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MAGEA10 is a member of the MAGEA gene family. Additionally we are shipping Melanoma Antigen Family A, 10 Proteins (2) and many more products for this protein.
Showing 10 out of 52 products:
Human Polyclonal MAGEA10 Primary Antibody for EIA, IHC (p) - ABIN358641
Rogner, Wilke, Steck, Korn, Poustka: The melanoma antigen gene (MAGE) family is clustered in the chromosomal band Xq28. in Genomics 1996
Show all 2 references for ABIN358641
Cow (Bovine) Polyclonal MAGEA10 Primary Antibody for WB - ABIN2777032
Lin, Lin, Thomas, Greenson, Giordano, Robinson, Barve, Weishaar, Taylor, Orringer, Beer: Melanoma-associated antigens in esophageal adenocarcinoma: identification of novel MAGE-A10 splice variants. in Clinical cancer research : an official journal of the American Association for Cancer Research 2004
CTAs (MAGE-A4, NY-ESO-1, MAGE-A10) were more likely expressed in patients with squamous cell carcinoma of the lung and when CTAs combined with CD133, they can be better prognostic factors.
By using Molecular-Docking, 3 MAGEA10 peptides were found binding to the groove position of HLA-A( *)0210 same as MAGEA4 peptide co-crystallized with HLA-A( *)0210, which indicates they could be promising for HLA-A( *)0201 presentation in immunotherapy.
We clearly showed that MAGE-A10 is frequently expressed in the group of triple-negative breast cancer patients
High MAGE-A10 is associated with lung, skin and urothelial malignancies.
This is the first report of these MAGE (show MAGEB10 Antibodies)-A10 alternative splice sequences, and characterization of MAGE (show MAGEB10 Antibodies)-A expression
This gene is a member of the MAGEA gene family. The members of this family encode proteins with 50 to 80% sequence identity to each other. The promoters and first exons of the MAGEA genes show considerable variability, suggesting that the existence of this gene family enables the same function to be expressed under different transcriptional controls. The MAGEA genes are clustered at chromosomal location Xq28. They have been implicated in some hereditary disorders, such as dyskeratosis congenita. Alternative splicing results in multiple transcript variants. Read-through transcription also exists between this gene and the downstream melanoma antigen family A, 5 (MAGEA5) gene.
melanoma antigen family A, 10
, MAGE-10 antigen
, cancer/testis antigen 1.10
, cancer/testis antigen family 1, member 10
, melanoma-associated antigen 10
, melanoma antigen family A, 11