D Site-Binding Protein Proteins (DBP)

The protein encoded by DBP is a member of the PAR bZIP transcription factor family and binds to specific sequences in the promoters of several genes, such as albumin, CYP2A4, and CYP2A5. Additionally we are shipping and many more products for this protein.

list all proteins Gene Name GeneID UniProt
Mouse DBP DBP 13170 Q60925
DBP 24309 P16443
Human DBP DBP 1628 Q10586
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Catalog No. Origin Source Conjugate Quantity Supplier Delivery Price Details
Yeast Rat His tag 1 mg Log in to see 60 to 71 Days
Yeast Cow His tag 1 mg Log in to see 60 to 71 Days

DBP Proteins by Origin and Source

Origin Expressed in Conjugate
Rat (Rattus)

More Proteins for D Site-Binding Protein (DBP) Interaction Partners

Mouse (Murine) D Site-Binding Protein (DBP) interaction partners

  1. PI3K signaling plays a modulatory role in the regulation of the transcriptional rhythm of the Dbp (show GC Proteins) gene by targeting BMAL1 (show ARNTL Proteins) and CLOCK.

  2. constitutive Dbp (show GC Proteins) expression plays an important role in mediating hepatic dysfunction under chronic kidney disease. DBP (show GC Proteins) was significantly decreased in 5/6Nx model mice.

  3. Dbp (show GC Proteins) plays a well-established role in circadian rhythms.

  4. Expressions of Dbp (show GC Proteins) and Arnt (show ARNT Proteins) were dampened in the islets of a diabetic model mouse.

  5. BMAL1 (show ARNTL Proteins) and CLOCK may act as Kamikaze activators, in that they are rapidly degraded once bound to Dbp (show GC Proteins) chromatin.

  6. In Ccdc80(-/-) mice, expression of the core clock member Arntl/Bmal1 (show ARNTL Proteins) was reduced whereas that of the oscillating transcription factors Dbp (show GC Proteins) and Tef (show TEF Proteins) was increased in all tissues examined.

  7. The daily mRNA expression profiles of the clock and clock-controlled genes Clock, Bmal1 (show ARNTL Proteins), Cry1 (show CRY1 Proteins), Per1 (show PER1 Proteins), Per2 (show PER2 Proteins), and Dbp (show GC Proteins) were substantially dampened in the liver and adipose tissue of 10-wk-old ob/ob mice.

  8. we demonstrate that D site albumin promoter binding protein/thyrotroph embryonic factor (show TEF Proteins)/hepatic leukemia factor (show HLF Proteins) triple knockout mice develop cardiac hypertrophy and left ventricular dysfunction associated with a low blood pressure

  9. CRY1 (show CRY1 Proteins) binding to the Dbp (show GC Proteins) promoter region delayed BMAL1 (show ARNTL Proteins) and CLOCK-mediated transcription of Dbp (show GC Proteins) compared with Rev-Erbalpha (show NR1D1 Proteins)

  10. mice deficient for all three PAR (show AFG3L2 Proteins) bZip proteins (DBP (show GC Proteins),HLF (show HLF Proteins),& TEF (show TEF Proteins)) are highly susceptible to generalized spontaneous and audiogenic epilepsies, frequently lethal. Pyridoxal kinase (show PDXK Proteins) is a target gene of PAR (show AFG3L2 Proteins) bZip proteins in both liver and brain

Human D Site-Binding Protein (DBP) interaction partners

  1. Data suggest that, while low circulating levels of DBP (show GC Proteins) contribute to low circulating levels of 25-hydroxyvitamin D in patients with PHPT (primary hyperparathyroidism), low DBP (show GC Proteins) alone is not responsible for the hypovitaminosis D observed in these patients; vitamin D metabolism is likely to be generally disturbed in PHPT. [EDITORIAL]

  2. The interaction between vitamin D status, as measured by circulating 25(OH)D and DBP (show GC Proteins) rs2282679 genotypes, modified the association between 25(OH)D and BMD (show BEST1 Proteins) and bone markers.

  3. the expression of DBP (show GC Proteins) and MAPK (show MAPK1 Proteins) in epilepsy patients was upregulated, suggesting a possible pathogenetic role in IE.

  4. Increased circulating levels of vitamin D binding protein (show GC Proteins) in multiple sclerosis patients.

  5. Results show that APOE (show APOE Proteins), DBP (show GC Proteins) and AGT (show AGXT Proteins) identified were associated with survival outcomes in metastatic colorectal cancer patients treated with chemotherapy and bevacizumab.

  6. DBP (show GC Proteins) strictly inhibited the production of 1alpha,25-dihydroxyvitamin D3 and 25-hydroxyvitamin D3-induced T cell responses.

  7. 25(OH)D2 half-life was shorter than 25(OH)D3 half-life, and half-lives were affected by DBP (show GC Proteins) concentration and genotype.

  8. Lean individuals exhibited significant (P < 0.05) temporal changes of core clock (PER1 (show PER1 Proteins), PER2 (show PER2 Proteins), PER3, CRY2 (show CRY2 Proteins), BMAL1 (show ARNTL Proteins), and DBP (show GC Proteins)) and metabolic (REVERBalpha (show NR1D1 Proteins), RIP140 (show NRIP1 Proteins), and PGC1alpha) genes.

  9. These results provide a direct evidence of cross-talk among the structural domains of DBP (show GC Proteins).

  10. Data show that vitamin D-binding protein (DBP (show GC Proteins)) is elevated in the CSF (show CSF2 Proteins) of temporal lobe epilepsy patients.

DBP Protein Profile

Protein Summary

The protein encoded by this gene is a member of the PAR bZIP transcription factor family and binds to specific sequences in the promoters of several genes, such as albumin, CYP2A4, and CYP2A5. The encoded protein can bind DNA as a homo- or heterodimer and is involved in the regulation of some circadian rhythym genes.

Gene names and symbols associated with D Site-Binding Protein Proteins (DBP)

  • D site albumin promoter binding protein (Dbp)
  • D site of albumin promoter (albumin D-box) binding protein (Dbp)
  • D site of albumin promoter (albumin D-box) binding protein (DBP)
  • DABP protein

Protein level used designations for D Site-Binding Protein Proteins (DBP)

D site-binding protein , albumin D box-binding protein , albumin D-element-binding protein , D site albumin promoter binding protein , d site albumin promoter-binding protein 1 , tax-responsive enhancer element-binding protein 302 , taxREB302 , albumin D-element binding protein

13170 Mus musculus
24309 Rattus norvegicus
1628 Homo sapiens
503577 Bos taurus
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