You are viewing an incomplete version of our website. Please click to reload the website as full version.

Hairy and Enhancer of Split 5 (Drosophila) (HES5) ELISA Kits

HES5 encodes a member of a family of basic helix-loop-helix transcriptional repressors. Additionally we are shipping HES5 Antibodies (37) and HES5 Proteins (5) and many more products for this protein.

list all ELISA KIts Gene Name GeneID UniProt
Anti-Human HES5 HES5 388585 Q5TA89
Anti-Mouse HES5 HES5 15208 P70120
Anti-Rat HES5 HES5 79225 Q03062
How to order from antibodies-online
  • +1 877 302 8632
  • +1 888 205 9894 (toll-free)
  • Order online
  • orders@antibodies-online.com

More ELISA Kits for HES5 Interaction Partners

Zebrafish Hairy and Enhancer of Split 5 (Drosophila) (HES5) interaction partners

  1. Over-expression of her12, her15 or her7 disrupts cyclic gene expression and somite border formation, and structure function analysis of Her7 indicates that DNA binding, but not Groucho-recruitment seems to be important in this process.

Human Hairy and Enhancer of Split 5 (Drosophila) (HES5) interaction partners

  1. that suppression of the HES5 leading to inhibition of proliferation may be one of the mechanisms against Hepatocellular carcinoma

  2. HES5 silencing is an early and recurrent change in prostate tumourigenesis

  3. these data identify HES5 as a key mediator of the Wnt-3a (show WNT3A ELISA Kits) proneurogenic effect occurring independently of the classical Wnt (show WNT2 ELISA Kits)/beta-catenin (show CTNNB1 ELISA Kits) signaling cascade thus further deciphering crosstalk mechanisms of Wnt (show WNT2 ELISA Kits) and Notch (show NOTCH1 ELISA Kits) signaling pathways regulating cell fate

  4. the downstream Notch (show NOTCH1 ELISA Kits) signalling effector HES5 directly represses transcription of the E3 ligase Fbw7beta.

  5. Notch3 (show NOTCH3 ELISA Kits) and Hes5 are hypermethylated in human B cell acute lymphoblastic leukemia (ALL).

  6. Hes5 overactivation is associated with cell differentiation, thereby resulting in uterine carcinogenesis.

  7. In this study, HES 5 transcription factor was detected in the lining epithelium of human periapical cysts with limited inflammation, showing Notch (show NOTCH1 ELISA Kits) pathway activation in those cells.

  8. The expression of HES5 in adenocarcinoma was significantly higher than those in adenoma and normal control

  9. For marking and purifying neural stem cells to ascertain whether differences exist, we generated transgenic mice using promoters from Hes (show RRBP1 ELISA Kits) genes (pHes1 or pHes5) to drive expression of destabilized enhanced green fluorescent protein.

  10. Notch protein binding to Hes5-GFP is extinguished fast and recovered slowly, whereas Hes1-GFP is inhibited late and recovered quickly

Mouse (Murine) Hairy and Enhancer of Split 5 (Drosophila) (HES5) interaction partners

  1. This study demonstrated that Hes1 and Hes5 modulate not only maintenance of progenitor cells but also pituicyte versus neuron fate specification during neurohypophysis development.

  2. delivery of siRNA to Hes1 and Hes5 using a transfection reagent or siRNA to Hes1 encapsulated within nanoparticles increased hair cell numbers in non-toxin treated organotypic cultures of cochleae and maculae

  3. The transcription and expression patterns of Notch pathway components (Notch 1-3, Delta1 and 4, Jagged1) and effectors (Hes1, Hes2, Hes5 and Nrarp) were evaluated in the mouse testis

  4. Hes1 and Hes5 regulate vascular remodeling and arterial fate specification of endothelial cells in the development of the brain; they are critical transducers of Notch (show NOTCH1 ELISA Kits) signals in brain vascular development.

  5. the downstream Notch (show NOTCH1 ELISA Kits) signalling effector HES5 directly represses transcription of the E3 ligase Fbw7beta.

  6. Data demonstrate that Hes5 levels in the utricle decreased after the application of siRNA and that the number of hair cells in these utricles was significantly larger than following control treatment.

  7. Epigenetic silencing of Hes5 leads to neuronal differentiation despite active Notch (show NOTCH1 ELISA Kits) signaling.

  8. Simultaneous overexpression of Hes5 and Sox21 (show SOX21 ELISA Kits) reveals Hes5 as a downstream effector of Sox21 (show SOX21 ELISA Kits) in adult hippocampus neurogenesis.

  9. This study suggested that Hes5 expression is serially activated first by Gcms and later by the canonical Notch (show NOTCH1 ELISA Kits) pathway

  10. Hes1, Hes5 and Hey1 (show HEY1 ELISA Kits) cooperatively inhibit hair cell formation, and one allele of Hes1, Hes5 or Hey1 (show HEY1 ELISA Kits) is sufficient for supporting cell (show PTPRJ ELISA Kits) production probably by lateral inhibition in the sensory epithelium.

HES5 Antigen Profile

Antigen Summary

This gene encodes a member of a family of basic helix-loop-helix transcriptional repressors. The protein product of this gene, which is activated downstream of the Notch pathway, regulates cell differentiation in multiple tissues. Disruptions in the normal expression of this gene have been associated with developmental diseases and cancer.

Gene names and symbols associated with HES5

  • hairy and enhancer of split-related 15.1 (her15.1) antibody
  • hairy and enhancer of split 5 (Drosophila) (HES5) antibody
  • hairy and enhancer of split 5 (Drosophila) (hes5) antibody
  • hairy and enhancer of split 5 (LOC100221262) antibody
  • hairy and enhancer of split 5 (LOC100226434) antibody
  • hairy and enhancer of split 5 (Drosophila) (Hes5) antibody
  • bHLHb38 antibody
  • fk51a02 antibody
  • her15 antibody
  • hes5 antibody
  • wu:fk51a02 antibody
  • zgc:136520 antibody

Protein level used designations for HES5

hairy and enhancer of split 5 , her15a , transcription factor HES-5 , Transcription factor HES-5 , class B basic helix-loop-helix protein 38

GENE ID SPECIES
359836 Danio rerio
419392 Gallus gallus
787633 Bos taurus
100195635 Salmo salar
100221262 Taeniopygia guttata
100226434 Taeniopygia guttata
388585 Homo sapiens
15208 Mus musculus
79225 Rattus norvegicus
Did you look for something else?