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Chemotactic factor. Additionally we are shipping CX3CL1 Kits (85) and CX3CL1 Proteins (68) and many more products for this protein.
Showing 10 out of 185 products:
Human Monoclonal CX3CL1 Primary Antibody for FACS - ABIN2664894
Ferretti, Pistoia, Corcione: Role of fractalkine/CX3CL1 and its receptor in the pathogenesis of inflammatory and malignant diseases with emphasis on B cell malignancies. in Mediators of inflammation 2014
Show all 7 references for ABIN2664894
Human Polyclonal CX3CL1 Primary Antibody for EIA, WB - ABIN499690
Mizuno, Kawanokuchi, Numata, Suzumura: Production and neuroprotective functions of fractalkine in the central nervous system. in Brain research 2003
Show all 4 references for ABIN499690
Human Polyclonal CX3CL1 Primary Antibody for IP, IHC - ABIN223526
Yeo, Kim, Ryu, Seo, Lee, Choi, Kim, Kang: The roles of fractalkine/CX3CR1 system in neuronal death following pilocarpine-induced status epilepticus. in Journal of neuroimmunology 2011
Show all 2 references for ABIN223526
Human Monoclonal CX3CL1 Primary Antibody for ELISA - ABIN1997219
Papadopoulos, Sassetti, Saeki, Yamada, Kawamura, Fitzhugh, Saraf, Schall, Blauvelt, Rosen, Hwang: Fractalkine, a CX3C chemokine, is expressed by dendritic cells and is up-regulated upon dendritic cell maturation. in European journal of immunology 1999
Human Polyclonal CX3CL1 Primary Antibody for IF (p), IHC (p) - ABIN728488
Xie, Shi, Zhang, Zhang, Gong, Guo, Wang, Xu, Wang, Chen, Liu, Dong: Abnormal activation of microglia accompanied with disrupted CX3CR1/CX3CL1 pathway in the brains of the hamsters infected with scrapie agent 263K. in Journal of molecular neuroscience : MN 2013
In the absence of the rd8 allele, deficiency of CCR2 (show CCR2 Antibodies) and CX3CL1 in mice leads to a mild form of retinal degeneration which is associated with the recruitment of macrophages, particularly to the subretinal space. This model enables to assess consequences of perturbed chemokine (show CCL1 Antibodies) signaling, but it does not recapitulate cardinal (show CARD8 Antibodies) age-related macular degeneration features.
Also icariin reduced CX3CR1 (show CX3CR1 Antibodies) and CX3CL1 protein levels in the artery wall. In conclusion, icariin could be a potential anti-atherosclerosis agent by downregulating the expression of CX3CR1 (show CX3CR1 Antibodies).
The biological activity of CX3CL1 is regulated by conversion of a membrane integrated to a soluble form during neurogenesis and in response to pathologic changes in the adult retinal milieu.
CX3CL1/CX3CR1 (show CX3CR1 Antibodies) signaling is involved in LTP (show SCP2 Antibodies) of C-fiber-evoked field potentials in the rodent spinal dorsal horn
CX3CL1/CX3CR1-mediated microglial activation plays a detrimental role in ischemic brain via p38MAPK/PKC signaling
Cx3cl1 overexpression suppresses alpha-synuclein (show SNCA Antibodies)-mediated neurodegeneration.
IFN-gamma (show IFNG Antibodies) induces aberrant CD49b (show ITGA2 Antibodies)+ NK cell recruitment and pregnancy failure through regulating CX3CL1.
tumours are characterized by expression of inflammatory chemokines (CCL2 (show CCL2 Antibodies), CCL5 (show CCL5 Antibodies), CCL7 (show CCL7 Antibodies), CCL8 (show CCL8 Antibodies), CCL12 (show Ccl12 Antibodies), CXCL9 (show CXCL9 Antibodies), CXCL10 (show CXCL10 Antibodies) and CX3CL1), reflected by an enrichment of activated Foxp3 (show FOXP3 Antibodies)(-) and Foxp3 (show FOXP3 Antibodies)(+) T cells
Insulin (show INS Antibodies) resistance increases plaque vulnerability by augmenting the CX3CL1/CX3CR1 (show CX3CR1 Antibodies) axis, which is mechanistically linked to reduced vascular smooth muscle cell survival
CX3CL1 may contribute to the regulation of toxigenic C. difficile infection.
CX3CL1 increased the migration and invasiveness of DU145 and PC-3 (show PCSK1 Antibodies), causing epithelial-to-mesenchymal transition. TACE (show ADAM17 Antibodies)/TGF-alpha (show TGFA Antibodies)/EGFR (show EGFR Antibodies) pathway activation and Slug (show SNAI2 Antibodies) upregulation were responsible for this.
Cell proliferation enhancing and anti-apoptosis activity requires the intracellular domain and apparently the dimerization of the transmembrane chemokine (show CCL1 Antibodies) ligand.
CX3CL1 was expressed only by the normal and cancer adjacent normal fallopian tube epithelium; its expression was largely lost in the malignant fallopian epithelium.
Skin and serum CX3CL1 elevated expression was associated with psoriasis severity.
CX3CL1(+) apo (show C9orf3 Antibodies)-MPs released by apoptotic cells mediate the chemotactic transmigration of alveolar macrophages.
Suggest that CX3CL1 participates in cross-talk mechanisms between endothelium and other muscle tissue cells and may promote a shift in the microenvironment toward a more regenerative milieu after exercise.
Report increased circulating fractalkine in STEMI patients, which was rapidly decreased after PCI (show SERPINA5 Antibodies).
This study found that CX3CL1 and TREM2 (show TREM2 Antibodies), two genes related to neuroinflammation, were expressed at higher levels in brain regions with pronounced vulnerability to Alzheimer disease-related changes.
MIP-1beta (show CCL4 Antibodies), TNF-alpha (show TNF Antibodies) and fractalkine are expressed in plasma, which reflects the levels of several pro-atherogenic cytokines in plaque tissue
The interactions of CX3CL1, LEPR and IL-6 genes might increase the risk of coronary artery disease in Chinese Han population.
Chemotactic factor. Binds to CX3CR1.
, chemokine (C-X3-C motif) ligand 1
, chemokine (C-X3-C motif) ligand 1, related sequence 1
, small inducible cytokine subfamily D, 1-related sequence 1
, CX3CL1 chemokine
, C-X3-C motif chemokine 1
, CX3C membrane-anchored chemokine
, small inducible cytokine subfamily D, 1
, small-inducible cytokine D1
, small inducible cytokine subfamily D (Cys-X3-Cys), member 1 (fractalkine, neurotactin)
, small inducible cytokine subfamily D (Cys-X3-Cys), member-1