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Receptor for the chemotactic and inflammatory peptide anaphylatoxin C5a. Additionally we are shipping C5AR1 Kits (13) and C5AR1 Proteins (7) and many more products for this protein.
Showing 10 out of 255 products:
Human Monoclonal C5AR1 Primary Antibody for FACS - ABIN951419
Kiafard, Tschernig, Schweyer, Bley, Neumann, Zwirner: Use of monoclonal antibodies to assess expression of anaphylatoxin receptors in tubular epithelial cells of human, murine and rat kidneys. in Immunobiology 2007
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Human Monoclonal C5AR1 Primary Antibody for FACS - ABIN951422
Zwirner, Fayyazi, Götze: Expression of the anaphylatoxin C5a receptor in non-myeloid cells. in Molecular immunology 2000
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Human Polyclonal C5AR1 Primary Antibody for EIA, FACS - ABIN951423
Skeie, Fingert, Russell, Stone, Mullins: Complement component C5a activates ICAM-1 expression on human choroidal endothelial cells. in Investigative ophthalmology & visual science 2010
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Human Polyclonal C5AR1 Primary Antibody for IHC, ELISA - ABIN1531549
Gerard, Gerard: The chemotactic receptor for human C5a anaphylatoxin. in Nature 1991
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Human Polyclonal C5AR1 Primary Antibody for FACS, WB - ABIN1742122
Laudes, Chu, Sikranth, Huber-Lang, Guo, Riedemann, Sarma, Schmaier, Ward: Anti-c5a ameliorates coagulation/fibrinolytic protein changes in a rat model of sepsis. in The American journal of pathology 2002
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Human Monoclonal C5AR1 Primary Antibody for FACS - ABIN119310
Elias, Reinhardt, Hautmann, Harms, Oppermann, Schroeter, Hellenbrand, Oster, Kurzai, Taha, Nossal, Frosch, Vogel: [3rd Workshop on Epidemiology, Prevention and Treatment of Invasive Meningococcal Disease in Würzburg 2006] in Gesundheitswesen (Bundesverband der Ärzte des Öffentlichen Gesundheitsdienstes (Germany)) 2007
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Human Monoclonal C5AR1 Primary Antibody for FACS, IHC (fro) - ABIN181855
Oppermann, Raedt, Hebell, Schmidt, Zimmermann, Götze: Probing the human receptor for C5a anaphylatoxin with site-directed antibodies. Identification of a potential ligand binding site on the NH2-terminal domain. in Journal of immunology (Baltimore, Md. : 1950) 1993
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Rat (Rattus) Monoclonal C5AR1 Primary Antibody for FACS - ABIN120012
Rothermel, Götze, Zahn, Schlaf: Analysis of the tissue distribution of the rat C5a receptor and inhibition of C5a-mediated effects through the use of two MoAbs. in Scandinavian journal of immunology 2000
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Human Polyclonal C5AR1 Primary Antibody for FACS, WB - ABIN654658
Marc, Kristan, Rozman, Kern, Flezar, Kosnik, Korosec: Complement factor C5a in acute exacerbation of Chronic Obstructive Pulmonary Disease. in Scandinavian journal of immunology 2010
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Cow (Bovine) Monoclonal C5AR1 Primary Antibody for FACS, IHC (p) - ABIN118843
Werfel, Zwirner, Oppermann, Sieber, Begemann, Drommer, Kapp, Götze: CD88 antibodies specifically bind to C5aR on dermal CD117+ and CD14+ cells and react with a desmosomal antigen in human skin. in Journal of immunology (Baltimore, Md. : 1950) 1996
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CYP4F18 (show CYP4F3 Antibodies)-deficient neutrophils exhibit increased chemotaxis to complement component C5a.
showed that C3a (show C3 Antibodies) receptor/C5a receptor-deficient mice developed much less severe uveitis than did control mice using all retinal examination methods
Knockout of the C5aR1 attenuated the effect of beta1-AR in the heart, suggesting an association between the beta1-AR and C5aR1, although further investigation is required to determine if this is a direct or causal association
Data (including data from studies in knockout mice) suggest that C5aR/C5aR (complement C5a/anaphylatoxin (show C5 Antibodies) C5a Receptor) signaling pathway is involved in neurocognitive injury in uninfected pups induced by malaria in pregnancy.
