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FCGR1A encodes a protein that plays an important role in the immune response. Additionally we are shipping Fc Fragment of IgG, High Affinity Ia, Receptor (CD64) Proteins (13) and Fc Fragment of IgG, High Affinity Ia, Receptor (CD64) Kits (1) and many more products for this protein.
Showing 10 out of 302 products:
Human Polyclonal FCGR1A Primary Antibody for FACS, IHC (p) - ABIN651123
Poon, Hulett, Parish: Histidine-rich glycoprotein is a novel plasma pattern recognition molecule that recruits IgG to facilitate necrotic cell clearance via FcgammaRI on phagocytes. in Blood 2010
Show all 3 references for ABIN651123
Human Monoclonal FCGR1A Primary Antibody for FACS - ABIN4895846
McEnaney, Fitzgerald, Zhang, Douglass, Shan, Balog, Kolesnikova, Spiegel: Chemically synthesized molecules with the targeting and effector functions of antibodies. in Journal of the American Chemical Society 2015
Show all 2 references for ABIN4895846
Human Monoclonal FCGR1A Primary Antibody for FACS - ABIN118775
Dougherty, Selvendran, Murdoch, Palmer, Hogg: The human mononuclear phagocyte high-affinity Fc receptor, FcRI, defined by a monoclonal antibody, 10.1. in European journal of immunology 1987
Show all 2 references for ABIN118775
Mouse (Murine) Monoclonal FCGR1A Primary Antibody for FACS - ABIN4897954
Xiang, Werner, Bohrmann, Liesz, Mazaheri, Capell, Feederle, Knuesel, Kleinberger, Haass: TREM2 deficiency reduces the efficacy of immunotherapeutic amyloid clearance. in EMBO molecular medicine 2016
Human Monoclonal FCGR1A Primary Antibody for FACS - ABIN4895843
Cassard, Jönsson, Arnaud, Daëron: Fcγ receptors inhibit mouse and human basophil activation. in Journal of immunology (Baltimore, Md. : 1950) 2012
Human Monoclonal FCGR1A Primary Antibody for FACS - ABIN4895845
Thomas, Moots, Edwards, Wright: Whose Gene Is It Anyway? The Effect of Preparation Purity on Neutrophil Transcriptome Studies. in PLoS ONE 2015
FCGR1A expression is significantly upregulated in human masticatory mucosa during wound healing
This study demonstrated that CD64 discriminates between critically ill patients with culture positive and negative sepsis and correlates with severity of disease. However, CD64 index is not a good predictor for 28-day mortality in the critically ill patient.
CD64 seems to be a promising marker of infection in the intensive care setting, with Leuko64 showing a slight advantage
An elevated neutrophil CD64 index was a reliable prognostic biomarker for both short-term and long-term mortality in patients admitted for acute exacerbation of Chronic obstructive pulmonary disease.
Monocyte HLA-DR and neutrophil CD64 expression in critically ill infants with sepsis are related to age and infection.
The three-dimensional structure of a human IgG1 Fc fragment bound to wild-type human Fc-gamma RI is reported
Combinatorial immunophenotypic analyses showed that loss of PTEN expression with concomitant IGFR-1 expression correlated with poor disease-free survival
Early RA patients display increased membrane and soluble CD64 and an impaired FcgammaR function correlating with joint disease activity.
CD64 index, CRP (show CRP Antibodies) and sCD14-ST served as good parameters to determine possible infection in patients that needed intensive care after major procedures.
GZMA (show GZMA Antibodies), GBP5 and CD64 genes show promise as a rapid diagnostic markers separating tuberculosis from other pulmonary diseases.
The IgG2 immune complexes activates osteoclastogenesis by binding to FcgammaRI.
Dbn1 regulates systemic anaphylaxis and IgE/Fcgr1-induced degranulation in mast cells by regulating actin reorganization and actin dynamics.
This study demonstrated that the activating FcgammaRs-mediated inflammation plays a critical role in anti-ganglioside Abs-induced neuropathy (injury to intact nerve fibers) in GBS (show GNB5 Antibodies).
N-glycans in FcgammaRIa interact with the OmpA of E. coli K1 for inducing disease pathogenesis
observations suggest that IgG-mediated inhibition of fibrocyte differentiation is mediated by FcgammaRs, with FcgammaRI mediating most of the signaling.
Inhibition of GCH1 (show GCH1 Antibodies) prevented the Escherichia coli K1 induced expression of CD64 in macrophages in vitro and the development of bacteremia in a newborn mouse model of meningitis.
The PI3K inhibitor LY294002 inhibited BCR (show BCR Antibodies)-mediated, but not TLR-mediated, induction of IkappaB-zeta (show NFKBIZ Antibodies), consistent with the role of PI3K in BCR (show BCR Antibodies) signaling and its suppression by FcgammaR.
Dap12 (show TYROBP Antibodies)- and FcRgamma (show FCER1G Antibodies)-deficiency exacerbates Granulocyte-Macrophage Colony-Stimulating Factor (show CSF2 Antibodies)-driven monocyte differentiation and production of inflammatory monocyte-derived dendritic cells.
Tpl2 (show MAP3K8 Antibodies) is activated by FcgammaR signals in macrophages and that its activation by these signals is required for ERK (show EPHB2 Antibodies) activation, cytoplasmic Ca(2 (show CA2 Antibodies)+) influx, the induction of cytokine and coreceptor gene expression, and phagocytosis.
FcgammaRI partially mediates the protective effect of monoclonal antibody TA99 on metastatic melanoma tumor loads.
This gene encodes a protein that plays an important role in the immune response. This protein is a high-affinity Fc-gamma receptor. The gene is one of three related gene family members located on chromosome 1.
Fc fragment of IgG, high affinity Ia, receptor for (CD64)
, Fc gamma receptor
, Fc-gamma RI
, Fc-gamma receptor I A1
, IgG Fc receptor I
, fc-gamma RIA
, high affinity immunoglobulin gamma Fc receptor I
, Fc fragment of IgG high affinity Ib receptor
, Fc fragment of IgG, high affinity Ib, receptor for (CD64)
, Fc receptor, IgG, high affinity I
, high affinity IgG Fc receptor, subunit beta
, high-affinity immunoglobulin gamma Fc receptor I
, IgG high affinity Fc receptor
, fc-gamma RI
, igG Fc receptor I
, FCGR1 variant 2
, FCGRI variant 1
, FCGRI variant 3