Use your antibodies-online credentials, if available.
No Products on your Comparison List.
Your basket is empty.
Find out more
The protein encoded by KLF7 is a member of the Kruppel-like transcriptional regulator family. Additionally we are shipping KLF7 Proteins (5) and KLF7 Kits (1) and many more products for this protein.
Showing 10 out of 55 products:
Human Polyclonal KLF7 Primary Antibody for ELISA, WB - ABIN563711
Cho, Park, Shin, Kim, Shin, Shin, Lee, Lee, Baik, Lee: (-)-Catechin suppresses expression of Kruppel-like factor 7 and increases expression and secretion of adiponectin protein in 3T3-L1 cells. in American journal of physiology. Endocrinology and metabolism 2007
Show all 2 Pubmed References
Human Monoclonal KLF7 Primary Antibody for ELISA, WB - ABIN563712
Caiazzo, Colucci-DAmato, Esposito, Parisi, Stifani, Ramirez, di Porzio: Transcription factor KLF7 regulates differentiation of neuroectodermal and mesodermal cell lineages. in Experimental cell research 2010
In the spinal cord, klf7- and klf6a (show KLF6 Antibodies)-expressing cells are found in both the dorsal and ventral horns. Numerous sensory structures (e.g. auditory, lateral line, olfactory and visual) and several motor nuclei (e.g. oculomotor, trigeminal, and vagal motor nuclei) contain klf7- and/or klf6a (show KLF6 Antibodies)-expressing cells
Data revealed that expression of DNA binding inhibitor 3 (show PPP1R11 Antibodies), early B cell factor 2 (show EBF2 Antibodies), Ebf3 (show EBF3 Antibodies), Iroquois related homeobox 1 (show IRX1 Antibodies), Kruppel-like factor 7 , mab-21-like 1 (show MAB21L1 Antibodies) , fatty acid binding protein 7 (show FABP7 Antibodies) and stathmin-like 4 (show STMN4 Antibodies),were enriched in the diencephalon of zebrafish.
Higher expression of TLR4 (show TLR4 Antibodies) and KLF7 may play a vital role in the process of inflammation induced by obesity in visceral adipose tissue
a dominant mutation in FBXO38 (show FBXO38 Antibodies) causes adult-onset, progressive spinal muscular atrophy with calf predominance
KLF7 was upregulated by morphine at both the transcriptional and the translational levels, promoting cell growth up to day 4
The gene encoding KLF7 is a novel candidate for conferring genetic susceptibility to type 2 diabetes.
KLF7 may contribute to the pathogenesis of type 2 diabetes through an impairment of insulin (show INS Antibodies) biosynthesis and secretion in pancreatic beta-cells and a reduction of insulin (show INS Antibodies) sensitivity in peripheral tissues.
We identified a novel association between the minor A-allele of KLF7 and protection against obesity in the Danish population
Data show that adult corticospinal tract (CST (show CORT Antibodies)) neurons failed to up-regulate KLF7 in response to axon injury, and overexpression of VP16-KLF7 in vivo promoted both sprouting and regenerative axon growth in the CST (show CORT Antibodies) of adult mice.
KLF7 inactivation in Klf7-null mice decreases the expression of the neurogenic marker brain lipid-binding protein/fatty acid-binding protein 7 (show FABP7 Antibodies) (BLBP/FABP7 (show FABP7 Antibodies)) in neural stem cells (NSCs).
Klf7 gene ablation reduces TrkA (show NTRK1 Antibodies) expression in sensory neurons due to effect on cell death
Klf7 is important for neuronal morphogenesis in selected regions of the central nervous system.
Cooperation between Brn3a (show POU4F1 Antibodies) and Klf7 is required for endogenous TrkA (show NTRK1 Antibodies) gene expression and the survival of nociceptive sensory neurons.
Results advance knowledge of transcriptional regulation of olfactory neurogenesis and KLF7 action.
Upregulation of adiponectin (show ADIPOQ Antibodies) protein by (-)-catechin may involve, at least in part, suppression of KLF7 in 3T3-L1 cells.
The protein encoded by this gene is a member of the Kruppel-like transcriptional regulator family. Members in this family regulate cell proliferation, differentiation and survival and contain three C2H2 zinc fingers at the C-terminus that mediate binding to GC-rich sites. This protein may contribute to the progression of type 2 diabetes by inhibiting insulin expression and secretion in pancreatic beta-cells and by deregulating adipocytokine secretion in adipocytes. A pseudogene of this gene is located on the long arm of chromosome 3. Alternative splicing results in multiple transcript variants.
Kruppel-like factor 7 (ubiquitous)
, Krueppel-like factor 7
, ubiquitous Kruppel-like transcription factor