Use your antibodies-online credentials, if available.
No Products on your Comparison List.
Your basket is empty.
Find out more
IL33 (MIM 608678) is a member of the IL1 (see MIM 147760) family that potently drives production of T helper-2 (Th2)-associated cytokines (e.g., IL4; MIM 147780). Additionally we are shipping Interleukin 33 Antibodies (364) and Interleukin 33 Proteins (108) and many more products for this protein.
Showing 10 out of 78 products:
Mouse (Murine) Interleukin 33 ELISA Kit for Sandwich ELISA - ABIN1672844
Bonilla, Fröhlich, Senn, Kallert, Fernandez, Johnson, Kreutzfeldt, Hegazy, Schrick, Fallon, Klemenz, Nakae, Adler, Merkler, Löhning, Pinschewer: The alarmin interleukin-33 drives protective antiviral CD8⁺ T cell responses. in Science (New York, N.Y.) 2012
Show all 2 references for ABIN1672844
Human Interleukin 33 ELISA Kit for Sandwich ELISA - ABIN1672843
Schmitz, Owyang, Oldham, Song, Murphy, McClanahan, Zurawski, Moshrefi, Qin, Li, Gorman, Bazan, Kastelein: IL-33, an interleukin-1-like cytokine that signals via the IL-1 receptor-related protein ST2 and induces T helper type 2-associated cytokines. in Immunity 2005
Show all 2 references for ABIN1672843
Rat (Rattus) Interleukin 33 ELISA Kit for Sandwich ELISA - ABIN578202
Demirtas, Turkmen, Kandemir, Ozkaraca, Kucukler, Gürbüzel, Comaklı: The possible role of interleukin-33 as a new player in the pathogenesis of contrast-induced nephropathy in diabetic rats. in Renal failure 2016
IL-33-ERK (show EPHB2 ELISA Kits)/JNK (show MAPK8 ELISA Kits)/p38 (show CRK ELISA Kits)/Egr-1 (show EGR1 ELISA Kits)/TSLP (show TSLP ELISA Kits) axis involved in allergic skin Th2 inflammation
data suggest that IL-33/ST2 (show SULT2A1 ELISA Kits) axis closely associates with poor prognosis of EOC patients, and it promotes ovarian cancer growth and metastasis through regulating ERK (show EPHB2 ELISA Kits) and JNK (show MAPK8 ELISA Kits) signaling pathways
Our findings indicate that the alarmin cytokines IL-33 and IL-25 (show IL25 ELISA Kits) increase basophil activation and migratory potential, and may pose as a novel therapeutic targets for the treatment of allergic asthma.
Targeting IL-33 was a promising strategy for controlling autoantibody production in AIHA patients
Data suggest that increased plasma Interleukin-33 (IL-33) levels in patients with rheumatoid arthritis (RA) could be a possible biomarker to reflect the potential risks of bone erosion.
IL13 (show IL13 ELISA Kits) gene transcription can be directly activated by nuclear IL-33, which is negatively regulated by the deubiquitinase USP17.
IL-33(+) Muller cells were more abundant and IL-33 cytokine was elevated in advanced age-related macular degeneration cases compared with age-matched controls.
Results indicate that IL-33 may reversibly destabilize the endothelial barrier, thus accelerating the supply with immunomodulators and assisting leukocytes to reach wounded tissue.
No significant association between IL33 rs7025417, IL33 rs7044343, IL1RL1 (show IL1RL1 ELISA Kits) rs2058660, IL1RL1 (show IL1RL1 ELISA Kits) rs2310173 and IL1RL1 (show IL1RL1 ELISA Kits) rs13015714 and cIMT values was observed.
Decreased expression of IL-33 in immune thrombocytopenia
pre-treatment with the ERK1/2 (show MAPK1/3 ELISA Kits) inhibitor U0126 or anti-IL-33 antibody significantly abolished the ovalbumin (show OVA ELISA Kits)-induced airway remodeling, increased expressions of IL-33
IL-33 promotes degranulation of bone marrow-derived mast cells and release of cytokines IL-1beta (show IL1B ELISA Kits), IL-6 (show IL6 ELISA Kits) and TNF-alpha (show TNF ELISA Kits).
pulmonary interstitial macrophages may not be a source of IL-33 during Respiratory syncytial virus infection
demonstrate a pathogenic role for IL-33-mediated eosinophilia and activation of Th2 immunity in chronic intestinal inflammation
IL-33 as a novel regulator of neutrophil function in antibacterial infection
Our results provide novel insights into the mechanism of action behind alum-induced cytokine responses and show that IL-33 is sufficient to provide a robust secondary antibody response independently of alum.
Results demonstrated that IL-33 was released by activated astrocytes actively, and by damaged neurons during experimental autoimmune encephalomyelitis
This study indicates a pathogenic role of alarmin IL-33 in a murine model of scrub typhus and highlights infection-triggered endothelial cell damage and IL-33-mediated pathological changes during the course of Orientia infection.
IL-33, an alarmin released in response to tissue damage, was secreted from vaginal epithelium after the depletion of commensal microbiota.
promotes muscle T reg (show KCNH2 ELISA Kits) homeostasis in young mice, and efficient regeneration of injured muscles in aged mice
IL-33 may be a key molecule operating in eosinophil-mediated fibrosis in high-dose-per fraction irradiated skin.
IL33 (MIM 608678) is a member of the IL1 (see MIM 147760) family that potently drives production of T helper-2 (Th2)-associated cytokines (e.g., IL4\; MIM 147780). IL33 is a ligand for IL33R (IL1RL1\; MIM 601203), an IL1 family receptor that is selectively expressed on Th2 cells and mast cells (summary by Yagami et al., 2010
, DVS27-related protein
, interleukin-1 family member 11
, nuclear factor for high endothelial venules
, nuclear factor from high endothelial venules