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GAD1 encodes one of several forms of glutamic acid decarboxylase, identified as a major autoantigen in insulin-dependent diabetes. Additionally we are shipping GAD Kits (32) and GAD Proteins (12) and many more products for this protein.
Showing 10 out of 177 products:
Human Polyclonal GAD Primary Antibody for WB - ABIN655175
Ruaño, Thompson, Kane, Pullinger, Windemuth, Seip, Kocherla, Holford, Wu: Physiogenomic analysis of statin-treated patients: domain-specific counter effects within the ACACB gene on low-density lipoprotein cholesterol? in Pharmacogenomics 2010
Mouse (Murine) Polyclonal GAD Primary Antibody for ELISA, WB - ABIN4313131
Wang, Wang, Ferris, Salit, Strulovici-Barel, Hackett, Crystal: Smoking-mediated up-regulation of GAD67 expression in the human airway epithelium. in Respiratory research 2010
Human Polyclonal GAD Primary Antibody for ICC, IF - ABIN314830
Valladolid-Acebes, Merino, Principato, Fole, Barbas, Lorenzo, García, Del Olmo, Ruiz-Gayo, Cano: High-fat diets induce changes in hippocampal glutamate metabolism and neurotransmission. in American journal of physiology. Endocrinology and metabolism 2012
Cow (Bovine) Polyclonal GAD Primary Antibody for WB - ABIN375070
Huang, Akbarian: GAD1 mRNA expression and DNA methylation in prefrontal cortex of subjects with schizophrenia. in PLoS ONE 2007
Human Polyclonal GAD Primary Antibody for ELISA, ICC - ABIN451637
Mouton-Liger, Sahún, Collin, Lopes Pereira, Masini, Thomas, Paly, Luilier, Même, Jouhault, Bennaï, Beloeil, Bizot, Hérault, Dierssen, Créau: Developmental molecular and functional cerebellar alterations induced by PCP4/PEP19 overexpression: implications for Down syndrome. in Neurobiology of disease 2014
our study provides novel insights into the mechanisms of UCH-L1 (show UCHL1 Antibodies)-mediated neurobiological functions and suggests that ubiquitination is an important regulatory signal for TrkB (show NTRK2 Antibodies)
These findings reveal that Th and Gad1 share a transcription regulatory mechanism that facilitates odorant-dependent regulation of dopamine and GABA expression levels.
Study describes in detail a population of choline acetyltransferase immunoreactive /glutamate decarboxylase 67 neurons predominantly ventral to the central canal of the cervical, thoracic and lumbar spinal cord of adult and juvenile mice. These cells potentially correspond to a sub-population of the cholinergic central canal cluster cells which may play a unique role in controlling spinal cord circuitry.
This study demonstrated that Alterations in hypoglossal motor neurons due to GAD67 deficiency in mice.
Treadmill running prevented partial sciatic nerve ligation-induced reductions in GAD65 (show GAD2 Antibodies)/67 production, and, thus, GABA levels may be retained in interneurons and neuropils in the superficial dorsal horn.
Effects of Ubiquitin C-Terminal Hydrolase (show UCHL1 Antibodies) L1 (UCH-L1 (show UCHL1 Antibodies)) inhibition on sperm incorporation and cortical tension in mouse eggs.
Gad1 knockdown mice have pronounced sensorimotor gating deficits, increased novelty-seeking and reduced fear extinction
The neuronal marker UCH-L1 (show UCHL1 Antibodies) is induced in, and specifically augments the oncogene (show RAB1A Antibodies)-induced transformation of, germinal center B cells.
Glutamate decarboxylase (show GLUL Antibodies) expression may be a reliable proxy of altered GABAergic transmission.
Activity deprivation due to TTX preferentially down-regulated GAD67. BDNF (show BDNF Antibodies)-induced increase in the GAD67 protein was markedly lower than that of GAD65 (show GAD2 Antibodies), indicating that BDNF (show BDNF Antibodies) differentially regulates activity-dependent gene expression of the 2 isoforms.
mutations in the two lobes affect GAD1 activation in similar ways and only intact AtCaM1 can fully activate GAD1. Taken together, our data provide new insights into the CaM (show CALM Antibodies) lobes role in interactions between CaM (show CALM Antibodies) and plant GAD.
