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IL33 (MIM 608678) is a member of the IL1 (see MIM 147760) family that potently drives production of T helper-2 (Th2)-associated cytokines (e.g., IL4; MIM 147780). Additionally we are shipping Interleukin 33 Antibodies (369) and Interleukin 33 Proteins (107) and many more products for this protein.
Showing 10 out of 59 products:
Human Interleukin 33 ELISA Kit for Sandwich ELISA - ABIN415121
Guzel, Serin, Guzel, Buyuk, Y?lmaz, Güvenen: Interleukin-33, matrix metalloproteinase-9, and tissue inhibitor [corrected] of matrix metalloproteinase-1 in myocardial infarction. in The Korean journal of internal medicine 2013
Show all 3 references for ABIN415121
Rat (Rattus) Interleukin 33 ELISA Kit for Sandwich ELISA - ABIN578202
Demirtas, Turkmen, Kandemir, Ozkaraca, Kucukler, Gürbüzel, Comaklı: The possible role of interleukin-33 as a new player in the pathogenesis of contrast-induced nephropathy in diabetic rats. in Renal failure 2016
Mouse (Murine) Interleukin 33 ELISA Kit for Sandwich ELISA - ABIN424266
Wu, Guo, Ding, Lv, Bi: CXCL13 blockade attenuates lupus nephritis of MRL/lpr mice. in Acta histochemica 2015
This study suggests a possible systemic role of IL-33 in the pathogenesis of vitiligo (show MITF ELISA Kits).
IL-33 expression may serve as an independent negative predictor of survival for patients with clear-cell renal cell carcinoma after surgery.
Serum upregulated IL-33 could contribute to the pathogenesis of chronic allograft dysfunction in kidney transplant recipients.
No significant differences in genotypic and allelic frequencies of the rs3939286 in interleukin 33 was observed between cases and controls and between mild/severe and early/late-onset Preeclampsia.
Reduced IL-33 levels and rs16924159 IL-33 variant may contribute to the pathogenesis of idiopathic recurrent miscarriage in Chinese Han population.
In a CML (show BCR ELISA Kits) patient with pruritus receiving imatinib mesylate, IL-31 (show IL31 ELISA Kits) and IL-33 serum levels were significantly higher than controls. Imatinib could cause keratinocyte injury, the release of IL-33, and the consequent interaction with its receptor on mast cells, inducing IL-31 (show IL31 ELISA Kits) secretion. The IL-31 (show IL31 ELISA Kits)/IL-33 axis may be involved in the pathogenesis of skin side effects related to imatinib mesylate treatment.
IL-33 and ST2 are expressed in periapical granulomas and radicular cysts.
in breast tumors from 40 female patients with absent or present tumor necrosis there was significantly higher expression of IL-33, IL-33R and VEGF in breast cancer tissues with absent tumor necrosis. Both, IL-33 and IL-33R expression correlated with VEGF expression in tumor cells.
Data suggest that interleukin 33 (IL-33) may play an important role in the development of non-small cell lung cancer (NSCLC) and the targeted therapy.
rs7044343 polymorphism of IL-33 gene is associated with the susceptibility to systemic sclerosis in Turkish population.
anti-allergic effect of anti-IL-33 antibodies associated with suppression of immunoglobulin free light chain and nitric oxide synthase (show NOS ELISA Kits) in a murine model of allergic rhinitis
Data suggest that IL-33 and ST2 function as a developmental switch to license thermogenesis during the perinatal period.
some SNPs of ST2-IL18R1-IL18RAP gene cluster might increase the risk of susceptibility of Graves' disease (GD) and Hashimoto's thyroiditis (HT) in Chinese Han population.
Furthermore, IL-33 injection modulates the innate immune response by polarizing microglia/macrophages toward an antiinflammatory phenotype and reducing the expression of proinflammatory genes, including IL-1beta (show IL1B ELISA Kits), IL-6 (show IL6 ELISA Kits), and NLRP3 (show NLRP3 ELISA Kits), in the cortices of APP (show APP ELISA Kits)/PS1 (show PSEN1 ELISA Kits) mice. Collectively, our results demonstrate a potential therapeutic role for IL-33 in AD.
absence of IL-33 has no negative consequences in normal bone homeostasis while high levels of circulating IL-33 contributes to pathological bone loss.
pre-treatment with the ERK1/2 (show MAPK1/3 ELISA Kits) inhibitor U0126 or anti-IL-33 antibody significantly abolished the ovalbumin (show OVA ELISA Kits)-induced airway remodeling, increased expressions of IL-33
IL-33 promotes degranulation of bone marrow-derived mast cells and release of cytokines IL-1beta (show IL1B ELISA Kits), IL-6 (show IL6 ELISA Kits) and TNF-alpha (show TNF ELISA Kits).
pulmonary interstitial macrophages may not be a source of IL-33 during Respiratory syncytial virus infection
demonstrate a pathogenic role for IL-33-mediated eosinophilia and activation of Th2 immunity in chronic intestinal inflammation
IL-33 as a novel regulator of neutrophil function in antibacterial infection
IL-33 may be a key molecule operating in eosinophil-mediated fibrosis in high-dose-per fraction irradiated skin.
IL33 (MIM 608678) is a member of the IL1 (see MIM 147760) family that potently drives production of T helper-2 (Th2)-associated cytokines (e.g., IL4\; MIM 147780). IL33 is a ligand for IL33R (IL1RL1\; MIM 601203), an IL1 family receptor that is selectively expressed on Th2 cells and mast cells (summary by Yagami et al., 2010
, DVS27-related protein
, interleukin-1 family member 11
, nuclear factor for high endothelial venules
, nuclear factor from high endothelial venules