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CpG methylation is an epigenetic modification that is important for embryonic development, imprinting, and X-chromosome inactivation. Additionally we are shipping DNMT3B Proteins (5) and DNMT3B Kits (1) and many more products for this protein.
Showing 10 out of 123 products:
Human Monoclonal DNMT3B Primary Antibody for ChIP, CyTOF - ABIN252478
Santoro, Li, Grummt: The nucleolar remodeling complex NoRC mediates heterochromatin formation and silencing of ribosomal gene transcription. in Nature genetics 2002
Show all 97 references for ABIN252478
Human Polyclonal DNMT3B Primary Antibody for ICC, IF - ABIN151734
Robert, Morin, Beaulieu, Gauthier, Chute, Barsalou, MacLeod: DNMT1 is required to maintain CpG methylation and aberrant gene silencing in human cancer cells. in Nature genetics 2003
Show all 15 references for ABIN151734
Human Polyclonal DNMT3B Primary Antibody for IHC (p), WB - ABIN387884
Lu, Markowetz, Unwin, Leek, Airoldi, MacArthur, Lachmann, Rozov, Maayan, Boyer, Troyanskaya, Whetton, Lemischka: Systems-level dynamic analyses of fate change in murine embryonic stem cells. in Nature 2009
Show all 13 references for ABIN387884
Human Polyclonal DNMT3B Primary Antibody for ChIP, IHC - ABIN152675
Okano, Bell, Haber, Li: DNA methyltransferases Dnmt3a and Dnmt3b are essential for de novo methylation and mammalian development. in Cell 1999
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Human Monoclonal DNMT3B Primary Antibody for ChIP, WB - ABIN2668954
Castro, Breiling, Luetkenhaus, Ceteci, Hausmann, Kress, Lyko, Rudel, Rapp: MYC-induced epigenetic activation of GATA4 in lung adenocarcinoma. in Molecular cancer research : MCR 2013
Show all 2 references for ABIN2668954
Human Polyclonal DNMT3B Primary Antibody for IF (p), IHC (p) - ABIN727623
Zhao, Hou, Chen, Shao, Zhu, Bu, Gu, Li, Zhang, Du, Fu, Kong, Luo, Long, Li, Deng, Zhao, Cen: Prenatal cocaine exposure impairs cognitive function of progeny via insulin growth factor II epigenetic regulation. in Neurobiology of disease 2015
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Human Polyclonal DNMT3B Primary Antibody for EIA, IHC (p) - ABIN356569
Stipani, Cappello, Daddabbo, Natuzzi, Miniero, Stipani, Palmieri: The mitochondrial oxoglutarate carrier: cysteine-scanning mutagenesis of transmembrane domain IV and sensitivity of Cys mutants to sulfhydryl reagents. in Biochemistry 2001
Human Polyclonal DNMT3B Primary Antibody for IF, IHC - ABIN1534163
Deloukas, Matthews, Ashurst, Burton, Gilbert, Jones, Stavrides, Almeida, Babbage, Bagguley, Bailey, Barlow, Bates, Beard, Beare, Beasley, Bird, Blakey, Bridgeman, Brown, Buck, Burrill, Butler, Carder et al.: The DNA sequence and comparative analysis of human chromosome 20. ... in Nature 2002
Cow (Bovine) Polyclonal DNMT3B Primary Antibody for WB - ABIN2783648
Boland, Christman: Characterization of Dnmt3b:thymine-DNA glycosylase interaction and stimulation of thymine glycosylase-mediated repair by DNA methyltransferase(s) and RNA. in Journal of molecular biology 2008
dnmt7 specifically methylates no tail gene in the genome
Among 18 genotypes analyzed, we were unable to record any significant differences in 5-methyl-2'-deoxycytidine levels, which suggested that age-related changes in global DNA methylation (show HELLS Antibodies) content are rather a function of time, and not a genetic component.
data indicated that Kaempferol is a novel DNMT3B inhibitor, which may promote the degradation of DNMT3B in bladder cancer.
TLR9 (show TLR9 Antibodies) upregulation in cases with episomal HPV16 was again higher among those with non-methylated immunostimulatory CpG motifs. Comparison of cases with HPV-negative controls revealed that DNMT3A (show DNMT3A Antibodies) was significantly downregulated only among integrated cases, DNMT3B was significantly overexpressed among both categories of cases, although at variable levels, while DNMT1 (show DNMT1 Antibodies) failed to show any deregulated expression among the cases
age affects the expression of DNMT1 (show DNMT1 Antibodies) and DNMT3B as an almost independent variable in respect of all other variables evaluated.
Findings suggest that PTEN and DNMT3B possess common regulation features as well as certain ethnic differences in expression between Han women and Uygur women.
