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transcriptional repressor and an immediate-early gene [RGD, Feb 2006].. Additionally we are shipping NAB1 Proteins (6) and NAB1 Kits (3) and many more products for this protein.
Showing 10 out of 41 products:
Chlamydomonas reinhardtii (C. reinhardtii) Polyclonal NAB1 Primary Antibody for IP, WB - ABIN2487326
Blifernez, Wobbe, Niehaus, Kruse: Protein arginine methylation modulates light-harvesting antenna translation in Chlamydomonas reinhardtii. in The Plant journal : for cell and molecular biology 2010
Show all 6 references for ABIN2487326
Chicken Polyclonal NAB1 Primary Antibody for IHC, WB - ABIN2779429
Brandenberger, Wei, Zhang, Lei, Murage, Fisk, Li, Xu, Fang, Guegler, Rao, Mandalam, Lebkowski, Stanton: Transcriptome characterization elucidates signaling networks that control human ES cell growth and differentiation. in Nature biotechnology 2004
Show all 2 references for ABIN2779429
Human Polyclonal NAB1 Primary Antibody for ChIP, IP - ABIN4337717
Kearney, Delgado, Eshleman, Hill, OConnor, Lenz: Type I IFNs downregulate myeloid cell IFN-γ receptor by inducing recruitment of an early growth response 3/NGFI-A binding protein 1 complex that silences ifngr1 transcription. in Journal of immunology (Baltimore, Md. : 1950) 2013
Chicken Polyclonal NAB1 Primary Antibody for WB - ABIN2780729
Moser, Molitor, Kumsta, Tatschner, Riederer, Meyer: The glucocorticoid receptor gene exon 1-F promoter is not methylated at the NGFI-A binding site in human hippocampus. in The world journal of biological psychiatry : the official journal of the World Federation of Societies of Biological Psychiatry 2007
These data suggest that type I IFN stimulation induces a rapid recruitment of a repressive Egr3 (show EGR3 Antibodies)/Nab1 complex that silences transcription from the ifngr1 (show IFNGR1 Antibodies) promoter.
Nab1 is a potent inhibitor of cardiac growth in response to pathological stimuli in vivo.
Nab1 and Nab2 proteins are necessary for Schwann cells to exit the cell cycle, downregulate suppressed cAMP-inducible protein (SCIP) expression and upregulate expression of critical myelination genes.
Egr2 (show EGR2 Antibodies) directly represses genes during myelination in conjunction with NAB corepressors
Data show that the suppressive effect of valproic acid on chondrocytes is not due to reduced expression or recruitment of Egr-1 (show EGR1 Antibodies) to the mPGES-1 (show PTGES Antibodies) promoter and involves upregulation of NAB1.
Methylation pattern of the corresponding NGFI-A (show EGR1 Antibodies) binding site in the human glucocorticoid receptor (show NR3C1 Antibodies) exon 1-F specific promoter in post mortem hippocampal tissue.
NAB1 associates with RNA transcripts through a mechanism involving its CSD (show CSAD Antibodies) that is conserved with mechanisms of sequence-specific nucleic acid recognition employed by ancestrally related bacterial cold-shock proteins (CSPs).
All examined cysteine single and double mutants exhibited a reduced antenna at photosystem II caused by a perturbed NAB1 deactivation mechanism.
transcriptional repressor and an immediate-early gene
NGFI-A binding protein 1 (EGR1 binding protein 1)
, NGFI-A-binding protein 1-like
, nucleic acid binding protein
, EGR-1-binding protein 1
, NGFI-A-binding protein 1
, EGR1 binding protein 1
, transcriptional regulatory protein p54
, NGF1A-binding protein
, transcriptional repressor TA16