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The protein encoded by IL23R is a subunit of the receptor for IL23A/IL23. Additionally we are shipping Interleukin 23 Receptor Proteins (13) and Interleukin 23 Receptor Kits (11) and many more products for this protein.
Showing 10 out of 122 products:
Human Monoclonal IL23R Primary Antibody for WB - ABIN393707
Bailey, Xie, Do, Montpetit, Diaz, Mohan, Keavney, Yusuf, Gerstein, Engert, Anand: Variation at the NFATC2 locus increases the risk of thiazolidinedione-induced edema in the Diabetes REduction Assessment with ramipril and rosiglitazone Medication (DREAM) study. in Diabetes Care 2010
Show all 5 references for ABIN393707
Human Polyclonal IL23R Primary Antibody for IF (p), IHC (p) - ABIN686857
Wang, Zhong, Zheng, Li, Chen, Ma, Sun, Yan, Li: Damage effect of interleukin (IL)-23 on oxygen-glucose-deprived cells of the neurovascular unit via IL-23 receptor. in Neuroscience 2015
Four SNPs in IFNGR2 (show IFNGR2 Antibodies), IL12RB1 (show IL12RB1 Antibodies), IL12RB2 (show IL12RB2 Antibodies), and IL23R were found to be associated with the MAP infection status of the resource population.
In this study, we were unable to establish a correlation between the IL-23R SNPs investigated and HLA-B27-associated acute anterior uveitis as well as idiopathic intermediate uveitis.
These results suggest that IL23R may contribute to the development of intracerebral hemorrhage.
The association of IL-23R and Ankylosing Spondylitis (AS) that is seen in Caucasian patients with AS is not present in Chinese patients with AS.
Differential splicing generates antagonistic soluble IL-23R (sIL (show PMEL Antibodies)-23R) variants, which might limit IL-23 (show IL23A Antibodies)-mediated immune responses. Here, ectodomain shedding of IL-23R was identified as an alternative pathway for the generation of sIL (show PMEL Antibodies)-23R.
The IL23R polymorphisms rs10889677, rs7517847, and the IL12B (show IL12B Antibodies) polymorphism rs3212227 are not associated with multiple sclerosis risk.
meta-analysis supports that two polymorphisms (rs11209026 and rs7517847) in the IL-23 (show IL23A Antibodies) gene may be considered to be protective factors against developing UC among Caucasian populations.
The results of the current study disclosers1343151 variant of IL23R as a susceptibility gene in CD.
Lower LCN2 (show LCN2 Antibodies) levels in Crohn's Disease patients carrying IBD risk-increasing IL23R variants may result from a restricted upregulation of LCN2 (show LCN2 Antibodies) due to an impaired Th17 immune response
IL23 receptor single nucleotide polymorphisms and gene copy number variation are associated with susceptibility to pulmonary tuberculosis in Chinese Uygurs.
These results indicated an association between the rs11209026 G>A polymorphism of the IL-23 receptor gene and the risk of atherosclerosis.
Differential splicing generates antagonistic soluble IL-23R (sIL (show STIL Antibodies)-23R) variants, which might limit IL-23 (show IL23A Antibodies)-mediated immune responses. Here, ectodomain shedding of IL-23R was identified as an alternative pathway for the generation of sIL (show STIL Antibodies)-23R.
Estrogen and progesterone decrease let-7f microRNA expression and increase IL-23/IL-23 (show IL23A Antibodies) receptor signaling and IL-17A (show IL17A Antibodies) production in patients with severe asthma.
Data show the contribution of IL-23/IL-23 (show IL23A Antibodies) receptor and IL-7/IL-7 (show IL7 Antibodies) receptor signaling in Th17 and Th1 (show HAND1 Antibodies) cell dynamics during experimental autoimmune encephalomyelitis (EAE).
Study provides evidence that IL-23 (show IL23A Antibodies)/IL-23R plays a critical role in brain ischemic injury
IL-23 (show IL23A Antibodies) plus T-cell receptor signalling results in significant upregulation of IL-23 receptor expressed predominantly on CD4 (show CD4 Antibodies)(hi)CD8 (show CD8A Antibodies)(hi)CD3 (show CD3E Antibodies)(+)alphabetaTCR(+) thymocytes, and leads to RORgammat-dependent apoptosis.
Homeostatic IL-23 receptor signaling limits Th17 response through IL-22 (show IL22 Antibodies)-mediated containment of commensal microbiota.
Characterization of potent IL-23R small-peptide modulator, 2305 (teeeqqly), that decreases inflammatory response.
Although they share a common subunit, IL-23 (show IL23A Antibodies) and IL-12 (show IL12A Antibodies) receptors are not expressed on the same cell populations
IL-23R+ innate lymphoid cells induce colitis via interleukin-22 (show IL22 Antibodies)-dependent mechanism.
the balance between the enhancing and inhibitory effects of gamma-delta T cells is regulated by their level of IL-23R expression.
The protein encoded by this gene is a subunit of the receptor for IL23A/IL23. This protein pairs with the receptor molecule IL12RB1/IL12Rbeta1, and both are required for IL23A signaling. This protein associates constitutively with Janus kinase 2 (JAK2), and also binds to transcription activator STAT3 in a ligand-dependent manner.
, interleukin 23 receptor
, interleukin-23 receptor-like
, IL-23 receptor
, interleukin 23 receptor-like