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Transcription factor involved in regulating gene activity following the primary growth factor response. Additionally we are shipping JUNB Kits (8) and JUNB Proteins (8) and many more products for this protein.
Showing 10 out of 246 products:
Human Polyclonal JUNB Primary Antibody for EIA, WB - ABIN952979
Strathdee, Ferguson, Sim, Brown: DNA methylation does not regulate JUNB expression in CML: comment on \Downregulation of JUNB mRNA expression in advanced phase chronic myelogenous leukemia\" by Hoshino et al. [Leuk. Res. 33 (2009) 1361-1366]." in Leukemia research 2010
Show all 5 references for ABIN952979
Human Polyclonal JUNB Primary Antibody for IF, IHC (p) - ABIN197138
Bockmeyer, Kern, Forstmeier, Lovric, Modde, Agustian, Steffens, Birschmann, Traeder, Dämmrich, Schwarz, Kreipe, Bröcker, Becker: Arteriolar vascular smooth muscle cell differentiation in benign nephrosclerosis. in Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association 2012
Show all 2 references for ABIN197138
Human Polyclonal JUNB Primary Antibody for IF, IHC (p) - ABIN196656
Narayanan, Srinivas, Peterson, Ramachandran, Hao, Thimmapaya, Scherer, George: Transcriptional regulation of dentin matrix protein 1 by JunB and p300 during osteoblast differentiation. in The Journal of biological chemistry 2004
Human Polyclonal JUNB Primary Antibody for IHC (p), WB - ABIN196658
Beausoleil, Jedrychowski, Schwartz, Elias, Villén, Li, Cohn, Cantley, Gygi: Large-scale characterization of HeLa cell nuclear phosphoproteins. in Proceedings of the National Academy of Sciences of the United States of America 2004
Cow (Bovine) Polyclonal JUNB Primary Antibody for WB - ABIN452429
Starr, Shiels, Harris, Pattie, Pearce, Relton, Deary: Oxidative stress, telomere length and biomarkers of physical aging in a cohort aged 79 years from the 1932 Scottish Mental Survey. in Mechanisms of ageing and development 2008
Chicken Polyclonal JUNB Primary Antibody for WB - ABIN2780414
Trøen, Nygaard, Jenssen, Ikonomou, Tierens, Matutes, Gruszka-Westwood, Catovsky, Myklebost, Lauritzsen, Hovig, Delabie: Constitutive expression of the AP-1 transcription factors c-jun, junD, junB, and c-fos and the marginal zone B-cell transcription factor Notch2 in splenic marginal zone lymphoma. in The Journal of molecular diagnostics : JMD 2004
Results suggested that JunB could play an important role in promoting cell invasion, migration and distant metastasis in head and neck squamous cell carcinoma via pathways other than epithelial-to-mesenchymal transition.
Highly recurrent mutation of JUNB is associated with nodular lymphocyte predominant Hodgkin lymphoma.
ETS2 (show ETS2 Antibodies), HNF4A (show HNF4A Antibodies) and JUNB are synergistic master regulators of epithelial-to-mesenchymal transition in cancer.
CARMA1 (show CARD11 Antibodies)- and MyD88 (show MYD88 Antibodies)-dependent activation of Jun (show JUN Antibodies)/ATF-type AP-1 (show FOSB Antibodies) complexes is a hallmark of ABC (show ABCB6 Antibodies) diffuse large B-cell lymphomas.
PDK1 (show PDK1 Antibodies) functions as a tumor promoter in human gallbladder cancer by upregulating JunB, promoting epithelial mesenchymal transformation, and cell migration.
Our findings demonstrate that miRNA-149* may serve as an oncogenic regulator in T-cell acute lymphoblastic leukemia by negatively regulating JunB
The MAPK (show MAPK1 Antibodies) pathway plays a primary role in the control of JUNB gene expression.
JunB is likely to be a key target of c-Abl (show ABL1 Antibodies) in expression of p21 (show CDKN1A Antibodies) in Adriamycin-induced DDR (show DDR1 Antibodies).
Caveolin 2 (show CAV2 Antibodies) disengages repressed Egr-1 (show EGR1 Antibodies) and JunB promoters from lamin A/C (show LMNA Antibodies) through disassembly of H3K9me3 in the inner nuclear membrane.
JunB expression was significantly increased while cyclin-D1 (show CCND1 Antibodies) expression was significantly down-regulated in pre-eclampsia relative to control placental mesenchymal stromal cells.
The present data indicate that bovine dialyzable leukocyte extract can block the AP-1 (show JUN Antibodies) DNA-binding activity and expression of several transcriptions factors in breast cancer cells.
Data demonstrate for the first time an essential role of JunB-CBFbeta (show CBFB Antibodies) signaling for maintaining sarcomere architecture and function.
JUNB is a significant modulator of both classical and alternative macrophage activation.
Study shows that myeloid deletion of JUNB dampens immune polarization and reshapes disease outcomes during infection with both P. berghei and N. brasiliensis by limiting type 1 and type 2 responses, respectively. Thus, JUNB is an important regulator of myeloid responses to both type 1 and type 2 infections in vivo.
Loss of JunB expression led to increased proliferation and decreased senescence, likely owing to decreased p16(Ink4a (show CDKN2A Antibodies)) and p21(CIP1 (show CDKN1A Antibodies)) in epithelial cells.
JunB and c-Jun (show JUN Antibodies) expression in post-mitotic oligodendrocytes is mostly dispensable for the maintainance of white matter tracts throughout adult life, even under demyelinating conditions.
JunB controls epidermal growth, barrier formation, and proinflammatory responses through direct and indirect mechanisms, pinpointing SQSTM1 (show SQSTM1 Antibodies) as a key mediator of JunB suppression of NF-kappaB (show NFKB1 Antibodies)-dependent inflammation
our results suggest a functional cooperation between NFAT1 (show NFAT1 Antibodies) and JunB in mediating IL-31 (show IL31 Antibodies) gene expression in CD4 (show CD4 Antibodies)(+) T cells
an important role of the A2B (show ADORA2B Antibodies) receptor-dependent upregulation of JunB in VEGF (show VEGFA Antibodies) production and possibly other AP-1 (show JUN Antibodies)-regulated events.
In experimental hepatitis, the absence of JUNB in immune cells decreased IFN-gamma (show IFNG Antibodies) expression and secretion from NK & NKT (show CTSL1 Antibodies) cells, leading to reduced STAT1 (show STAT1 Antibodies) pathway activation.
The results show that the junb gene is organized in a nuclear chromatin loop bringing into close spatial proximity the upstream promoter region and the downstream enhancer and that this configuration permits immediate RNA Pol II (show POLR2F Antibodies) release on the junb body on binding of LPS (show TLR4 Antibodies)-activated NF-kappaB (show NFKB1 Antibodies) to the enhancer.
Data indicate that the C-terminal JunB caspase (show CASP3 Antibodies) cleavage product functions as a potent inhibitor of AP-1 (show JUN Antibodies)-dependent transcription.
Transcription factor involved in regulating gene activity following the primary growth factor response. Binds to the DNA sequence 5'-TGATCA-3'.
jun B proto-oncogene
, jun-B proto-oncogene
, activator protein 1
, transcription factor jun-B
, Jun-B oncogene