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The protein encoded by FAM134B is a cis-Golgi transmembrane protein that may be necessary for the long-term survival of nociceptive and autonomic ganglion neurons. Additionally we are shipping FAM134B Antibodies (30) and many more products for this protein.
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The changes in JK1 DNA copy number associated with progression of colorectal neoplasms.A decrease in JK-1 copy number was correlated with a shorter disease-free survival time.
FAM134B represses cell migration in colon cancer.FAM134B has functional role in the pathogenesis of colon cancer.
Low FAM134B expression is associated with colorectal cancer.
Results show the interplay between promoter SNPs of CD39 (show ENTPD1 Proteins) and FAM134B results in an intercellular epistasis which influences the risk of a complex inflammatory disease.
Homozygous FAM134B mutation was found in Turkish siblings with hereditary sensory neuropathy type 2.
The FAM134B and TNFRSF19 (show TNFRSF19 Proteins) showed a dramatically strong synergistic epistasis in explaining the genetic dissection of the susceptibility to complex vascular dementia.
The overexpression of JK-1(FAM134b) and its transforming capacity in normal cells may play a critical role in the molecular pathogenesis of esophageal squamous cell carcinoma.
show that loss-of-function mutations in FAM134B, encoding a newly identified cis (show CISH Proteins)-Golgi protein (show GOLPH3 Proteins), cause hereditary sensory and autonomic neuropathy type II.
The protein encoded by this gene is a cis-Golgi transmembrane protein that may be necessary for the long-term survival of nociceptive and autonomic ganglion neurons. Mutations in this gene are a cause of hereditary sensory and autonomic neuropathy type IIB (HSAN IIB), and this gene may also play a role in susceptibility to vascular dementia. Alternatively spliced transcript variants encoding multiple isoforms have been observed for this gene.