Data indicate that C5ar1 activation plays a role in seizure initiation and severity, as well as neuronal degeneration following status epilepticus.
identifies a key role for C5aR signaling in astrocyte proliferation and glial scarring during the chronic phase of Spinal Cord Injury.
Porphyromonas gingivalis induced C5aR-TLR2 (show TLR2 Antibodies) crosstalk leads to MyD88 degradation.
Complement receptor C5aR1/CD88 and dipeptidyl peptidase-4/CD26 (show DPP4 Antibodies) define distinct hematopoietic lineages of dendritic cells.
Deletion of the complement C5a receptor alleviates the severity of acute pneumococcal otitis media following influenza A virus infection in mice.
our findings suggest that C5aR1 expression in mice is largely restricted to cells of the myeloid lineage
that C5aR stimulates cell invasion and migration via ERK1/2 (show MAPK1/3 Antibodies)-mediated epithelial-mesenchymal transition in hepatocellular carcinoma cells
Inhibition of C5aR1 leads to a significant reduction of CD66b and CD11b (show ITGAM Antibodies) expression indicating a lower neutrophil activation status and block of granule exocytosis.
that rs10853784 of C5aR1 gene are associated with coronary artery disease in Han population of China
96.7% of the metastatic RCCs had C5aR expression, vs. 50.5% of the non-metastatic ones. C5a triggered ERK (show EPHB2 Antibodies)- and PI3Kdependent invasion of the C5aR-expressing renal carcinoma (show TSC2 Antibodies) cells. The C5a-C5aR axis plays a crucial role in renal carcinoma (show TSC2 Antibodies) cell invasion.
These results illustrate the novel activity of the C5a-C5aR axis that promotes human NPC (show NPC1 Antibodies) cell proliferation through PCAFmediated STAT3 (show STAT3 Antibodies) acetylation.
C5a/C5aR1 signalling plays a key role in disrupting blood-brain barrier integrity through different cascades including NF-KappaB translocation leading to altered tight junction proteins, Claudin-5 and ZO-1, and actin reorganization in the lupus setting
). Low frequency of the C5aR 450 CT genotype, which apparently is a feature of certain kidney diseases, appears to be associated with good graft outcome in kidney transplantation.
Observed a positive correlation between the expression of anaphylatoxin-receptors C3aR (show C3AR1 Antibodies) and C5aR with platelet activation in patients with coronary artery disease.
C5AR1 SNP -1330T/G may serve as a useful pharmacodynamic predictor of nonsedating H1-antihistamine efficacy in chronic spontaneous urticaria (CSU), and -1330T alleles may be taken as a risk factor for CSU.
Porphyromonas gingivalis induces C5aR-TLR2 (show TLR2 Antibodies) coassociation in neutrophils.
Studies provide information regarding distinct structural and functional features that may contribute to the unique pharmacological properties of bovine C5aR.
Receptor for the chemotactic and inflammatory peptide anaphylatoxin C5a. This receptor stimulates chemotaxis, granule enzyme release and superoxide anion production.
complement component 5a receptor 1
, C5a anaphylatoxin receptor
, G protein-coupled receptor 77
, C5a anaphylatoxin chemotactic receptor 1
, C5a ligand
, complement component 5, receptor 1
, complement component 5 receptor 1
, complement C5a receptor
, anaphylatoxin C5a receptor
, complement component 5 receptor 1 (C5a ligand)
, C5a anaphylatoxin chemotactic receptor