Use your antibodies-online credentials, if available.
No Products on your Comparison List.
Your basket is empty.
Find out more
GJB1 encodes a member of the gap junction protein family. Additionally we are shipping Gap Junction Protein, beta 1, 32kDa Proteins (4) and Gap Junction Protein, beta 1, 32kDa Kits (2) and many more products for this protein.
Showing 10 out of 147 products:
Human Monoclonal GJB1 Primary Antibody for IHC (fro), WB - ABIN1107368
Corcos, Lafrenière, Begy, Loch-Caruso, Willard, Glover: Refined localization of human connexin32 gene locus, GJB1, to Xq13.1. in Genomics 1992
Show all 3 references for ABIN1107368
Chicken Polyclonal GJB1 Primary Antibody for IHC, WB - ABIN2774855
Dagli, Yamasaki, Krutovskikh, Omori: Delayed liver regeneration and increased susceptibility to chemical hepatocarcinogenesis in transgenic mice expressing a dominant-negative mutant of connexin32 only in the liver. in Carcinogenesis 2004
The results of this study indicated that the Leu89Pro substitution in the second transmembrane domain of CX32 disrupts the trafficking of the protein, inhibiting the assembly of CX32 gap junctions, which in turn may result in peripheral neuropathy.
We show that the cell-surface and secreted isoforms of CSF-1 (show CSF1 Antibodies) have opposing effects on macrophage activation and disease progression in a mouse model of connexin32-deficientconnexin32-deficient mice
Results identify CSF-1 (show CSF1 Antibodies)-activated macrophages as crucial mediators of detrimental Schwann cell dedifferentiation in Cx32-deficient mice
Blockade of endothelial Cx32 increased tissue factor (show F3 Antibodies) expression induced by TNF-alpha (show TNF Antibodies) stimulation and cell-cell interaction via ICAM1 (show ICAM1 Antibodies). Direct Cx32-mediated interaction modulates TF expression in ECs during vascular inflammation.
These findings support a role for Cx32 in non-myelinating and regenerating populations of Schwann cells in normal axonal maintenance in re-myelination, and regeneration of peripheral nerve following injury
Connexin32 interacts with connexin26 (show GJB2 Antibodies) and the mitochondrial protein (show COX6B2 Antibodies), sideroflexin-1 (show SFXN1 Antibodies), at the plasma membrane forming a novel signaling nexus.
These results demonstrate that the incidence of hepatocellular carcinoma increases only in male Cx32KO mice, presumably due to enhanced tumor promotion and progression signals associated with Cx32 deficiency.
Cx32 is differentially phosphorylated and exists in a complex with SAP97 (show DLG1 Antibodies) and CaM (show Calm2 Antibodies).
Cx32 therapy improves gap junctional conductance results in larger infarct size, and no antiarrhythmic efficacy.
Connexin 36 (show GJD2 Antibodies) is expressed in beta and connexins 26 and 32 in acinar cells at the end of the secondary transition of mouse pancreatic development and increase during fetal and perinatal life.
A novel point mutation in GJB1 was detected, expanding the spectrum of GJB1 mutations known to be associated with CMTX.
Complete loss of connexin32 function is sufficient to produce central nervous system dysfunction with clinical manifestations.
This study reveals for the first time that Cx43 (show GJA1 Antibodies) and Cx32 are down-regulated in keratocystic odontogenic tumours
Transgenic expression of hCx32 in Cx32/Cx47dKO mice resulted in almost complete rescue of behavioral abnormalities in a hypomyelinating leukodystrophy model.
No mutations were found in GJB1 in a cohort of 38 Italian CMT2 patients.
Cx32 and Cx43 (show GJA1 Antibodies) mRNA expression decreased gradually during H. pylori infection-associated gastric carcinogenesis, and it is associated with hypermethylation of these genes' promoter.
findings suggest that the cytoplasmic tail of Cx32 may be involved in regulating the permeability of gap junctions by regulating their size.
This study reported four novel mutations, c.191G > A, c.508G > T, c.778A > G and c.300C > G of GJB1 in four Greek families with variable clinical features and mild clinical CNS manifestations in three of them.
Mutations in connexin 32 gene in patients with chronic rhinosinusitis, including recurrent acute rhinosinusitis, appear to be rare
intermediate invasive status of bovine trophoblast is supported by the fact that trophoblast giant cells coexpress connexins (Cx)26 (show GJB2 Antibodies), Cx32, and Cx43 (show GJA1 Antibodies)
This gene encodes a member of the gap junction protein family. The gap junction proteins are membrane-spanning proteins that assemble to form gap junction channels that facilitate the transfer of ions and small molecules between cells. According to sequence similarities at the nucleotide and amino acid levels, the gap junction proteins are divided into two categories, alpha and beta. Mutations in this gene cause X-linked Charcot-Marie-Tooth disease, an inherited peripheral neuropathy. Alternatively spliced transcript variants encoding the same protein have been found for this gene.
, connexin 32
, gap junction protein, beta 1, 32kDa
, gap junction beta-1 protein
, gap junction membrane channel protein beta 1
, GAP junction 28 kDa liver protein
, gap junction protein, beta 1, 32 kD
, gap junction protein beta 1