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Transcription repressor. Additionally we are shipping FEZ Family Zinc Finger 1 Proteins (5) and and many more products for this protein.
Showing 10 out of 45 products:
Human Polyclonal FEZF1 Primary Antibody for EIA, WB - ABIN952301
Song, Oh, Kim, Ha, Jeong, Kim, Kim, Yoo, Kim, Kim: Human ZNF312b promotes the progression of gastric cancer by transcriptional activation of the K-ras gene. in Cancer research 2009
Show all 2 references for ABIN952301
Human Polyclonal FEZF1 Primary Antibody for IHC, WB - ABIN2784171
Strausberg, Feingold, Grouse, Derge, Klausner, Collins, Wagner, Shenmen, Schuler, Altschul, Zeeberg, Buetow, Schaefer, Bhat, Hopkins, Jordan, Moore, Max, Wang, Hsieh, Diatchenko, Marusina, Farmer et al.: Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences. ... in Proceedings of the National Academy of Sciences of the United States of America 2002
Human Polyclonal FEZF1 Primary Antibody for WB - ABIN655035
Maestrini, Pagnamenta, Lamb, Bacchelli, Sykes, Sousa, Toma, Barnby, Butler, Winchester, Scerri, Minopoli, Reichert, Cai, Buxbaum, Korvatska, Schellenberg, Dawson, de Bildt, Minderaa, Mulder, Morris, Bailey, Monaco,: High-density SNP association study and copy number variation analysis of the AUTS1 and AUTS5 loci implicate the IMMP2L-DOCK4 gene region in autism susceptibility. in Molecular psychiatry 2010
SEZ6L, HISPPD1, FEZF1, SAMD11 gene variants may be associated with autism spectrum disorder.
Fezf1 and Fezf2 (show FEZF2 Antibodies) accomplish both independent and redundant functions across diverse tissue and cell types. [Review]
Mutations in FEZF1 cause Kallmann syndrome.
the aberrant expression of ZNF312b promotes gastric tumorigenesis through epigenetic modification of its promoter region and provides a molecular mechanism for ZNF312b expression to contribute to the progression of gastric cancer.
Study is the first to report that ZNF312b, a novel transcription factor, was associated with tumorigenicity of gastric cancer.
These results identify Fezf1 and Fezf2 (show FEZF2 Antibodies) as important regulators of olfactory system development and sensory neuron identity.
Fezf1 and Fezf2 (show FEZF2 Antibodies) control differentiation of neural stem cells by repressing Hes5 (show HES5 Antibodies) and, in turn, by derepressing neurogenin 2 (show NEUROG2 Antibodies) in the forebrain.
Fez (show FEZF2 Antibodies) is involved in the axonal projection of olfactory sensory neurons.
These data indicate that Fez (show FEZF2 Antibodies) and Fezl (show FEZF2 Antibodies) repress the caudal (show CAD Antibodies) diencephalon fate in the rostral diencephalon, and ZLI formation probably depends on Fez/Fezl (show FEZF2 Antibodies)-mediated formation of diencephalon subdivisions.
These data indicate that Fezf1 is required for the penetration of olfactory axons through the CNS basal lamina before they innervate the olfactory bulb.
Transcription repressor. Involved in the axonal projection and proper termination of olfactory sensory neurons (OSN). Plays a role in rostro-caudal patterning of the diencephalon and in prethalamic formation. Expression is required in OSN to cell-autonomously regulate OSN axon projections. Regulates non-cell-autonomously the layer formation of the olfactory bulb development and the interneurons. May be required for correct rostral migration of the interneuron progenitors (By similarity).
fez family zinc finger protein 1
, zinc finger protein 312B
, FEZ family zinc finger 1
, fez family zinc finger protein 1-like
, Fez family zinc finger protein 1
, FEZ family zinc finger 2
, zinc finger protein Fez