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Drosophila that have mutations in their mago nashi (grandchildless) gene produce progeny with defects in germplasm assembly and germline development. Additionally we are shipping Mago-Nashi Homolog, Proliferation-Associated (Drosophila) Proteins (14) and many more products for this protein.
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Human Polyclonal MAGOH Primary Antibody for ELISA, WB - ABIN268734
Zhao, Colaizzo-Anas, Nowak, Shows, Elliott, Aplan: The mammalian homologue of mago nashi encodes a serum-inducible protein. in Genomics 1998
findings show 2 genes MAGOH and MAGOHB (show MAGOHB Antibodies) are expressed in mammals; in macrophages, expression of MAGOHB (show MAGOHB Antibodies) but not MAGOH mRNA increases after LPS (show IRF6 Antibodies) stimulation; both MAGOH proteins interact with other exon junction complex (EJC) components, incorporate into mRNA-bound EJCs and activate nonsense-mediated decay
The stable association of multiprotein exon junction complex core with RNA is maintained by inhibition of eIF4AIII (show EIF4A3 Antibodies) ATPase (show DNAH8 Antibodies) activity by MAGOH-Y14 (show RBM8A Antibodies).
crystal structure of a tetrameric exon junction core complex containing the DEAD-box adenosine triphosphatase eukaryotic initiation factor 4AIII (show EIF4A3 Antibodies) bound to an ATP analog, MAGOH, Y14 (show RBM8A Antibodies), a fragment of MLN51 (show CASC3 Antibodies), and a polyuracil mRNA mimic
These results indicate that MAGOH regulates the transcriptional activation of STAT3 (show STAT3 Antibodies) by interfering complex formation between STAT3 (show STAT3 Antibodies) and Y14 (show RBM8A Antibodies).
we first show that Eif4a3 (show EIF4A3 Antibodies) haploinsufficiency phenocopies aberrant neurogenesis and microcephaly of Magoh and Rbm8a (show RBM8A Antibodies) mutant mice.we show that genetic ablation of one downstream pathway, p53 (show TP53 Antibodies), significantly rescues microcephaly of all 3 EJC mutant
findings show 2 genes MAGOH and MAGOHB (show MAGOHB Antibodies) are expressed in mammals; in macrophages, expression of MAGOHB (show MAGOHB Antibodies) but not MAGOH mRNA increases after LPS (show TLR4 Antibodies) stimulation; both MAGOH proteins interact with other exon junction complex (EJC) components, incorporate into mRNA-bound EJCs and activate nonsense-mediated decay
These results point to a central role for Magoh in melanocyte development.
mouse magoh is involved in cyclin-dependent kinase (show CDK1 Antibodies) regulation.
Magoh haploinsufficiency causes microcephaly because of intermediate neural progenitors depletion and neuronal apoptosis.
Zygotic mago nashi is expressed into the dorsal-marginal region during gastrulation.
MAPK (show MAPK1 Antibodies) is the primary functional target of mago in eye development; in cultured cells, Mago knockdown disproportionately affects other large genes located in heterochromatin
Mago Nashi, Tsunagi/Y14 (show RBM8A Antibodies), and Ranshi form a complex that influences oocyte differentiation in Drosophila melanogaster
We have determined the crystal structure of the complex between Drosophila melanogaster Y14 (show RBM8A Antibodies) and Mago at a resolution of 2.5 A. The structure reveals an atypical mode of protein-protein recognition mediated by an RNA-binding domain (RBD (show CACNA1D Antibodies)).
mago nashi is required for germline stem cell differentiation, but surprisingly mago nashi functions independently of tsunagi/Y14 (show RBM8A Antibodies) in this process
Drosophila that have mutations in their mago nashi (grandchildless) gene produce progeny with defects in germplasm assembly and germline development. This gene encodes the mammalian mago nashi homolog. In mammals, mRNA expression is not limited to the germ plasm, but is expressed ubiquitously in adult tissues and can be induced by serum stimulation of quiescent fibroblasts.
protein mago nashi homolog
, mago-nashi-like proliferation-associated protein
, mago-nashi homolog, proliferation-associated (Drosophila)
, mago nashi
, CG9401 gene product from transcript CG9401-RA
, mago nishi
, mago nashi homolog