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IGKV1-27 was identified as a gene whose expression is rapidly induced by the tumor necrosis factor (TNF).
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Human Monoclonal IGKV1-27 Primary Antibody for WB - ABIN2668824
Wu, Xu, Chang: The promyelocytic leukemia protein represses A20-mediated transcription. in The Journal of biological chemistry 2002
A20 (show TNFAIP3 Antibodies) acts as a regulator of the ectodysplasin-A1 (show EDA Antibodies) pathway to NF-kappaB (show NFKB1 Antibodies) activation in primary Sjogren's syndrome salivary glands epithelial cells
knockdown of A20 (show TNFAIP3 Antibodies) in tumor site inhibited tumor growth at least through inducing the apoptosis of MDSCs. A20 (show TNFAIP3 Antibodies) might be a potential target in anticancer therapy.
A20 targets caspase-8 and FADD to protect HTLV-I-infected cells.
Recruitment of A20 (show TNFAIP3 Antibodies) to the C-terminal domain of NEMO (show IKBKG Antibodies) represents a novel mechanism limiting NF-kappaB (show NFKB1 Antibodies) activation by NEMO (show IKBKG Antibodies), and its absence results in autoinflammatory disease.
Our findings indicate that a genetic variant of TNFAIP3 (show TNFAIP3 Antibodies) is strongly associated with the silica-induced profibrotic response of fibroblasts.
There was no significant difference in PFS and OS in patients with or without A20 (show TNFAIP3 Antibodies) mutations.
Increased A20 (show TNFAIP3 Antibodies) expression is associated with cholangiocarcinoma.
FOXP1 (show FOXP1 Antibodies), TP53INP1 (show TP53INP1 Antibodies), TNFAIP3 (show TNFAIP3 Antibodies), and TUSC2 (show TUSC2 Antibodies) were identified as miR (show MLXIP Antibodies)-19a targets.
Loss-of-function mutations in TNFAIP3 (show TNFAIP3 Antibodies) leading to A20 (show TNFAIP3 Antibodies) haploinsufficiency cause an early-onset autoinflammatory disease.
Hepatitis C virus antigen stimulation up-regulates A20/A20 (show TNFAIP3 Antibodies)-binding inhibitor of NF-kappaB (show NFKB1 Antibodies) binding protein expression, which consequently contributes to inefficient M1 macrophage polarization.
This gene was identified as a gene whose expression is rapidly induced by the tumor necrosis factor (TNF). The protein encoded by this gene is a zinc finger protein and ubiqitin-editing enzyme, and has been shown to inhibit NF-kappa B activation as well as TNF-mediated apoptosis. The encoded protein, which has both ubiquitin ligase and deubiquitinase activities, is involved in the cytokine-mediated immune and inflammatory responses. Several transcript variants encoding the same protein have been found for this gene.
OTU domain-containing protein 7C
, TNF alpha-induced protein 3
, putative DNA-binding protein A20
, tumor necrosis factor alpha-induced protein 3
, tumor necrosis factor inducible protein A20
, zinc finger protein A20