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The human complement subcomponent C1q associates with C1r and C1s in order to yield the first component of the serum complement system. Additionally we are shipping C1QBP Kits (27) and C1QBP Proteins (21) and many more products for this protein.
Showing 10 out of 108 products:
Human Polyclonal C1QBP Primary Antibody for EIA, WB - ABIN360189
Biswas, Hafiz, Banerjee, Kim, Datta, Chitnis: Plasmodium falciparum uses gC1qR/HABP1/p32 as a receptor to bind to vascular endothelium and for platelet-mediated clumping. in PLoS pathogens 2007
Show all 3 references for ABIN360189
Human Polyclonal C1QBP Primary Antibody for EIA, WB - ABIN360188
Lauzon, Mian, Ashkar: Toll-like receptors, natural killer cells and innate immunity. in Advances in experimental medicine and biology 2007
Show all 3 references for ABIN360188
Mouse (Murine) Polyclonal C1QBP Primary Antibody for IP, ELISA - ABIN2000541
LESKOWITZ, OVARY: The relation between molecular weight of antigen and ability to elicit passive cutaneous anaphylaxis. in Immunology 1970
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Human Polyclonal C1QBP Primary Antibody for IHC, ELISA - ABIN1534795
Honoré, Madsen, Rasmussen, Vandekerckhove, Celis, Leffers: Cloning and expression of a cDNA covering the complete coding region of the P32 subunit of human pre-mRNA splicing factor SF2. in Gene 1994
C1QBP interacts with DLAT (show DLAT Antibodies) and regulates the enzyme activity of pyruvate dehydrogenase (show PDP Antibodies).
Sp1 (show PSG1 Antibodies)-ZNF32 (show ADRA2A Antibodies)-C1QBP axis protects against oxidative stress/apoptosis in hepatocellular carcinoma cells.
Findings highlight a cytoprotective role of p32 under starvation conditions by regulating ULK1 (show ULK1 Antibodies) stability, and uncover a crucial role of the p32-ULK1 (show ULK1 Antibodies)-autophagy axis in coordinating stress response, cell survival and mitochondrial homeostasis.
p32 appeared to be a core component of herpesvirus nuclear egress complexes, like UL31 and UL34 homologs in other herpesviruses, and to play multiple roles in herpesvirus nuclear egress.
data suggest that C1QBP is a novel regulator of YBX1 (show YBX1 Antibodies), and the expression of C1QBP and the nuclear expression of YBX1 (show YBX1 Antibodies) could both be used as independent prognostic makers for cancer progression in the RCC (show XRCC1 Antibodies) patients
Data show that p32 hyaluronan binding protein (p32) is a direct transcriptional target of oncogene Myc and that high level of Myc in malignant brain cancers correlates with high expression of p32.
HABP1 protein high expression may contribute to the tumor progression and poor prognosis of TNBC, especially in predicting prognosis in TNBCs without lymph node metastasis.
our findings suggest that the C1QBP protein could be a potential proliferative marker in breast cancer
Our results indicated that overexpression of HABP1 may serve as a new biomarker to predict the progression and prognosis of endometrial cancer.
Interaction between HSV-1 ICP34.5 and p32 leads to the disintegration of nuclear lamina and facilitates the nuclear egress of HSV-1 particles.
application of p33 (show LTB Antibodies) significantly improved survival in mice receiving an otherwise lethal dose of histones.
Authors propose that p32 is required for functional mitoribosome formation to synthesize proteins within mitochondria.
we have identified a mitochondrial protein (show COX6B2 Antibodies) p32 as a novel interactor of parkin (show PARK2 Antibodies) in the brain
C1qbp could directly bind to CypD (show CYPD Antibodies). Therefore C1qbp appears to act as an endogenous inhibitor of the MPT pore, most likely through binding to CypD (show CYPD Antibodies), and thus protects cells against oxidative stress.
Intracellular localization and further functional studies suggested that CHCHD2 and HABP1 may mutually regulate each other to balance cell migration.
Rat and mouse homologs of the HABP1 pseudogene also contain multiple mutations, leading to the insertion of premature stop codons confirming the identity of a processed pseudogene.
Study demonstrates the differential expression of HABP1 during progression of epidermal carcinoma.
p32 phosphorylation by ATM (show ATM Antibodies) might be a new transcriptional regulatory pathway for specific DNA damage responses in heart.
The reduction in oxidant generation and drop in apoptotic cell population caused by disruption of HABP1, corroborating the fact that excess ROS (show ROS1 Antibodies) generation in HABP1 overexpressing cells leading to apoptosis was due to mitochondrial HABP1 accumulation.
Differential isoform expression and interaction with the P32 regulatory protein (show TGFB1 Antibodies) controls the subcellular localization of the splicing factor (show SLU7 Antibodies) U2AF26 (show U2AF1L4 Antibodies)
The human complement subcomponent C1q associates with C1r and C1s in order to yield the first component of the serum complement system. The protein encoded by this gene is known to bind to the globular heads of C1q molecules and inhibit C1 activation. This protein has also been identified as the p32 subunit of pre-mRNA splicing factor SF2, as well as a hyaluronic acid-binding protein.
p32 subunit of splicing factor SF2
, 38k protein
, complement component 1, q subcomponent binding protein
, ASF/SF2-associated protein p32
, C1q globular domain-binding protein
, complement component 1 Q subcomponent-binding protein, mitochondrial
, glycoprotein gC1qBP
, hyaluronan-binding protein 1
, mitochondrial matrix protein p32
, splicing factor SF2-associated protein
, GC1q-R protein