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IFI6 was first identified as one of the many genes induced by interferon. Additionally we are shipping IFI6 Kits (4) and IFI6 Proteins (3) and many more products for this protein.
Showing 10 out of 48 products:
Human Monoclonal IFI6 Primary Antibody for ELISA - ABIN393218
Johnatty, Beesley, Chen, Macgregor, Duffy, Spurdle, deFazio, Gava, Webb, Rossing, Doherty, Goodman, Lurie, Thompson, Wilkens, Ness, Moysich, Chang-Claude, Wang-Gohrke, Cramer, Terry, Hankinson, Tworoger, Garcia-Closas, Yang, Lissowska, Chanock, Pharoah, S: Evaluation of candidate stromal epithelial cross-talk genes identifies association between risk of serous ovarian cancer and TERT, a cancer susceptibility "hot-spot". in PLoS genetics 2010
Show all 5 references for ABIN393218
Human Polyclonal IFI6 Primary Antibody for EIA, WB - ABIN952822
Szegedi, Sonkoly, Nagy, Németh, Bata-Csörgo, Kemény, Dobozy, Széll: The anti-apoptotic protein G1P3 is overexpressed in psoriasis and regulated by the non-coding RNA, PRINS. in Experimental dermatology 2010
Show all 2 references for ABIN952822
Human Polyclonal IFI6 Primary Antibody for WB - ABIN656889
Davila, Froeling, Tan, Bonnard, Boland, Snippe, Hibberd, Seielstad: New genetic associations detected in a host response study to hepatitis B vaccine. in Genes and immunity 2010
In conclusion, over-expression of IFI6 promotes hepatitis C virus RNA replication and rescues the interferon alpha (show IFNA Antibodies)-mediated anti-hepatitis C virus activity.
IFI6 inhibits HCV entry by impairing EGFR (show EGFR Antibodies) mediated CD81 (show CD81 Antibodies)/CLDN1 (show CLDN1 Antibodies) interactions. This may be relevant to other virus entry processes employing EGFR (show EGFR Antibodies).
Here we elucidate G1P3, a survival protein induced by interferons (IFNs), as a target of estrogen signaling and a contributor to poor outcomes in estrogen receptor (show ESR1 Antibodies)-positive (ER(+)) breast cancer.
PRINS regulates G1P3, a gene with anti-apoptotic effects in keratinocytes. siRNA-mediated inhibition of PRINS gene resulted in altered cell morphology and gene expression alterations.
G1P3 protein may have function as a cell survival protein by inhibiting mitochondrial-mediated apoptosis
Respiratory syncytial virus upregulated the mRNA expression of chemokines CC and CXC and interfered with type alpha/beta interferon (show IFNA Antibodies)-inducible gene expression by upregulation of MG11 (show SAMHD1 Antibodies) and downregulation of G1P3.
IFN-alpha2b (show ADRA2B Antibodies) induced the expression of G1P3 and antagonized the TRAIL induced apoptosis in myeloma suggesting that either the deregulated or the induced expression of G1P3 could lead to apoptosis resistance in tumor cells.
This gene was first identified as one of the many genes induced by interferon. The encoded protein may play a critical role in the regulation of apoptosis. A minisatellite that consists of 26 repeats of a 12 nucleotide repeating element resembling the mammalian splice donor consensus sequence begins near the end of the second exon. Alternatively spliced transcript variants that encode different isoforms by using the two downstream repeat units as splice donor sites have been described.
interferon alpha-inducible protein 6
, interferon, alpha-inducible protein clone IFI-6-16
, interferon-induced protein 6-16