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Receptor for gastrin releasing peptide (GRP). Additionally we are shipping GRPR Antibodies (45) and GRPR Proteins (4) and many more products for this protein.
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ablating GRP (show UCMA ELISA Kits) receptor-expressing neurons eliminated basal and hypoxia-induced sighing, but left breathing otherwise intact initially
Together, we define the respective function of NMBR (show NMBR ELISA Kits) and GRPR in itch transmission, and reveal an unexpected relationship not only between the two receptors but also between the two populations of interneurons in itch signaling.
BNP-NPRA (show NPR1 ELISA Kits) signaling is involved in both itch and pain and does not function upstream of the GRP (show UCMA ELISA Kits)-GRPR dedicated neuronal pathway.
The present results suggest that BB2 receptor-expressing spinal neurons transmit herpes-associated itch by BB2 receptor-independent signaling.
spinal GRPr and NMBr (show NMBR ELISA Kits) independently drive itch neurotransmission in mice.
GRPR and stathmin (show STMN1 ELISA Kits) control in opposite directions both cued fear extinction and neural activities of the amygdala and prefrontal cortex
Gastrin-releasing peptide receptor mediates chemotaxis in neutrophils, and may be an alternative chemotactic receptor playing a role in the pathogenesis of inflammatory disorders.
The data suggest that opioid-induced itch is an active process concomitant with but independent of opioid analgesia, occurring via the unidirectional cross-activation of GRPR signaling by MOR1D heterodimerization
We propose that deletion of GRPR leads to the induction of depression-like behavior which is paralleled by dysregulation of amygdala gene expression, potentially resulting from deficient light-induced corticosterone release in GRPR-knockout mice
GRP-R-deficient mice showed no significant different in risk assessment behavior of conventional anxiwety parameters, so GRP-R may not impact this anxiety or emotional behavior.
GRP-R regulates glucose metabolism in neuroblastoma by modulating HIF-1alpha, PDK4 and PDP2.
BBS caused a significant increase in Shh (show SHH ELISA Kits) gene transcription and protein secretion that was dependent on BBS-induced GPCR (show NMUR1 ELISA Kits)/Galphaq (show GNAQ ELISA Kits)-//Rho mediated activation of nuclear factor kappaB (NFkappaB (show NFKB1 ELISA Kits)), which can stimulate a NF-kappaB (show NFKB1 ELISA Kits) response element in the Shh (show SHH ELISA Kits) gene promoter
No association of 16 GRP (show LSM4 ELISA Kits) and 7 GRPR variants were found with agoraphobia with/without panic disorder.
GRPR is highly expressed in epidermoid carcinoma of the anal canal, suggesting this receptor might have a role in anal carcinogenesis.
a key mechanism for GRPR-regulated colon cancer cell migration through the Galpha13-PRG (show PRB3 ELISA Kits)-RhoA (show RHOA ELISA Kits)-ROCK pathway.
GRPR expression was more pronounced in an advanced-stage lung cancer
integrin ss1 subunit critically regulates GRP-R-mediated neuroblastoma (show ARHGEF16 ELISA Kits) cell migration and invasion
Elevated buccal GRPR espression was significantly associated with squamous cell carcinoma of the head and neck.
These findings highlight the role of GRPR signaling in sepsis outcome.
Protein kinase C (show PKC ELISA Kits) is critically responsible for rapid VEGF (show VEGFA ELISA Kits) secretion by gastrin-releasing peptide receptor signaling in neuroblastoma (show ARHGEF16 ELISA Kits) cells
Receptor for gastrin releasing peptide (GRP). This receptor mediates its action by association with G proteins that activate a phosphatidylinositol-calcium second messenger system.
gastrin-releasing peptide receptor
, gastrin-releasing peptide receptor-like
, GRP-preferring bombesin receptor
, bombesin receptor