High CXCR4 (show CXCR4 Antibodies) expression in primary breast tumors (PTs (show PTS Antibodies)) was found to be associated with luminal A type tumor, suggesting more favorable outcome. In contrast, CCR7 and FOXP3 (show FOXP3 Antibodies) expressions in PTs (show PTS Antibodies) represented luminal B tumors, pointing to more aggressive tumor behavior. Maspin (show SERPINB5 Antibodies) expression did not differ between luminal types.
Results indicated that CCR7 is overexpressed in gallbladder cancer tissues and its expression correlates with staging and lymph node metastasis.
CCL21 (show CCL21 Antibodies)/CCR7 induce VEGF-D (show Figf Antibodies) up-regulation and promote lymphangiogenesis via ERK (show EPHB2 Antibodies)/Akt (show AKT1 Antibodies) pathway in lung cancer.
The present study demonstrated that down-regulation of CCR7 reduced proliferation, cell cycle, cell migration and invasion in prostate cancer cells
Fluorescent dye-labeled gammadelta T cells from afferent and efferent lymph lack CCR7 surface expression and display high levels of CD62L (show SELL Antibodies) compared with CD4 (show CD4 Antibodies) T cells, which do express CCR7.
CCR7 transcripts were minimally expressed in ex vivo and proliferating WC1(+)gammadelta T cells, a unique cell population with proinflammatory characteristics
the pattern of expression of CCR7 in different populations of porcine lymphocytes appears to be similar to that of human, highlighting the value of the pig as a useful animal model for biomedical studies.
Pre-translntation of sertli cell CCR7 significantly suppresses lymphocyte proliferation and prolongs allogeneic skin graft survival.
The protein encoded by this gene is a member of the G protein-coupled receptor family. This receptor was identified as a gene induced by the Epstein-Barr virus (EBV), and is thought to be a mediator of EBV effects on B lymphocytes. This receptor is expressed in various lymphoid tissues and activates B and T lymphocytes. It has been shown to control the migration of memory T cells to inflamed tissues, as well as stimulate dendritic cell maturation. The chemokine (C-C motif) ligand 19 (CCL19/ECL) has been reported to be a specific ligand of this receptor.
C-C chemokine receptor type 7
, CC chemokine receptor 7
, chemokine (C-C motif) receptor 7 a
, chemokine (C-C motif) receptor 7
, c-C chemokine receptor type 7-like
, C-C CKR-7
, EBV-induced G-protein coupled receptor 1
, MIP-3 beta receptor
, chemokine (C-C) receptor 7
, epstein-Barr virus-induced G-protein coupled receptor 1
, EBV-induced G protein-coupled receptor 1
, Epstein-Barr virus induced G-protein coupled receptor
, Epstein-Barr virus induced gene 1
, lymphocyte-specific G protein-coupled peptide receptor
, chemokine receptor 7