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The protein encoded by FAM134B is a cis-Golgi transmembrane protein that may be necessary for the long-term survival of nociceptive and autonomic ganglion neurons. Additionally we are shipping FAM134B Proteins (5) and many more products for this protein.
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Cow (Bovine) Polyclonal FAM134B Primary Antibody for WB - ABIN2782025
Tang, Chui, Fatima, Kok, Pak, Ou, Hui, Wong, Wong, Law, Tsao, Lam, Beh, Srivastava, Chan, Ho, Tang: Oncogenic properties of a novel gene JK-1 located in chromosome 5p and its overexpression in human esophageal squamous cell carcinoma. in International journal of molecular medicine 2007
miR (show MLXIP Antibodies)-186-5p expression promotes colorectal cancer pathogenesis by regulating tumour suppressor FAM134B
FAM134B protein was noted in both cytoplasm and nuclei of colon cancer cells. In cancer cells derived from stage IV colon cancer, FAM134B expression was remarkably reduced when compared to non-cancer colon cells and cancer cells derived from stage II colon cancer. FAM134B knockdown significantly (P < 0.05) increased the proliferation of colon cancer cells following lentiviral transfection.
FAM134B mutation is common in colorectal cancer. The association of the mutation of this gene with adverse clinical and pathological parameters is congruent with the tumour suppressive properties of the gene
The changes in JK1 DNA copy number associated with progression of colorectal neoplasms.A decrease in JK-1 copy number was correlated with a shorter disease-free survival time.
FAM134B represses cell migration in colon cancer.FAM134B has functional role in the pathogenesis of colon cancer.
Low FAM134B expression is associated with colorectal cancer.
Results show the interplay between promoter SNPs of CD39 (show ENTPD1 Antibodies) and FAM134B results in an intercellular epistasis which influences the risk of a complex inflammatory disease.
Homozygous FAM134B mutation was found in Turkish siblings with hereditary sensory neuropathy type 2.
The FAM134B and TNFRSF19 (show TNFRSF19 Antibodies) showed a dramatically strong synergistic epistasis in explaining the genetic dissection of the susceptibility to complex vascular dementia.
The overexpression of JK-1(FAM134b) and its transforming capacity in normal cells may play a critical role in the molecular pathogenesis of esophageal squamous cell carcinoma.
FAM134B-dependent endoplasmic reticulum (ER)-phagy is an important limiting event in Ebola virus (EBOV) replication in mouse cells
show that loss-of-function mutations in FAM134B, encoding a newly identified cis (show CISH Antibodies)-Golgi protein (show GOLPH3 Antibodies), cause hereditary sensory and autonomic neuropathy type II.
The protein encoded by this gene is a cis-Golgi transmembrane protein that may be necessary for the long-term survival of nociceptive and autonomic ganglion neurons. Mutations in this gene are a cause of hereditary sensory and autonomic neuropathy type IIB (HSAN IIB), and this gene may also play a role in susceptibility to vascular dementia. Alternatively spliced transcript variants encoding multiple isoforms have been observed for this gene.