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N-methyl-D-aspartate (NMDA) receptors are a class of ionotropic glutamate receptors. Additionally we are shipping GRIN2B Kits (15) and GRIN2B Proteins (9) and many more products for this protein.
Showing 10 out of 378 products:
Human Polyclonal GRIN2B Primary Antibody for IHC (fro), IP - ABIN372682
Carroll, Zukin: NMDA-receptor trafficking and targeting: implications for synaptic transmission and plasticity. in Trends in neurosciences 2002
Show all 16 references for ABIN372682
Cow (Bovine) Polyclonal GRIN2B Primary Antibody for WB - ABIN372687
Nakazawa, Komai, Tezuka, Hisatsune, Umemori, Semba, Mishina, Manabe, Yamamoto: Characterization of Fyn-mediated tyrosine phosphorylation sites on GluR epsilon 2 (NR2B) subunit of the N-methyl-D-aspartate receptor. in The Journal of biological chemistry 2001
Show all 14 references for ABIN372687
Cow (Bovine) Polyclonal GRIN2B Primary Antibody for IHC (fro), WB - ABIN372684
Itoh, Matsumoto, Inoue: Spherical aberration correction suitable for a wavefront controller. in Optics express 2009
Show all 10 references for ABIN372684
Human Polyclonal GRIN2B Primary Antibody for IHC (fro), IF - ABIN372686
Grosshans, Clayton, Coultrap, Browning: LTP leads to rapid surface expression of NMDA but not AMPA receptors in adult rat CA1. in Nature neuroscience 2001
Show all 6 references for ABIN372686
Cow (Bovine) Polyclonal GRIN2B Primary Antibody for IHC (fro), IF - ABIN372685
Wenthold, Prybylowski, Standley, Sans, Petralia: Trafficking of NMDA receptors. in Annual review of pharmacology and toxicology 2003
Show all 5 references for ABIN372685
Rat (Rattus) GRIN2B Primary Antibody for ELISA, WB - ABIN1742592
Strack, McNeill, Colbran: Mechanism and regulation of calcium/calmodulin-dependent protein kinase II targeting to the NR2B subunit of the N-methyl-D-aspartate receptor. in The Journal of biological chemistry 2000
Show all 2 references for ABIN1742592
Rat (Rattus) Polyclonal GRIN2B Primary Antibody for ELISA, WB - ABIN185658
Arning, Kraus, Valentin, Saft, Andrich, Epplen: NR2A and NR2B receptor gene variations modify age at onset in Huntington disease. in Neurogenetics 2005
Human Polyclonal GRIN2B Primary Antibody for ELISA, WB - ABIN1533143
Mandich, Schito, Bellone, Antonacci, Finelli, Rocchi, Ajmar: Mapping of the human NMDAR2B receptor subunit gene (GRIN2B) to chromosome 12p12. in Genomics 1994
Human Monoclonal GRIN2B Primary Antibody for ICC, IF - ABIN261572
Ramberger, Peschl, Schanda, Irschick, Höftberger, Deisenhammer, Rostásy, Berger, Dalmau, Reindl: Comparison of diagnostic accuracy of microscopy and flow cytometry in evaluating N-methyl-D-aspartate receptor antibodies in serum using a live cell-based assay. in PLoS ONE 2015
Dog (Canine) Polyclonal GRIN2B Primary Antibody for IHC, WB - ABIN2776121
Cais, Sedlacek, Horak, Dittert, Vyklicky: Temperature dependence of NR1/NR2B NMDA receptor channels. in Neuroscience 2008
GRIN2B polymorphisms do not influence Parkinson disease age of onset in the US Caucasian population.
GluN2B appears to exhibit allosteric regulation/inhibition by two distinct classes of antagonists; data from in silico docking suggest that GluN2B-selective antagonists broadly divide into two distinct classes according to binding pose.
Knockdown of PKD1 (show PKD1 Antibodies) did not affect NMDAR (show GRIN1 Antibodies) internalization but prevented the phosphorylation and inhibition of remaining surface NMDARs and NMDAR (show GRIN1 Antibodies)-mediated synaptic functions.
