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The protein encoded by CHRNA5 is a nicotinic acetylcholine receptor subunit and a member of a superfamily of ligand-gated ion channels that mediate fast signal transmission at synapses. Additionally we are shipping CHRNA5 Antibodies (73) and CHRNA5 Proteins (5) and many more products for this protein.
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The results of this study suggests a pleiotropic role of Chr15q25 CHRNA5-CHRNA3-CHRNB4 gene cluster with complex influences in ADHD, tobacco smoking and cognitive performance.
Polymorphisms in the CHRNA5 and NRXN1 (show NRXN1 ELISA Kits) genes, influenced greater consumption of cigarettes in a Mexican Mestizo population.
RP11 (show PRPF31 ELISA Kits)-650L12.2 rs149941240 polymorphism was associated with the risk of colorectal cancer.
We determined that the other patients harbored mutations in genes that have not previously been linked to RTT or other neurodevelopmental syndromes, such as the ankyrin repeat containing protein ANKRD31 or the neuronal acetylcholine receptor subunit alpha-5 (CHRNA5).
quitting smoking is highly beneficial in reducing lung cancer risks for smokers regardless of their CHRNA5 rs16969968 genetic risk status. Smokers with high-risk CHRNA5 genotypes, on average, can largely eliminate their elevated genetic risk for lung cancer by quitting smoking- cutting their risk of lung cancer in half and delaying its onset by 7years for those who develop it.
Study showed that multiple rare, low frequency and common variants in the CHRNA5 gene contribute to the development of nicotine dependence
the association of 3 selected single-nucleotide polymorphisms (CHRNA3 rs1051730, rs6495308, and CHRNA5 rs55853898) with nicotine dependence in an isolated population of Kashubians from Poland, is reported.
There was no association of rs16969968 polymorphism at CHRNA5 with cancer risk in the overall pooled analysis
focus on the CHRNA5/A3/B4 gene cluster and its role in nicotine dependence (review)
Preventive interventions in adolescents reduced the genetic risk for smoking from rs16969968 CHRNA5.
These results highlight the involvement of the alpha5 nAChR (show CHRNA4 ELISA Kits) subunit in the development of hyperalgesia, allodynia and inflammation associated with chronic neuropathic and inflammatory pain models.
Overexpression of the genomic cluster CHRNA5/A3/B4 in mice increases somatic signs associated with morphine withdrawal and Knockout of alpha5 gene reduced somatic signs in morphine-treated mice
Data reveal a highly regulated temporal-spatial pattern of alpha5 nAChR (show CHRNA4 ELISA Kits) subunit expression during important periods of lung morphogenesis by FoxA2 (show FOXA2 ELISA Kits).
increased nicotine intake in mice with a null mutation in Chrna5
Polymorphisms located within the Chrna5-Chrna3-Chrnb4 cluster on mouse chromosome 9 were found to co-segregate with alcohol preference, with high-drinking F(2) mice carrying B6 alleles and low-drinking F(2) mice carrying D2 alleles.
The protein encoded by this gene is a nicotinic acetylcholine receptor subunit and a member of a superfamily of ligand-gated ion channels that mediate fast signal transmission at synapses. These receptors are thought to be heteropentamers composed of separate but similar subunits. Defects in this gene have been linked to susceptibility to lung cancer type 2 (LNCR2).
neuronal acetylcholine receptor subunit alpha-5
, cholinergic receptor, nicotinic, alpha polypeptide 5
, Cholinergic receptor, neuronal nicotinic, alpha polypeptide-5
, acetylcholine receptor, nicotinic, alpha 5 (neuronal)
, neuronal nicotinic acetylcholine receptor, alpha5 subunit
, acetylcholine receptor alpha 5
, cholinergic receptor neuronal nicotinic alpha protein 5
, nicotinic acetylcholine receptor alpha5 subunit