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Members of the dystrophin family of proteins perform a critical role in the maintenance of membrane-associated complexes at points of intercellular contact in vertebrate cells. Additionally we are shipping DRP2 Antibodies (23) and and many more products for this protein.
study implies that dysregulation of CRMP2 (show DPYSL2 ELISA Kits) may be involved in pathophysiology of neuropsychiatric disorders
we demonstrate that Cdk5 (show CDK5 ELISA Kits) phosphorylates collapsin response mediator protein 2 (CRMP2 (show DPYSL2 ELISA Kits)) in the dendritic spines of cultured hippocampal neurons and in vivo in the mouse brain. When we eliminated CRMP2 (show DPYSL2 ELISA Kits) phosphorylation in CRMP2 (show DPYSL2 ELISA Kits)(KI/KI (show AXIN1 ELISA Kits)) mice, the densities of dendritic spines significantly decreased in hippocampal CA1 (show CA1 ELISA Kits) pyramidal neurons in the mouse brain.
We propose that CRMP2 (show DPYSL2 ELISA Kits) could contribute to long-lasting synaptic plasticity.
This study shows that in young adult mice, motor and sensory functions lost by SCI can be recovered with a single genetic mutation to inhibit CRMP2 (show DPYSL2 ELISA Kits) phosphorylation
Study showed that axon degeneration is an early and dominant feature in dopaminergic neurons treated with 1-methyl-4-phenylpyridiniumion (MPP+), and that the Akt/GSK-3beta/CRMP-2 pathway is involved in axon degeneration induced by MPP+
Clear differences in CRMP2 (show DPYSL2 ELISA Kits) protein abundance and degradation product/short isoform were observed between ischemic core and penumbra (show TSPAN33 ELISA Kits) and also compared to the contralateral healthy tissues after PACAP38 (show ADCYAP1 ELISA Kits) or saline treatment.
Glycogen synthase kinase 3beta has a role in microtubule remodeling in compensatory glomerular adaptation to podocyte depletion through Tau and CRMP2 (show DPYSL2 ELISA Kits)
Levels of total GSK3 were decreased in the Huntington disease (show HTT ELISA Kits)-affected frontal cortex and this correlated with decreased phosphorylated CRMP2 (show DPYSL2 ELISA Kits).
Long-term vitamin E-deficiency led to significantly increased expression of the phosphorylated form of collapsin (show SEMA3A ELISA Kits) response mediator protein (CRMP)-2 (show DPYSL2 ELISA Kits) compared to short-term deficiency.
Noncanonical kainate receptor signaling influences neuronal development by modulating CRMP2 (show DPYSL2 ELISA Kits) activity.
Among the genes found disrupted in this study, there is evidence suggesting that YWHAZ (show YWHAZ ELISA Kits) and also the X-linked DRP2 (show DPYSL2 ELISA Kits) may be considered as novel autism candidate genes.
Members of the dystrophin family of proteins perform a critical role in the maintenance of membrane-associated complexes at points of intercellular contact in vertebrate cells. The protein encoded by this gene is predicted to resemble certain short C-terminal isoforms of dystrophin and dystrophin-related protein 1 (DRP1 or utrophin). DRP2 is expressed principally in the brain and spinal cord. Two transcript variants encoding different isoforms have been found for this gene.
dystrophin related protein 2
, dystrophin-related protein 2-like
, dystrophin-related protein 2
, collapsin response mediator protein 2
, dihydropyrimidinase-related protein 2
, unc-33-like phosphoprotein 2
, dystrophin-related protein 2 A-form