GRIN2B encodes the NR2 subunit of NMDA receptors, which are a class of ionotropic glutamate (show GRIN1 Antibodies) receptors involved in long-term potentiation, an activity-dependent increase in the efficiency of synaptic transmission.
ligand binding to cell surface GD2 induces rapid and transient activation of Src (show SRC Antibodies)-family kinases, followed by Src (show SRC Antibodies)-dependent phosphorylation of NMDA-receptor NR2B subunits selectively.
Study provides evidence of an association between GRIN2B polymorphisms and Schizophrenia susceptibility and symptoms in the Han Chinese population.
GFAP peaks early during haemorrhagic brain lesions (at significantly higher levels), and late in ischaemic events, whereas antibodies against NR2 RNMDA have significantly higher levels during ischemic stroke at all time-points.
Polymorphisms in DRD1 (show DRD1 Antibodies), DRD2 (show DRD2 Antibodies) and GRIN2B confer increased risk of impulse control problems among PD patients.
BDNF (show BDNF Antibodies), APOE (show APOE Antibodies), and HSP70-1 (show HSPA1A Antibodies) genes, but not GRIN2B, might be associated with a risk of POAG occurrence in the Polish population
This study provides further evidence in support of GRIN2B contributing toward Developmental Dyslexia and deficits in Developmental Dyslexia
The results suggest that NR2B vulnerability represents a target for environmental toxicants in the brain.
Results show that monomeric Abeta1-42 application induces an increase of the Ca2 (show CA2 Antibodies)+-response and of the membrane expression of the extrasynaptic subunit of the NMDA receptor GluN2B in PC12 cells, while the opposite effects were observed in cultured neurons
Results demonstrate that up-regulation of NR2B expression facilitates acquisition of auditory cued fear memory and enhances long-term potentiation at thalamus-lateral amygdala synapses
Results suggest that EGFR (show EGFR Antibodies) signaling upregulates NMDA receptors through modification of the GluN2B subunit, and is required for high-frequency stimulation-induced long-term potentiation in the hippocampus
Loss of GluN2B in principal neurons of the forebrain results in reduced punished food reward-seeking.
The sigma-1R deficiency through suppressing NR2B function and DAT (show SLC6A3 Antibodies) expression can reduce MPTP (show PTPN2 Antibodies)-induced death of dopaminergic neurons and parkinsonism.
Study demonstrated that the GluN2B carboxy-terminal domain is necessary for enhanced social recognition memory in GluN2B transgenic overexpression mice
Homer1a (show HOMER1 Antibodies) may be a key neuroprotective endogenous molecule that protects against NMDA-induced neuronal injury by disassembling NR2B-PSD95 (show DLG4 Antibodies)-nNOS (show NOS1 Antibodies) complexes and reducing the membrane distribution of NR2B.
Findings reveal an intimate reciprocal relationship between ErbB4 (show ERBB4 Antibodies) and GluN2b NMDA receptors via NRG2 (show NRG2 Antibodies)/ErbB4 (show ERBB4 Antibodies) signaling.
Visual plasticity in adult mice was dramatically enhanced following magnesium treatment, which was concurrent with an increase in the expression of NR2 subunits of N-methyl-D-aspartate receptors.
N-methyl-D-aspartate (NMDA) receptors are a class of ionotropic glutamate receptors. NMDA receptor channel has been shown to be involved in long-term potentiation, an activity-dependent increase in the efficiency of synaptic transmission thought to underlie certain kinds of memory and learning. NMDA receptor channels are heteromers composed of three different subunits: NR1 (GRIN1), NR2 (GRIN2A, GRIN2B, GRIN2C, or GRIN2D) and NR3 (GRIN3A or GRIN3B). The NR2 subunit acts as the agonist binding site for glutamate. This receptor is the predominant excitatory neurotransmitter receptor in the mammalian brain.
NMDA type glutamate receptor
, N-methyl D-aspartate receptor subtype 2B
, N-methyl-D-aspartate receptor subunit 2B
, glutamate [NMDA] receptor subunit epsilon-2
, glutamate receptor ionotropic, NMDA 2B
, N-methyl-D-aspartate receptor subunit 3
, glutamate receptor subunit epsilon-2
, glutamate receptor, ionotropic, NMDA2B
, N-methyl-D-aspartate receptor subunit NR2B
, N-methyl D-aspartate receptor 2B