Insertion mutants of GAD1 revealed that GABA levels in roots were drastically reduced compared with those in the wild type.
Calmodulin (show CALM Antibodies) activates Gad1 in a unique way by relieving two C-terminal autoinhibition domains of adjacent active sites, forming a 393 kDa Gad1-calmodulin (show CALM Antibodies) complex with an unusual 1:3 stoichiometry.
GAD1 is reactivated by DNA methylation (show HELLS Antibodies), which provided a model for DNA methylation (show HELLS Antibodies) and the active orchestration of oncogenic gene expression by CTCF (show CTCF Antibodies) in cancer cells.
There was no difference in GAD25 and GAD67 gene expression level, and GAD25/GAD67 ratio between patients with first episode psychosis and healthy controls
Genetic variability in GAD1 and GAD2 (show GAD2 Antibodies) contributes to the risk of methamphetamine dependence in the Thai population.
Reduced GAD67 expression in PV neurons is not an upstream cause of the lower levels of GABA-associated transcripts, or of the characteristic behaviors, in schizophrenia.
Glutamic acid decarboxylase 67 (GAD67) is one of the isoforms that catalyze GABA synthesis. Here, we used recombinant herpes simplex virus (HSV-1) vectors that encode gad1 gene to evaluate the therapeutic potential of GAD67 in peripheral HIV gp120 (show ITIH4 Antibodies)-induced neuropathic pain
This study showed that in female groups, the expression of GABRA5 (show GABRA5 Antibodies) was generally higher in schizophrenia cases compared to the control.
GAD autoantibodies were associated with risk of developing type 1 diabetes.
Conversion of GAD autoantibody is associated with patients with presumed type 2 diabetes.
The GAD65 (show GAD2 Antibodies)-independent mechanism for targeting of GAD67 to synaptic vesicles in neurons is not functional in islet beta-cells.
Gene ransfer of GAD67 by herpes simplex virus vectors prevents HIV gp120 (show ITIH4 Antibodies)/ddC (show DDC Antibodies)-induced neuropathic pain.
These results suggest that the GAD1 gene is a strong candidate gene that affects growth traits in cattle.
relative expression domains of the dlx and gad1 genes in the zebrafish telencephalon and diencephalon
High levels of GAD67 mRNA were observed in the intermediate and ventral parts of the medial pallium
the distribution of GAD67-expressing cells highly resembles the distribution of gamma-aminobutyric acid (GABA)/GAD67-expressing cells found in the early zebrafish (teleost) forebrain, suggesting a prosomeric fate map of GABAergic cell populations
This gene encodes one of several forms of glutamic acid decarboxylase, identified as a major autoantigen in insulin-dependent diabetes. The enzyme encoded is responsible for catalyzing the production of gamma-aminobutyric acid from L-glutamic acid. A pathogenic role for this enzyme has been identified in the human pancreas since it has been identified as an autoantigen and an autoreactive T cell target in insulin-dependent diabetes. This gene may also play a role in the stiff man syndrome. Deficiency in this enzyme has been shown to lead to pyridoxine dependency with seizures. Alternative splicing of this gene results in two products, the predominant 67-kD form and a less-frequent 25-kD form.
glutamate decarboxylase 1
, glutamate decarboxylase 2 (pancreatic islets and brain, 65kDa)
, glutamic acid decarboxylase 65-like protein
, 67 kDa glutamic acid decarboxylase
, GLUTAMATE DECARBOXYLASE, 67 KD ISOFORM (GAD-67) (67 KD GLUTAMIC ACID DECARBOXYLASE)
, glutamate decarboxylase 67 kDa isoform
, glutamic acid decarboxylase 1
, Glutamate decarboxylase 1 (brain)
, glutamate decarboxylase 1 variant GAD67NT
, glutamic acid decarboxylase
, glutamate decarboxylase 67
, glutamate decarboxylase, 67 kDa isoform
, gracile axonal dystrophy
, neuron cytoplasmic protein 9.5
, protein gene product 9.5
, ubiquitin carboxyl-terminal hydrolase isozyme L1
, ubiquitin thioesterase L1