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Receptor for the chemotactic and inflammatory peptide anaphylatoxin C3a. Additionally we are shipping C3AR1 Kits (16) and C3AR1 Proteins (6) and many more products for this protein.
Showing 10 out of 49 products:
Human Monoclonal C3AR1 Primary Antibody for FACS, IP - ABIN2662915
Honczarenko, Ratajczak, Nicholson-Weller, Silberstein: Complement C3a enhances CXCL12 (SDF-1)-mediated chemotaxis of bone marrow hematopoietic cells independently of C3a receptor. in Journal of immunology (Baltimore, Md. : 1950) 2005
Show all 6 references for ABIN2662915
Human Monoclonal C3AR1 Primary Antibody for FACS, IHC - ABIN2664086
Reca, Mastellos, Majka, Marquez, Ratajczak, Franchini, Glodek, Honczarenko, Spruce, Janowska-Wieczorek, Lambris, Ratajczak: Functional receptor for C3a anaphylatoxin is expressed by normal hematopoietic stem/progenitor cells, and C3a enhances their homing-related responses to SDF-1. in Blood 2003
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Human Monoclonal C3AR1 Primary Antibody for FACS - ABIN2656881
Wysoczynski, Reca, Lee, Wu, Ratajczak, Ratajczak: Defective engraftment of C3aR-/- hematopoietic stem progenitor cells shows a novel role of the C3a-C3aR axis in bone marrow homing. in Leukemia 2009
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Neural crest cells are coattracted via the complement fragment C3a (show C3 Antibodies) and its receptor C3aR, revealing an unexpected role of complement proteins in early vertebrate development.
High C3aR1 expression is associated with acute myeloid leukemia (show BCL11A Antibodies)-M2.
these findings suggest a critical role for C3a (show C3 Antibodies) through autocrine/paracrine induction of C3aR in the pathogenesis of cigarette smoke-induced sterile inflammation and provide new therapeutic targets for the treatment of emphysema.
Medulloblastoma cells express C3aR, and siRNA-mediated knockdown of C3aR inhibits proliferation of these cells in vitro.
Observed a positive correlation between the expression of anaphylatoxin-receptors C3aR and C5aR (show C5AR1 Antibodies) with platelet activation in patients with coronary artery disease.
The obesity-induced down-regulation of complement C3 (show C3 Antibodies) and C3aR which is specific to subcutaneous adipose tissue.
In patients with IgAN, urinary and renal C3a (show C3 Antibodies) and C5a and renal expression of C3aR and C5aR (show C5AR1 Antibodies) are significantly correlated with the activity and severity of renal injury.
C3aR and CD46 (show CD46 Antibodies) activation via intrinsic generation of their respective ligands is an integral part of human Th1 (show TH1L Antibodies) (but not Th2) immunity.
functional regulation of C3aR by NHERFs in human mast cells
significantly decreased expression of C3aR mRNA and protein expression in placentas with preeclampsia compared to controls
study demonstrates that although C3a (show C3 Antibodies) causes phosphorylation of its receptor at multiple sites, Ser459, Thr463, Ser465, Thr466 and Ser470 participate in C3aR desensitization
The roles of C3, CfB (show CFB Antibodies), and C3a (show C3 Antibodies) receptor in the severity of S. aureus induced septic arthritis are reported.
increased circulating lipopolysaccharide induces upregulation of complement C3a (show C3 Antibodies) receptor on neutrophils and activation of the complement cascade.
showed that C3a (show C3 Antibodies) receptor/C5a receptor-deficient mice developed much less severe uveitis than did control mice using all retinal examination methods
C3a (show C3 Antibodies)/C3aR signaling during host immune response suppresses Fas (show FAS Antibodies) expression and caspase-3 (show CASP3 Antibodies) activity while increasing Bcl-2 (show BCL2 Antibodies) proto-oncogene (show RAB1A Antibodies) protein expression, providing protection to myeloid and lymphoid cells against L. monocytogenes-induced apoptosis.
This study indicated that C3a (show C3 Antibodies) and C5a-mediated functions are not critical to the development of experimental autoimmune uveitis.
The binding of TLQP-21 to C3AR1 not only has effects on signaling but also modulates cellular functions
C3a (show C3 Antibodies) receptor promotes viral containment in mice inoculated with vaccinia virus at sites of allergic skin inflammation.
The role of the anaphylatoxin C3a receptor in acute dextran sulfate sodium-induced colitis in mice, was investigated.
ALI in H5N1-infected mice was caused by excessive complement activation, as demonstrated by deposition of C3, C5b-9, and MBL-C in lung tissue, and by up-regulation of MBL-associated serine protease-2 (show MASP2 Antibodies) and the complement receptors C3aR and C5aR (show C5AR1 Antibodies).
Receptor for the chemotactic and inflammatory peptide anaphylatoxin C3a. This receptor stimulates chemotaxis, granule enzyme release and superoxide anion production (By similarity).
c3a anaphylatoxin chemotactic receptor
, C3a anaphylatoxin chemotactic receptor
, complement component 3a receptor 1
, complement component 3 receptor 1
, anaphylatoxin C3a receptor