Myristoylated Alanine Rich Protein Kinase C Substrate (MARCKS) ELISA Kits

The protein encoded by MARCKS is a substrate for protein kinase C.

list all ELISA KIts Gene Name GeneID UniProt
Anti-Mouse MARCKS MARCKS 17118 P26645
Anti-Human MARCKS MARCKS 4082 P29966
How to order from antibodies-online
  • +1 877 302 8632
  • +1 888 205 9894 (toll-free)
  • Order online
  • orders@antibodies-online.com

Top MARCKS ELISA Kits at antibodies-online.com

Showing 6 out of 20 products:

Catalog No. Reactivity Sensitivity Range Images Quantity Supplier Delivery Price Details
Human 15.6 pg/mL 62.5-4000 pg/mL Typical standard curve 96 Tests Log in to see 15 to 18 Days
$910.56
Details
Rat 0.55 ng/mL 1.56-100 ng/mL 96 Tests Log in to see 13 to 16 Days
$640.00
Details
Human
  96 Tests Log in to see 2 to 3 Days
$480.00
Details
Human
  96 Tests Log in to see 2 to 3 Days
$578.60
Details
Cow
  96 Tests Log in to see 15 to 18 Days
$1,029.60
Details
Chicken
  96 Tests Log in to see 15 to 18 Days
$1,095.60
Details

More ELISA Kits for Myristoylated Alanine Rich Protein Kinase C Substrate Interaction Partners

Mouse (Murine) Myristoylated Alanine Rich Protein Kinase C Substrate (MARCKS) interaction partners

  1. ENaC (show SCNN1A ELISA Kits) activity is regulated by calpain-2 (show CAPN2 ELISA Kits) proteolysis of MARCKS.

  2. In the absence of Pin1 (show PIN1 ELISA Kits), MARCKS is hyper-phosphorylated, leading to loss of cell adhesions, and collapse of the growth cone.

  3. Findings suggest that MIR429 modulates mucin (show SLC13A2 ELISA Kits) secretion in human colorectal cells and mouse colitis tissues by up-regulating of MARCKS expression.

  4. Conditional deletion of MARCKS in ECs induces intracellular accumulation of mucins, elevated oxidative stress, and lipid droplet buildup.

  5. MARCKS acts as a "molecular switch," binding to and regulating PIP2 signaling to regulate processes like proplatelet extension (microtubule-driven) vs proplatelet branching (Arp2/3 and actin polymerization-driven).

  6. MARCKS knockdown arrested VSMC cell cycle by decreasing KIS (show UHMK1 ELISA Kits) expression. Decreased KIS (show UHMK1 ELISA Kits) expression resulted in nuclear trapping of p27kip1 (show CDKN1B ELISA Kits) in VSMCs.

  7. MARCKS regulates the expression of proinflammatory cytokines in macrophages through activation of p38 (show CRK ELISA Kits)/JNK (show MAPK8 ELISA Kits) MAPK (show MAPK1 ELISA Kits) and NF-kappaB (show NFKB1 ELISA Kits).

  8. Targeting phospho-MARCKS overcomes drug-resistance and induces antitumor activity in preclinical models of multiple myeloma.

  9. Grin 1 is identified as a novel Cdk5 (show CDK5 ELISA Kits) substrate and MARCKS is confirmed as a Cdk5 (show CDK5 ELISA Kits) substrate.

  10. MARCKS appears to be a nonessential regulatory protein in mast cell exocytosis but exerts a negative modulation.

Human Myristoylated Alanine Rich Protein Kinase C Substrate (MARCKS) interaction partners

  1. These data suggested that miR34c3p acts as a tumor suppressor via regulation of MARCKS expression in OS progression

  2. Ca(2+)-PKC-MARCKS-PIP2-PI3K-PIP3 system functions as an activation module in vitro

  3. Findings show that calmodulin (CaM (show CALM1 ELISA Kits)) stimulates phosphoinositide-3-kinase (PI3K (show PIK3CA ELISA Kits)) lipid kinase activity by binding MARCKS and displacing it from phosphatidylinositol 4,5-bisphosphate (PIP2) headgroups, thereby releasing free PIP2 that recruits active PI3K (show PIK3CA ELISA Kits) to the membrane and serves as the substrate for the generation of phosphatidylinositol 3,4,5-trisphosphate (PIP3).

  4. Findings suggest that MIR429 modulates mucin (show SLC13A2 ELISA Kits) secretion in human colorectal cells and mouse colitis tissues by up-regulating of MARCKS expression.

  5. Knockdown of MARCKS in HepG2 cells reduced cell migration and invasion, but not cell proliferation.

  6. MARCKS upregulation increases vascular smooth muscle cell motility by activation of Rac1 and Cdc42 (show CDC42 ELISA Kits), promoting neointima formation.

  7. A novel role for MARCKS in regulating nuclear functions such as gene expression.

  8. MARCKS knockdown arrested VSMC cell cycle by decreasing KIS (show UHMK1 ELISA Kits) expression. Decreased KIS (show UHMK1 ELISA Kits) expression resulted in nuclear trapping of p27kip1 (show CDKN1B ELISA Kits) in VSMCs.

  9. unresponsiveness of breast cancer to paclitaxel treatment is, at least in part, mediated by phospho-MARCKS

  10. MARCKS and PPP1R9A (show PPP1R9A ELISA Kits) might contribute to spine loss in schizophrenia and bipolar disorder through their interactions.

Myristoylated Alanine Rich Protein Kinase C Substrate (MARCKS) Antigen Profile

Antigen Summary

The protein encoded by this gene is a substrate for protein kinase C. It is localized to the plasma membrane and is an actin filament crosslinking protein. Phosphorylation by protein kinase C or binding to calcium-calmodulin inhibits its association with actin and with the plasma membrane, leading to its presence in the cytoplasm. The protein is thought to be involved in cell motility, phagocytosis, membrane trafficking and mitogenesis.

Gene names and symbols associated with MARCKS

  • myristoylated alanine rich protein kinase C substrate (Marcks) antibody
  • myristoylated alanine-rich protein kinase C substrate (MARCKS) antibody
  • 80K-L antibody
  • MACS antibody
  • PKCSL antibody
  • PRKCSL antibody

Protein level used designations for MARCKS

myristoylated alanine-rich C-kinase substrate , myristoylated alanine-rich protein kinase C substrate (MARCKS, 80K-L) , phosphomyristin , protein kinase C substrate, 80 kDa protein, light chain

GENE ID SPECIES
17118 Mus musculus
4082 Homo sapiens
Did you look for something else?