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binds long chain fatty acids. Additionally we are shipping Fatty Acid Binding Protein 5-Like 1 (Pseudogene) Kits (36) and Fatty Acid Binding Protein 5-Like 1 (Pseudogene) Proteins (21) and many more products for this protein.
Showing 10 out of 71 products:
Human Monoclonal FABP5L1 Primary Antibody for FACS, ICC - ABIN4899421
Liebertz, Lechner, Masood, Sinha, Han, Puri, Correa, Epstein: Establishment and characterization of a novel head and neck squamous cell carcinoma cell line USC-HN1. in Head & neck oncology 2010
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Human Polyclonal FABP5L1 Primary Antibody for WB - ABIN515491
Kovacevic, Chikhani, Lui, Sivagurunathan, Richardson: The iron-regulated metastasis suppressor NDRG1 targets NEDD4L, PTEN, and SMAD4 and inhibits the PI3K and Ras signaling pathways. in Antioxidants & redox signaling 2013
QTL and haplotype analysis revealed that both FABP4 (show FABP4 Antibodies) and FABP5 (clustered in mammals) are major candidate genes for the FAT1 (show FAT1 Antibodies) QTL; the most likely position for the FAT1 (show FAT1 Antibodies) QTL is between these two genes.
Following infection with Listeria monocytogenes, we observed similar clonal expansion, contraction and formation of memory CD8 (show CD8A Antibodies) T cells in WT and E-FABP-/- mice. Analysis of Listeria-specific CD4 (show CD4 Antibodies) T cells revealed no defect in the expansion, contraction, and formation of memory CD4 (show CD4 Antibodies) T cells in E-FABP-/- mice. Our data demonstrate that E-FABP is dispensable for antigen-specific T cell response following bacterial infection.
Genetic deletion of fatty acid-binding protein 5 (FABP5 (show FABP5 Antibodies)) in mice reduces uptake of exogenous docosahexaenoic acid (DHA) into brain endothelial cells and brain capillaries and reduces brain parenchymal levels of endogenous DHA. Therefore, FABP5 (show FABP5 Antibodies) in the brain endothelial cell is a crucial contributor to the brain levels of DHA. Critically, lowered brain DHA levels in FABP5 (show FABP5 Antibodies)(-/-) mice occurred in tandem with cognitive deficits
the balance between FABP4 (show FABP4 Antibodies) and FABP5 (show FABP5 Antibodies) in endothelial cells may be important in regulation of angiogenic versus quiescent phenotypes in blood vessels.
Mice lacking FABP5 (show FABP5 Antibodies) and FABP7 (show FABP7 Antibodies), which exhibit highest affinities for endocannabinoids, possessed elevated levels of the endocannabinoid anandamide and the related N-acylethanolamines palmitoylethanolamide and oleoylethanolamide.
These studies show, for the first time, a correlation between FABP5 (show FABP5 Antibodies) and EGFR (show EGFR Antibodies) in enhancing TNBC metastasis through a novel mechanism
loss of FABP5 (show FABP5 Antibodies) promotes a more anti-inflammatory response.
Circulating levels of GIP (show GIP Antibodies) were significantly decreased in FABP5 (show FABP5 Antibodies)-deficient mice.
The frameshift and missense mutations in FABP3 (show FABP3 Antibodies), FABP5 (show FABP5 Antibodies), and FABP7 (show FABP7 Antibodies) genes have been identified in schizophrenia and autism spectrum disorder in humans and in mouse behavioral studies.
FABP5 (show FABP5 Antibodies) contributes to the development of Diet-induced obesity in a gastric inhibitory polypeptide (show GIP Antibodies)-dependent manner.
Data indicate that epidermal fatty acid binding protein (E-FABP)-immunoreactivity was found in the entire cytoplasm and on the mitochondrial outer membrane.
The protein encoded by this gene is part of the fatty acid binding protein family (FABP). FABPs are a family of small, highly conserved, cytoplasmic proteins that bind long-chain fatty acids and other hydrophobic ligands and participate in fatty acid uptake, transport, and metabolism. In humans this gene has been associated with psoriasis and type 2 diabetes. In mouse deficiency of this gene in combination with a deficiency in Fabp4 confers protection against atherosclerosis, diet-induced obesity, insulin resistance and experimental autoimmune encephalomyelitis (the mouse model for multiple sclerosis). Alternative splicing results in multiple transcript variants that encode different protein isoforms. The mouse genome contains many pseudogenes similar to this locus.
epidermal fatty acid-binding protein
, fatty acid-binding protein, epidermal
, cutaneous fatty acid-binding protein
, epidermal-type fatty acid-binding protein
, fatty acid-binding protein 5
, epithelial fatty acid-binding protein
, keratinocyte lipid-binding protein
, psoriasis-associated fatty acid-binding protein homolog