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N-Acetyltransferase 8-Like (GCN5-Related, Putative) Proteins (NAT8L)

NAT8L encodes a single-pass membrane protein, which contains a conserved sequence of the GCN5 or NAT superfamily of N-acetyltransferases and is a member of the N-acyltransferase (NAT) superfamily. Additionally we are shipping N-Acetyltransferase 8-Like (GCN5-Related, Putative) Antibodies (17) and N-Acetyltransferase 8-Like (GCN5-Related, Putative) Kits (10) and many more products for this protein.

list all proteins Gene Name GeneID UniProt
NAT8L 269642 Q3UGX3
Rat NAT8L NAT8L 289727 D3ZVU9
NAT8L 339983 Q8N9F0
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Top N-Acetyltransferase 8-Like (GCN5-Related, Putative) Proteins at antibodies-online.com

Showing 3 out of 3 products:

Catalog No. Origin Source Conjugate Images Quantity Supplier Delivery Price Details
Insect Cells Mouse rho-1D4 tag „Crystallography Grade“ protein due to multi-step, protein-specific purification process 0.25 mg Log in to see 49 to 54 Days
$4,244.78
Details
Insect Cells Human rho-1D4 tag „Crystallography Grade“ protein due to multi-step, protein-specific purification process 0.5 mg Log in to see 49 to 54 Days
$6,041.49
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HOST_Wheat germ Human GST tag 10 μg Log in to see 9 Days
$405.71
Details

NAT8L Proteins by Origin and Source

Origin Expressed in Conjugate
Mouse (Murine)

Human ,
,

More Proteins for N-Acetyltransferase 8-Like (GCN5-Related, Putative) (NAT8L) Interaction Partners

Mouse (Murine) N-Acetyltransferase 8-Like (GCN5-Related, Putative) (NAT8L) interaction partners

  1. Results showed that both Shati+/- and Shati-/- mice demonstrated lower levels of NAA in the brain than Shati+/+ mice, suggesting that SHATI predominantly regulates NAA content in the brain

  2. Study shows that Shati/Nat8l in the neuron may induce axon outgrowth by ATP synthesis and not through mGluR3 (show GRM3 Proteins) signaling.

  3. Dysregulated expression of Nat8L is associated with deficits in oxidative phosphorylation in 5xFAD model of Alzheimer disease.

  4. results indicate that Shati/Nat8l in the Nucleus Accumbens, but not in the dorsal striatum, plays a suppressive role in the behavioral responses to METHamphetamine by controlling the dopaminergic system via activation of group II mGlutamate receptors

  5. The results of this study showed that Deletion of NAT8L increases dopamine D1 receptor on the cell surface in the nucleus accumbens, accelerating methamphetamine dependence in mice.

  6. Nat8l impacts on the brown adipogenic phenotype

  7. Deficiency of SHATI may affect behavior.

  8. shati expression affects emoutional behaivors in mice

  9. Nat8l is highly specific to aspartate. It is localized in mitochondria and microsomes, with a pattern comparable to the localization of NAA in brain. It is similar to NAG synthetase in catalytic mechanism without the involvement of cysteine.

  10. Results indicated that NAT8L, but not NAT14 (show NAT14 Proteins), catalysed the synthesis of NAA from L-aspartate and acetyl-CoA (show LPCAT1 Proteins).

Human N-Acetyltransferase 8-Like (GCN5-Related, Putative) (NAT8L) interaction partners

  1. Characterization and recombinant expression of the nat8l gene that codes for ANAT.

  2. NAA is produced specifically in NSCLC tumors through NAT8L overexpression, and its extracellular secretion can be detected in blood

  3. This study demonistrated that the human NAT8L gene related to reward dependence, a personality trait, and grey matter volume in the caudate nucleus in healthy subjects, suggesting that NAT8L might also affect human personality

  4. Nat8l impacts on the brown adipogenic phenotype

  5. Results indicated that NAT8L, but not NAT14 (show NAT14 Proteins), catalysed the synthesis of NAA from L-aspartate and acetyl-CoA (show LPCAT2 Proteins).

N-Acetyltransferase 8-Like (GCN5-Related, Putative) (NAT8L) Protein Profile

Protein Summary

This gene encodes a single-pass membrane protein, which contains a conserved sequence of the GCN5 or NAT superfamily of N-acetyltransferases and is a member of the N-acyltransferase (NAT) superfamily. This protein is a neuron-specific protein and is the N-acetylaspartate (NAA) biosynthetic enzyme, catalyzing the NAA synthesis from L-aspartate and acetyl-CoA. NAA is a major storage and transport form of acetyl coenzyme A specific to the nervous system. The gene mutation results in primary NAA deficiency (hypoacetylaspartia).

Gene names and symbols associated with NAT8L

  • N-acetyltransferase 8-like (NAT8L)
  • N-acetyltransferase 8-like (nat8l)
  • N-acetyltransferase 8-like protein (HPB8_615)
  • N-acetyltransferase 8-like (GCN5-related, putative) (nat8l)
  • N-acetyltransferase 8-like (Nat8l)
  • N-acetyltransferase 8-like (GCN5-related, putative) (NAT8L)
  • 1110038O08Rik protein
  • CML3 protein
  • NACED protein
  • NAT8-LIKE protein
  • RGD1305719 protein
  • sb:cb562 protein
  • Shati protein
  • zgc:162648 protein

Protein level used designations for NAT8L

N-acetyltransferase 8-like , N-acetylaspartate synthetase , N-acetyltransferase 8-like protein , NAA synthetase , camello 3 , protein Shati , camello-like protein 3

GENE ID SPECIES
422893 Gallus gallus
564754 Danio rerio
9349254 Helicobacter pylori B8
100124940 Xenopus (Silurana) tropicalis
269642 Mus musculus
289727 Rattus norvegicus
339983 Homo sapiens
612955 Canis lupus familiaris
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