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Epoxide hydrolase that catalyzes the final step in the biosynthesis of the proinflammatory mediator leukotriene B4. Additionally we are shipping Leukotriene A4 Hydrolase Antibodies (86) and Leukotriene A4 Hydrolase Proteins (11) and many more products for this protein.
Showing 9 out of 16 products:
Human LTA4H ELISA Kit for Sandwich ELISA - ABIN418927
Wells, Jackson, Viera, Bhatt, Gautney, Handley, King, Xu, Gaggar, Bailey, Dransfield, Blalock: A Randomized, Placebo-controlled Trial of Roflumilast. Effect on Proline-Glycine-Proline and Neutrophilic Inflammation in Chronic Obstructive Pulmonary Disease. in American journal of respiratory and critical care medicine 2015
lta4h mutations confer hypersusceptibility to mycobacterial infection by redirecting eicosanoid substrates to anti-inflammatory lipoxins.
Mutating F375 in xlLTA4H to tyrosine abolishes the formation of the LTB4 (show PTGR1 ELISA Kits) isomeric product Delta(6)-trans-Delta(8)-cis (show CISH ELISA Kits)-LTB4 (show PTGR1 ELISA Kits).
LTA4H promoter region SNP was associated with susceptibility to bacteriologically confirmed bacterial meningitis but did not influence clinical presentation, disease severity or survival following dexamethasone treatment.
Report no association between LTA4H SNPs and atherosclerotic plaque phenotypes.
The best proteinogenic amino acid recognized by LTA4H is arginine.
The results of our study failed to confirm whether the selected variants in LTA4H gene within the LT metabolism pathway contribute to platelet reactivity in a diabetic population treated with ASA (show ARSA ELISA Kits).
The LTA4H A-9188>G polymorphism has a strong association with severe asthma in children.
Data indicate that 13S,14S-epoxy-docosahexaenoic acid (DHA) inhibits leukotriene B4 (LTB4 (show PTGR1 ELISA Kits)) biosynthesis by leukotriene A4 hydrolase (LTA4H).
genetic association studies in population of Han Chinese in Eastern China: Using a recessive genetic model, an SNP in LTA4H (rs6538697) is associated with increased risk of ischemic stroke.
Structural origins for the loss of catalytic activities of bifunctional human LTA4H revealed through molecular dynamics simulations.
It was found modest association with LTA4H rs1978331C (intron 11) with increased FEV1 (p = 0.029) and with increased FEV1/FVC ratio (p = 0.020).
common polymorphisms in the LTA4H gene do not play any major role in susceptibility to clinical pulmonary tuberculosis
There is a critical role for the secondary anti-inflammatory activity of LTA4H in acute pulmonary infection.
CS exposure promotes the development of CS-induced emphysema by suppressing the enzymatic activities of the LTA4H aminopeptidase in lung tissues and accumulating PGP and neutrophils in the airspaces.
in acute neutrophil-driven lung inflammation, PGP was degraded by LTA(4)H which facilitated resolution of inflammation; cigarette smoke, a major risk factor for COPD,inhibited LTA(4)H aminopeptidase activity leading to accumulation of PGP and neutrophils
5-lipoxygenase (show ALOX5 ELISA Kits) and leukotriene A4 hydrolase expression in atherosclerotic lesions correlates with symptoms of plaque instability
In the mouse nervous system ,Immunoreactivity for leukotriene A4 hydrolase showed widespread distribution with preference to the sensory-associated structures.
Epoxide hydrolase that catalyzes the final step in the biosynthesis of the proinflammatory mediator leukotriene B4. Has also aminopeptidase activity.
leukotriene A4 hydrolase
, leukotriene A-4 hydrolase
, Leukotriene A4 hydrolase
, leukotriene A-4 hydrolase-like
, LTA-4 hydrolase
, aminopeptidase T
, leukotriene A(4) hydrolase
, LTA4 hydrodase
, LTA4 hydrolase