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The protein encoded by IL1RL1 is a member of the interleukin 1 receptor family. Additionally we are shipping IL1RL1 Antibodies (81) and IL1RL1 Kits (23) and many more products for this protein.
Showing 10 out of 24 products:
Human IL1RL1 Protein expressed in HEK-293 - ABIN2666553
Demyanets, Kaun, Pentz, Krychtiuk, Rauscher, Pfaffenberger, Zuckermann, Aliabadi, Gröger, Maurer, Huber, Wojta: Components of the interleukin-33/ST2 system are differentially expressed and regulated in human cardiac cells and in cells of the cardiac vasculature. in Journal of molecular and cellular cardiology 2013
Show all 5 references for ABIN2666553
Mouse (Murine) IL1RL1 Protein expressed in Human Cells - ABIN2008684
Schmitz, Owyang, Oldham, Song, Murphy, McClanahan, Zurawski, Moshrefi, Qin, Li, Gorman, Bazan, Kastelein: IL-33, an interleukin-1-like cytokine that signals via the IL-1 receptor-related protein ST2 and induces T helper type 2-associated cytokines. in Immunity 2005
Show all 5 references for ABIN2008684
Human IL1RL1 Protein expressed in Human Cells - ABIN2002075
Trajkovic, Sweet, Xu: T1/ST2--an IL-1 receptor-like modulator of immune responses. in Cytokine & growth factor reviews 2004
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Human IL1RL1 Protein expressed in Human Cells - ABIN2002071
Hayakawa, Hayakawa, Kume, Tominaga: Soluble ST2 blocks interleukin-33 signaling in allergic airway inflammation. in The Journal of biological chemistry 2007
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Human IL1RL1 Protein expressed in HEK-293 Cells - ABIN2181368
Tago, Noda, Hayakawa, Iwahana, Yanagisawa, Yashiro, Tominaga: Tissue distribution and subcellular localization of a variant form of the human ST2 gene product, ST2V. in Biochemical and biophysical research communications 2001
Show all 2 references for ABIN2181368
ST2 deletion increases inflammatory bone loss in experimental periapical lesions in mice.
ST2 receptor invalidation maintains wound inflammation, delays healing and increases fibrosis
the results of the present study suggest a possible asthma phenotype that involves the IL-33 (show IL33 Proteins)/ST2 (show SULT2A1 Proteins) pathway and is important for the development of airway inflammation and AHR (show AHR Proteins) in the peripheral airways.
Data suggest that IL-33 and ST2 function as a developmental switch to license thermogenesis during the perinatal period.
The IL-33/ST2 axis may play a crucial role in the pathogenesis of angiostrongylosis.
Natural helper cells contribute to pulmonary eosinophilia by producing IL-13 via IL-33/ST2 pathway in a murine model of respiratory syncytial virus infection
Blockade of ST2 (show SULT2A1 Proteins) markedly improves survival of lymphocytic choriomeningitis virus -infected Prf1 (show PRF1 Proteins)-/- mice and reduces the severity of multiple disease parameters, including serum levels of IFNgamma.
the IL-33/ST2 axis specifically controls visceral adipose tissue-Treg cell development was revealed.
The activity of IL-33 at its receptor ST2 is terminated by the formation of two disulphide bridges, resulting in conformational change that disrupts the ST2 binding site.
IL-1beta (show IL1B Proteins)-induced ST2L (show TMED1 Proteins) expression suppressed the responsiveness of rapamycin-dendritic cells to TLR or CD40 (show CD40 Proteins) ligation
IL-33 and ST2 are expressed in periapical granulomas and radicular cysts.
in breast tumors from 40 female patients with absent or present tumor necrosis there was significantly higher expression of IL-33, IL-33R and VEGF in breast cancer tissues with absent tumor necrosis. Both, IL-33 and IL-33R expression correlated with VEGF expression in tumor cells.
An hyperexpression of soluble ST2 was found in Sjogren syndrome patients.
ST2 is associated with advanced and metastatic disease in GC patients and significantly correlates with the duration of the disease.
In patients with endometriosis, ST2 (show SULT2A1 Proteins) was positively associated with VAS (show AVP Proteins) score.
two new polymorphisms in the distal promoter region of the ST2 gene that possibly influence susceptibility to severe coronary artery disease
IL-33 and ST2 can be detected in lysates from both normal and pre-eclampsia placentas
in an unselected cohort of patients admitted to the ICU, sST2 (show SSTR2 Proteins) was an independent predictor of 90-day all-cause mortality
The pro-T helper (Th)2 cell effects of mature IL-33 are due to differential utilization of the interleukin (IL)-33 receptor chain ST2, whereas their similar effects result from regulation of gene expression.
Expression of IL-33 receptor ST2 in human adipose tissue is increased by severe obesity indicating an autocrine action. Thus, the adipose tissue microvasculature could participate in obesity-associated inflammation and related complications via IL-33/ST2.
The protein encoded by this gene is a member of the interleukin 1 receptor family. Studies of the similar gene in mouse suggested that this receptor can be induced by proinflammatory stimuli, and may be involved in the function of helper T cells. This gene, interleukin 1 receptor, type I (IL1R1), interleukin 1 receptor, type II (IL1R2) and interleukin 1 receptor-like 2 (IL1RL2) form a cytokine receptor gene cluster in a region mapped to chromosome 2q12. Alternative splicing of this gene results in multiple transcript variants.
, interleukin-1 receptor-like 1
, lymphocyte antigen 84
, protein T1
, growth stimulation-expressed
, homolog of mouse growth stimulation-expressed
, interleukin 1 receptor-related protein
, fos-responsive gene 1 protein