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The protein encoded by NPPA belongs to the natriuretic peptide family. Additionally we are shipping Natriuretic Peptide A Antibodies (224) and Natriuretic Peptide A Kits (118) and many more products for this protein.
Showing 9 out of 14 products:
Expression of atrial natriuretic peptide, C-type natriuretic peptide-1, and the guanylyl cyclase-linked (GC) natriuretic peptide receptor-A (show NPR1 Proteins) and -B-type receptors was determined by PCR from the mRNA of head kidney leucocytes
Atrial natriuretic peptide and vasopressin (show AVP Proteins)-presence in the ciliary body of eye in the pig (sus domesticus).
ANP and BNP gene expression differs considerably between cardiac chambers in the first 72 hours of life in healthy piglets, resembling the transition from fetal to neonate circulation. The cardiac gene expression does not explain plasma concentrations.
Up-regulation of ANP and a reduction in a ratio of sarcoplasmic reticulum calcium ATPase 2a and phospholamban (show PLN Proteins) in failing myocardium are observed in a porcine model of tachycardia.
Arial natriuretic factor (ANF (show HESX1 Proteins)) and N-terminal proANF may be better suited than brain natriuretic peptide (show BNP Proteins) as markers of cardiac preload during the development and treatment of acute heart failure.
Stimulation of prepubertal granulosa cells with FSH (show BRD2 Proteins) resulted in a moderate expression of ANP. When FSH (show BRD2 Proteins) treatment was followed by LH, the pronounced expression of ANP was observed in all cells.
ANP in plasma is increased in horses with heart disease associated with altered left-sided chamber dimensions and/or function.
Cardiac ANP was increased in horses with mitral regurgitation (MR) and cardiac troponin levels were low in healthy and MR affected horses.
the ANP T1766C gene polymorphism may be associated with the risk of essential hypertension, and the 1766C allele may be a protective factor against the disease
Corin (show CORIN Proteins) and atrial natriuretic peptide A were most abundant in the proximal convoluted tubules and the medullary connecting ducts.
Higher levels of PCT (show UROD Proteins), a marker of infection, and MRproANP, a marker for hemodynamic stress, were independently associated with ischemic stroke risk. PCT (show UROD Proteins) was specifically associated with small vessel and MRproANP with cardioembolic stroke risk.
The present results demonstrate that C2238/ANP-minor allele carrier status is an independent risk factor for acute coronary syndromes recurrence in an Italian cohort of ischemic heart disease patients over the long term, and they support the role of C2238/ANP-minor allele as a negative prognostic factor in coronary artery disease patients.
Both NT-proANP and NT-proBNP levels were higher in coronary artery disease patients experiencing MACEs following percutaneous coronary intervention in a non-primary setting. Notably, only NT-proANP levels significantly affected prognosis after PCI (show SERPINA5 Proteins).
As a risk factor, ANP gene polymorphisms may affect hypertension. Despite the small sample size in this study, it is the first research assessing the ANP gene polymorphisms in both Essential Hypertensive and Type 2 Diabetes Mellitus patients among Malaysian population.
The NPPA and NPPB (show BNP Proteins) gene polymorphisms are associated with blood pressure in newborns.
A panel of three biomarkers (MR-proANP, PCT (show UROD Proteins), and MR-proADM) in patients presenting to the emergency unit with acute dyspnea provides information about the probability of acute heart failure, nonsurvival, and pneumonia.
anti-inflammatory and immunomodulatory effect on IL-1beta (show IL1B Proteins) release and on NF-kB/NALP3 (show NLRP3 Proteins)/caspase-1 (show CASP1 Proteins) activation
Gene polymorphism of ANP (2238T>C) may be associated with developing preeclampsia during pregnancy.
ANP acts as an anti-permeability factor by inhibiting the signaling functions of vascular permeability factor (show VEGFA Proteins) and by preserving the endothelial cell tight junctions functional morphology
These studies support the therapeutic potential of dual inhibition of NEP (show MME Proteins) and ACE (show ACE Proteins) in the prevention of increased arterial ECP (show ECP Proteins) and atherogenesis which may be linked to the ANP/cGMP system.
Diabetes may modify the vascular actions of ANP in isolated rabbit carotid arteries and the mechanisms involved in these actions; ANP induces relaxation of precontracted carotid arteries, which is lower in diabetic than in control rabbits.
The study suggests that Beta1-adrenergic receptor activation decreases atrial natriuretic peptide release via cAMP- and Ca(2 (show CA2 Proteins)+)-dependent mechanisms.
we investigated pregnancies of ANP wild type, heterozygous, and knockout mice. Maternal blood pressure did not differ between genotypes, and fetal weight was unaffected. Placental weight was greater in ANP(-/-) versus ANP(+/+) mice. Therefore, in our hands, the ANP model does not express phenotypic features of preeclampsia with fetal growth restriction.
Data (including data from studies in knockout mice) suggest expression of ANP/BNP/GC-A (show GUCA1A Proteins) (atrial natriuretic peptide, brain natriuretic peptide (show BNP Proteins), and guanylyl cyclase-A (show NPR1 Proteins)) is important in embryonic neovascularization and organogenesis.
Goalpha plays a role in ANF sorting during intracellular vectorial transport and with the presence of a mechanism that preserves the molar relationship between cellular ANF and BNP.
ANP prevents cancer metastasis by inhibiting the adhesion of tumor cells to inflamed endothelial cells
Expression of NPPA splice variants is differentially regulated in the developing mouse heart.
NPPA acts on the NPR1 receptor, is expressed in mouse dorsal root and trigeminal ganglia, and the spinal cord.
Natriuretic peptides buffer renin (show REN Proteins)-dependent hypertension.
Data indicate that the identified stress-responsive enhancer of Nppa and Nppb (show BNP Proteins), the most common markers of heart failure, is a 650-bp fragment within 50 kb of the Nppa and Nppb (show BNP Proteins) loci.
Absence of maternal atrial natriuretic peptide leads to gestational hypertension and adaptive changes fetal cardiac growth and blood pressure regulation.
This study investigated a novel mechanism of vascular barrier protection by ANP via modulation of GEF-H1 (show ARHGEF2 Proteins) function.
The protein encoded by this gene belongs to the natriuretic peptide family. Natriuretic peptides are implicated in the control of extracellular fluid volume and electrolyte homeostasis. This protein is synthesized as a large precursor (containing a signal peptide), which is processed to release a peptide from the N-terminus with similarity to vasoactive peptide, cardiodilatin, and another peptide from the C-terminus with natriuretic-diuretic activity. Mutations in this gene have been associated with atrial fibrillation familial type 6.
atrial natriuretic peptide
, atrial natriuretic factor
, natriuretic peptides A
, natriuretic peptide precursor A
, prohormone for atrial natriuretic polypeptide (ANP)
, Natriuretic peptide precursor A (pronatriodilatin, also Anf, Pnd)
, natriuretic peptide precursor type A
, Atrial natriuretic factor
, Atrial natriuretic peptide
, natriuretic peptide type A
, Natriuretic peptides A