This condensed chromatin structure is associated with binding of DNMT3B and decreased occupancy of OCT1 (show POU2F1 Antibodies) transcription factor at MAML2 (show MAML2 Antibodies) enhancer, suggesting a role of DNMT3B in increasing methylation of MAML2 (show MAML2 Antibodies) after stilbenoid treatment.
show that heterozygous mutations in DNA methyltransferase 3B (DNMT3B) are a likely cause of D4Z4 derepression associated with low levels of DUX4 expression
The de novo methyltransferases DNMT3A (show DNMT3A Antibodies) and 3B play a vital role in methylating the genome of the developing embryo in regions devoid of methylation marks.These observations suggest a potential interaction of G-quadruplexes with the DNA methylation (show HELLS Antibodies) machinery, which may be of epigenetic and biological significance
149C>T and -2437T>A DNMT3B polymorphisms are not correlated with lung cancer risk among Chinese population nor the haplotype of them.
DNMT1 (show DNMT1 Antibodies), DNMT3A (show DNMT3A Antibodies) and DNMT3B are highly expressed in human medulloblastoma samples
The DNMT3A (show DNMT3A Antibodies) -448A > G polymorphism may be a novel functional SNP and contribute to its genetic susceptibility to spontaneous abortion in Chinese women, and ART may not affect the distribution of -448A > G in pregnancy loss and normal pregnancy. The observed TT genotype of DMNT3B suggests that this is the predominant genotype of this population
The epiblast expressed epithelial markers, MUC1 (show MUC1 Antibodies) and E-CADHERIN (show CDH1 Antibodies), and the pluripotency markers, DNMT3B and CRIPTO (show TDGF1 Antibodies).
Developmental changes in expression of DNMT3B are indicative of a possible role in changes in methylation in cattle.
The expression levels of DNMT3a (show DNMT3A Antibodies) and DNMT3b were associated with several beef quality traits.
While lens epithelial cell survival requires DNMT1 (show DNMT1 Antibodies), morphologically normal lenses develop in the absence of both DNMT3A (show DNMT3A Antibodies) and DNMT3B.
Mechanical stimulation regulates osteoblastic genes expression via direct regulation of Dnmt3b.
a miR (show MLXIP Antibodies)-125b-DNMT3b-p53 (show TP53 Antibodies) signal pathway may exist in the vascular smooth muscle cells proliferation induced by homocysteine.
miR (show MLXIP Antibodies)-29a mimic transfection lowered collagen 1alpha1, DNMT1 (show DNMT1 Antibodies), DNMT3b and SET1A (show SETD1A Antibodies) expression in hepatic stellate cells.
Loss of DNMT3B results in hypomethylation of the miR (show MLXIP Antibodies)-196b promoter and increased miR (show MLXIP Antibodies)-196b expression, which directly targets the mTORC2 (show CRTC2 Antibodies) component Rictor (show RICTOR Antibodies).
The findings define PRMT7 (show PRMT7 Antibodies) as a regulator of the DNMT3b/p21 (show D4S234E Antibodies) axis required to maintain muscle stem cell regenerative capacity.
These results demonstrate that Dnmt1 (show DNMT1 Antibodies) and Dnmt3b cooperate to maintain DNA methylation (show HELLS Antibodies) and genomic integrity in the intestinal epithelium.
Downregulation of DNMT3B, one of the targets identified using this method, radiosensitizes cancer cells by disturbing multiple DNA damage responses.
High levels of Dnmt3b expression prolong leukemia latency.
The expression of DNMT1 (show DNMT1 Antibodies) and DNMT3b was decreased at 1 mT, and 50 Hz ELF (show SPTBN1 Antibodies)-EMF can increase the expression...the alterations of genome-wide methylation and DNMTs expression may play an important role in the biological effects of 50 Hz ELF (show SPTBN1 Antibodies)-EMF exposure
CpG methylation is an epigenetic modification that is important for embryonic development, imprinting, and X-chromosome inactivation. Studies in mice have demonstrated that DNA methylation is required for mammalian development. This gene encodes a DNA methyltransferase which is thought to function in de novo methylation, rather than maintenance methylation. The protein localizes primarily to the nucleus and its expression is developmentally regulated. Mutations in this gene cause the immunodeficiency-centromeric instability-facial anomalies (ICF) syndrome. Eight alternatively spliced transcript variants have been described. The full length sequences of variants 4 and 5 have not been determined.
DNA (cytosine-5)-methyltransferase 3B
, DNA (cytosine-5-)-methyltransferase 3 beta
, DNA cytosine-5 methyltransferase 3 beta
, DNA (cytosine-5)-methyltransferase 3B-like
, DNA methyl transferase beta
, DNA methyltransferase 3B
, DNA MTase HsaIIIB
, DNA methyltransferase HsaIIIB
, DNA (cytosine-5-)-methyltransferase 3 beta, like
, DNA (cytosine-5-)-methyltransferase 7
, DNA MTase MmuIIIB
, DNA methyltransferase MmuIIIB