Use your antibodies-online credentials, if available.
No Products on your Comparison List.
Your basket is empty.
Find out more
Natural killer (NK) cells are a distinct lineage of lymphocytes that mediate cytotoxic activity and secrete cytokines upon immune stimulation. Additionally we are shipping Killer Cell Lectin-Like Receptor Subfamily D, Member 1 Proteins (16) and many more products for this protein.
Showing 10 out of 260 products:
Mouse (Murine) Monoclonal KLRD1 Primary Antibody for FACS, IP - ABIN2663655
Moretta, Biassoni, Bottino, Pende, Vitale, Poggi, Mingari, Moretta: Major histocompatibility complex class I-specific receptors on human natural killer and T lymphocytes. in Immunological reviews 1997
Show all 5 references for ABIN2663655
Mouse (Murine) Monoclonal KLRD1 Primary Antibody for EIA, FACS - ABIN951438
Meyers, Ryu, Monney, Nguyen, Greenfield, Freeman, Kuchroo: Cutting edge: CD94/NKG2 is expressed on Th1 but not Th2 cells and costimulates Th1 effector functions. in Journal of immunology (Baltimore, Md. : 1950) 2002
Show all 3 references for ABIN951438
Mouse (Murine) Monoclonal KLRD1 Primary Antibody for EIA, FACS - ABIN951437
Toyama-Sorimachi, Taguchi, Yagita, Kitamura, Kawasaki, Koyasu, Karasuyama: Mouse CD94 participates in Qa-1-mediated self recognition by NK cells and delivers inhibitory signals independent of Ly-49. in Journal of immunology (Baltimore, Md. : 1950) 2001
Show all 3 references for ABIN951437
Mouse (Murine) Monoclonal KLRD1 Primary Antibody for EIA, FACS - ABIN951439
Vance, Kraft, Altman, Jensen, Raulet: Mouse CD94/NKG2A is a natural killer cell receptor for the nonclassical major histocompatibility complex (MHC) class I molecule Qa-1(b). in The Journal of experimental medicine 1999
Show all 3 references for ABIN951439
Human Polyclonal KLRD1 Primary Antibody for EIA, WB - ABIN453720
Rodríguez, Carretero, Glienke, Bellón, Ramírez, Lehrach, Francis, López-Botet: Structure of the human CD94 C-type lectin gene. in Immunogenetics 1998
Show all 2 references for ABIN453720
Human Monoclonal KLRD1 Primary Antibody for FACS, ELISA - ABIN969031
Lonigro, Mirabile, Munda, Barchitta, Bottino, Fazzio, Di Cataldo, Agodi, Schilirò: Association between high expression of natural killer related-genes (NCAM/CD94) and early death during induction in children with acute myeloid leukemia. in Leukemia 2008
Show all 2 references for ABIN969031
Mouse (Murine) Monoclonal KLRD1 Primary Antibody for FACS - ABIN316814
Vance, Jamieson, Raulet: Recognition of the class Ib molecule Qa-1(b) by putative activating receptors CD94/NKG2C and CD94/NKG2E on mouse natural killer cells. in The Journal of experimental medicine 2000
Show all 2 references for ABIN316814
Human Monoclonal KLRD1 Primary Antibody for ELISA, WB - ABIN969032
Masilamani, Narayanan, Prieto, Borrego, Coligan: Uncommon endocytic and trafficking pathway of the natural killer cell CD94/NKG2A inhibitory receptor. in Traffic (Copenhagen, Denmark) 2008
Show all 2 references for ABIN969032
Human Monoclonal KLRD1 Primary Antibody for FACS, IHC (fro) - ABIN319857
Aramburu, Balboa, Ramírez, Silva, Acevedo, Sánchez-Madrid, De Landázuri, López-Botet: A novel functional cell surface dimer (Kp43) expressed by natural killer cells and T cell receptor-gamma/delta+ T lymphocytes. I. Inhibition of the IL-2-dependent proliferation by anti-Kp43 monoclonal antibody. in Journal of immunology (Baltimore, Md. : 1950) 1990
CD94 and NKG2A (show KLRC1 Antibodies) polymorphisms may contribute to genetic susceptibility to rheumatoid arthritis or affect the response to anti-TNF (show TNF Antibodies) therapy in patients of Caucasian origin.
