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The protein encoded by KLF11 is a zinc finger transcription factor that binds to SP1-like sequences in epsilon- and gamma-globin gene promoters. Additionally we are shipping KLF11 Proteins (4) and KLF11 Kits (1) and many more products for this protein.
Showing 10 out of 72 products:
Human Monoclonal KLF11 Primary Antibody for IF, IHC (p) - ABIN563638
Daftary, Zheng, Tabbaa, Schoolmeester, Gada, Grzenda, Mathison, Keeney, Lomberk, Urrutia: A novel role of the Sp/KLF transcription factor KLF11 in arresting progression of endometriosis. in PLoS ONE 2013
Show all 2 references for ABIN563638
Human Polyclonal KLF11 Primary Antibody for EIA, WB - ABIN453419
Kuroda, Horikawa, Enya, Oda, Suzuki, Iizuka, Takeda: Identification of minimal promoter and genetic variants of Kruppel-like factor 11 gene and association analysis with type 2 diabetes in Japanese. in Endocrine journal 2009
Show all 2 references for ABIN453419
Human Polyclonal KLF11 Primary Antibody for FACS, IF - ABIN390561
Ma, Hanson, Que, Mack, Franks, Infante, Kobes, Bogardus, Baier: Association analysis of Krüppel-like factor 11 variants with type 2 diabetes in Pima Indians. in The Journal of clinical endocrinology and metabolism 2008
Chicken Polyclonal KLF11 Primary Antibody for WB - ABIN2779252
Neve, Fernandez-Zapico, Ashkenazi-Katalan, Dina, Hamid, Joly, Vaillant, Benmezroua, Durand, Bakaher, Delannoy, Vaxillaire, Cook, Dallinga-Thie, Jansen, Charles, Clément, Galan, Hercberg, Helbecque, Charpentier, Prentki, Hansen, Pedersen, Urrutia, Melloul,: Role of transcription factor KLF11 and its diabetes-associated gene variants in pancreatic beta cell function. in Proceedings of the National Academy of Sciences of the United States of America 2005
Results suggest a role for KLF11 in upregulating MAO-A (show MAOA Antibodies) in depressive disorder and chronic social stress, suggesting that inhibition of the pathways regulated by this transcription factor may aid in the therapeutics of neuropsychiatric illnesses
The data suggest a relationship between promoter DNA methylation (show HELLS Antibodies) and KLF11 gene expression in ovarian cancer tumorigenesis.
identified an evolutionarily conserved metabolic regulator, Kruppel-like factor 11 (KLF11), as a novel browning transcription factor in human adipocytes that is required for rosiglitazone-induced browning.
KLF11 repressed most endometrial CYP (show PPIG Antibodies) enzymes
Levels of KLF11 are significantly increased in the postmortem prefrontal cortex of subjects with alcohol dependence compared to controls.
KLF11 regulates distinct gene networks.
KLF111 represses glycodelin-A (show PAEP Antibodies) levels and influences its role in the ovarian endometrium.
KLF11 is a critical transcription factor in the function of mesenchymal cells and, specifically, as a negative regulator of collagen secretion.
Data identify a novel pathogenic role for KLF11 in preventing de novo disease-associated fibrosis in endometriosis.
The A347S mutation disrupted KLF11-mediated increases in basal insulin (show INS Antibodies) levels and promoter activity and blunted glucose-stimulated insulin (show INS Antibodies) secretion.
Data show that the major phenotype given by the whole-body deletion of Kruppel-like factor 11 (Klf11) is not diabetes but increased insulin (show INS Antibodies) sensitivity.
Loss of KLF11 expression is associated with hyperglycemia in Type 2 diabetes.
this study revealed a new role for the KLF11 gene in the mechanisms underlying the antinociceptive effects of chronic ethanol exposure
Taken together, our findings identify a novel anti-hypertrophic and anti-fibrotic role of KLF11, and KLF11 activator may serve as candidate drug for heart failure patients.
Overexpression of KLF11 in the livers of diabetic and obese mice activates the PPARalpha (show PPARA Antibodies) signaling pathway, subsequently markedly improving the fatty liver phenotype. Knockdown of KLF11 increases hepatic triglycerides.
Recruitment of KLF11 as a novel co-regulator (with peroxisome proliferator-activated receptor PPARgamma (show PPARG Antibodies)) plays a critical role in cerebrovascular protection after ischemic insults.
Kruppel-like factor-11, a transcription factor involved in diabetes mellitus, suppresses endothelial cell activation via the nuclear factor-kappaB signaling pathway.
The protein encoded by this gene is a zinc finger transcription factor that binds to SP1-like sequences in epsilon- and gamma-globin gene promoters. This binding inhibits cell growth and causes apoptosis. Defects in this gene are a cause of maturity-onset diabetes of the young type 7 (MODY7). Three transcript variants encoding two different isoforms have been found for this gene.
Kruppel-like factor 11
, novel kruppel-like factor family protein
, krueppel-like factor 11-like
, Krueppel-like factor 11-like
, Krueppel-like factor 11
, TGFB-inducible early growth response protein 2
, transforming growth factor-beta-inducible early growth response protein 2
, TGFB inducible early growth response 2
, TGFB inducible early growth response 3
, TGFB-inducible early growth response protein 2b
, TGFB-inducible early growth response protein 3
, transforming growth factor-beta-inducible early growth response protein 3
, transcription factor CP2-like 2
, TGF-beta inducible early growth response protein 2