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NLRX1 encodes a member of the NLR family. Additionally we are shipping NLRX1 Antibodies (48) and and many more products for this protein.
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NLRX1, NLRP12 and NLRC3 (show NLRC3 ELISA Kits) negatively modulate the host immune response following virus exposure. (Review)
NLRX1 acts as a potential tumor suppressor by regulating the TNF-alpha (show TNF ELISA Kits) induced cell death and metabolism.
These data support a model in which CS-dependent NLRX1 inhibition facilitates MAVS (show MAVS ELISA Kits)/RHL (show DDX46 ELISA Kits) activation and subsequent inflammation, remodeling, protease, cell death, and inflammasome responses.
Together, these results suggest a novel mechanism for rhinovirus-induced epithelial barrier disruption involving NLRX-1 and mitochondrial reactive oxygen species generation.
NLRX1 and TUFM work in concert to reduce cytokine response and augment autophagy.
Results showed that missense mutations in transmembrane protein 2 p.Ser1254Asn, interferon alpha 2 p.Ala120Thr, its regulator NLR family member X1 p.Arg707Cys, and complement component 2 p.Glu318Asp were associated with chronic hepatitis B.
Post-transcriptional inhibition of luciferase reporter assays by the Nod-like receptor proteins NLRX1 and NLRC3 (show NLRC3 ELISA Kits).
NLRX1 attenuated IFN-I production & promoted autophagy during viral infection cells. NLRX1 interacts with TUFM.
NLRX1 interacts directly with RNA and suggestts a role for NLRX1 in recognition of intracellular viral RNA in antiviral immunity.
the presence of NLRX1 is required for optimal chlamydial growth and reactive oxygen species production
NLRX1 may relate to hair cell maturity, hearing formation and maintenance, and promote hair cell apoptosis through JNK (show MAPK8 ELISA Kits) pathway
The regulatory role of NLRX1 in mechanisms of host tolerance employed by macrophages to respond to Helicobacter pylori infection.NLRX1 modulates innate immune responses to Helicobacter pylori infection.
Nlrx1 may play an important role in neurodegenerative diseases, where necrosis is a prominent factor.
The unique capacity of NLRX1 to regulate the cellular sensitivity toward intrinsic versus extrinsic apoptotic signals suggests a critical role for this protein in numerous physiological processes and pathological conditions.
NLRX1 enhances macrophage antiviral immunity by interacting with influenza virus PB1-F2 protein, as well as preventing mitochondrial-induced apoptosis
Analysis of the macrophage and microglial populations indicates that NLRX1 reduces activation during both active and passive EAE models
does not participate in the negative regulation of MAVS (show MAVS ELISA Kits)-dependent antiviral responses
NLRX1 attenuated IFN-I production & promoted autophagy during viral infection. NLRX1 interacts with TUFM.
study identifies a previously unrecognized role for NLRX1 in the negative regulation of TLR-induced NF-kappaB (show NFKB1 ELISA Kits) activation by dynamically interacting with TRAF6 (show TRAF6 ELISA Kits) and the IKK (show CHUK ELISA Kits) complex
This gene encodes a member of the NLR family. Alternative splicing has been observed at this gene locus and two transcript variants, encoding distinct isoforms, have been identified.
NLR family member X1
, NLR family, X1
, NOD-like receptor X1
, caterpiller protein 11.3
, nucleotide-binding oligomerization domain protein 26
, nucleotide-binding oligomerization domain protein 5
, nucleotide-binding oligomerization domain protein 9
, nucleotide-binding oligomerization domain, leucine rich repeat containing X1
, nucleotide-binding domain and leucine-rich repeat containing