Coengagement of inhibitory receptors, either KIR2DL1 (show KIR2DL1 Antibodies) or CD94-NKG2A (show KLRC1 Antibodies), did not inhibit phosphorylation of Stat5 (show STAT5A Antibodies) but inhibited selectively phosphorylation of Akt (show AKT1 Antibodies) and S6 ribosomal protein (show RPS6 Antibodies).
it is not clear if high expression of CD94 on peripheral blood NK cells is related to abnormal activity of endometrial NK cells.
Data indicate that NKG2 receptor NKG2E (show KLRC3 Antibodies) was capable of associating with CD94 and DAP12 (show TYROBP Antibodies) but that the complex was retained intracellularly at the endoplasmic reticulum.
Balance between activating NKG2D (show KLRK1 Antibodies), DNAM-1 (show CD226 Antibodies), NKp44 (show NCR2 Antibodies) and NKp46 (show NCR1 Antibodies) and inhibitory CD94/NKG2A (show KLRC1 Antibodies) receptors determine natural killer degranulation towards rheumatoid arthritis synovial fibroblasts.
Data indicate that the expression of KLRD1 (CD94) and NKG2E (KLRC3 (show KLRC3 Antibodies)) was reduced in NK-enriched cells in fulminant type 1 diabetes.
NKG2C (show KLRC2 Antibodies) zygosity influences CD94/NKG2C (show KLRC2 Antibodies) receptor function and the NK-cell compartment redistribution in response to human cytomegalovirus.
Synergistic inhibition of natural killer cells by the nonsignaling molecule CD94.
Studies indicate that HLA-E (show HLAE Antibodies) interacts with CD94/NKG2 receptors expressed mainly on the surface of natural killer (NK) cells, thus confining its role to the regulation of NK-cell function.
Results suggested that the low expression level of CD94/NKG2A (show KLRC1 Antibodies) upon gammadelta T cell activation might lead to the over-activation of gammadelta T cells in patients with SLE.
These findings suggest that the CD94/NKG2A (show KLRC1 Antibodies) heterodimers in cattle, in contrast to other species, are binding several different ligands.
Segregation of a spontaneous Klrd1 mutation in DBA (show RPS19 Antibodies)/2 mouse substrains
The transmembrane region sequences of CD94 and NKG2 in mouse and rat differ markedly from other mammalian orthologs by the presence of a lysine residue in the transmembrane region.
The complex interplay between various stimuli may account for the variable expression of CD94/NKG2A (show KLRC1 Antibodies) during responses to different pathogens in vivo.
expression of CD94 and its associated NKG2A (show KLRC1 Antibodies), NKG2C (show KLRC2 Antibodies), and NKG2E (show KLRC3 Antibodies) subunits is dispensable for NK cell development, education, and many NK cell functions
show that CD94, a molecule preferentially expressed by NK cells, is essential for the resistance of C57BL/6 mice to mousepox, a disease caused by the Orthopoxvirus ectromelia virus
Implications of CD94 deficiency and monoallelic NKG2A (show KLRC1 Antibodies) expression for natural killer cell development and repertoire formation.
CD94 and NKG2 were both expressed early in NK cell development, sometimes in the absence of NK1.1, with CD94 invariably being expressed at two different levels. IL-4 differentially inhibited the expression of CD94 and Ly49 receptors.
Inhibitory receptor CD94 is expressed on mature fetal thymic and adult epidermal TCR Vgamma3+ lymphocytes.
The acquisition of individual receptor gene expressions during various stages of differentiation in culture from embryonic stem cells to NK cells follows a predetermined order, with the order of receptor acquisition being first CD94.
There is no evidence that CD94 inhibits either the lytic function of lymphocytic choriomeningitis virus-specific T cells or their capacity to produce effector cytokines upon peptide stimulation.
Natural killer (NK) cells are a distinct lineage of lymphocytes that mediate cytotoxic activity and secrete cytokines upon immune stimulation. Several genes of the C-type lectin superfamily, including members of the NKG2 family, are expressed by NK cells and may be involved in the regulation of NK cell function. KLRD1 (CD94) is an antigen preferentially expressed on NK cells and is classified as a type II membrane protein because it has an external C terminus. Three transcript variants encoding two different isoforms have been found for this gene.
, NK cell receptor
, natural killer cells antigen CD94
, killer cell lectin-like receptor family D member 1
, killer cell lectin-like receptor subfamily D member 1
, Killer cell lectin-like receptor subfamily D member 1
, CD94 antigen (located within the rat natural killer gene complex)