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NPPB is a member of the natriuretic peptide family and encodes a secreted protein which functions as a cardiac hormone. Additionally we are shipping Natriuretic Peptides B Antibodies (209) and Natriuretic Peptides B Proteins (20) and many more products for this protein.
Showing 10 out of 26 products:
Human NPPB ELISA Kit for Competition ELISA - ABIN1979095
Martín, Cordova, San Román, Gutierrez, Cachofeiro, Nieto: Oleanolic acid modulates the immune-inflammatory response in mice with experimental autoimmune myocarditis and protects from cardiac injury. Therapeutic implications for the human disease. in Journal of molecular and cellular cardiology 2014
Show all 30 references for ABIN1979095
Human NPPB ELISA Kit for Sandwich ELISA - ABIN415051
Ge, Mo, Januzzi, Jin, Yang, Han, Wood, Levine: B-type natriuretic peptide, vascular endothelial growth factor, endothelin-1, and nitric oxide synthase in chronic mountain sickness. in American journal of physiology. Heart and circulatory physiology 2011
Show all 6 references for ABIN415051
Rat (Rattus) NPPB ELISA Kit for Competition ELISA - ABIN416384
Gu, Lu, Xie, Bai, Xu: Renalase deficiency in heart failure model of rats--a potential mechanism underlying circulating norepinephrine accumulation. in PLoS ONE 2011
Show all 5 references for ABIN416384
Mouse (Murine) NPPB ELISA Kit for Competition ELISA - ABIN415632
Prévilon, Pezet, Semprez, Mercadier, Rouet-Benzineb: FKBP12.6 mice display temporal gender differences in cardiac Ca(2+)-signalling phenotype upon chronic pressure overload. in Canadian journal of physiology and pharmacology 2011
CNP and BNP serve as oocyte maturation inhibitor to inhibit oocyte maturation in mammals
ANP and BNP gene expression differs considerably between cardiac chambers in the first 72 hours of life in healthy piglets, resembling the transition from fetal to neonate circulation. The cardiac gene expression does not explain plasma concentrations.
Up-regulation of BNP and a reduction in a ratio of sarcoplasmic reticulum calcium ATPase (show CA-P60A ELISA Kits) 2a in failing myocardium are observed in a porcine model of tachycardia.
Data show that all five molecules, BNP, ICAM-1 (show ICAM1 ELISA Kits), TNF-alpha (show TNF ELISA Kits), VCAM-1 (show VCAM1 ELISA Kits) and IL-6 (show IL6 ELISA Kits), quickly and reliably signaled adverse interactions.
identification and characterization of a novel alternatively spliced variant of porcine and human NPPB (show BNP ELISA Kits) resulting from exon 2 skipping
Arial natriuretic factor (ANF (show HESX1 ELISA Kits)) and N-terminal proANF may be better suited than brain natriuretic peptide (show BNP ELISA Kits) as markers of cardiac preload during the development and treatment of acute heart failure.
Regional differences in BNP and CNP (show NPPC ELISA Kits) expression were observed in myocardium with sustained left ventricular dyssynchronous contraction.
N-terminal pro-brain natriuretic peptide (show BNP ELISA Kits) may be an important marker for cardiac dysfunction in patients with chronic liver failure in accordance with Child-Pugh stage.
The combination of a clinical model with copeptin (show AVP ELISA Kits) and NTproBNP, which are available in the Emergency Department, is able to prognose early mortality in patients with an episode of Acute Heart Failure.
Blood concentration of BNP is a useful biomarker in the diagnosis of acute heart failure.
Both NT-proANP and NT-proBNP levels were higher in coronary artery disease patients experiencing MACEs following percutaneous coronary intervention in a non-primary setting. Notably, only NT-proANP levels significantly affected prognosis after PCI (show SERPINA5 ELISA Kits).
BNP was statistically significantly higher in patients with nasal polyps than in controls.
Among persons occupationally exposed to lead, certain relationships were demonstrated between rs632793, rs198388 and rs198389 polymorphisms of NPPB (show BNP ELISA Kits) gene and principal toxicological parameters characterizing exposure to lead
In patients with NSTE-ACS (show PLA2G15 ELISA Kits), NT-proBNP and the GRACE risk score (but not the TIMI risk score) both have good and comparable predictive value for 30-day mortality.
The NPPB (show BNP ELISA Kits) gene polymorphism is associated with modulation of left ventricular mass in newborns. The NPPA (show NPPA ELISA Kits) and NPPB (show BNP ELISA Kits) gene polymorphisms are associated with blood pressure in newborns.
High plasma BNP levels and left ventricular wall motion abnormalities in the segments perfused with left anterior descending coronary artery or right coronary artery show a high risk for cardioembolic stroke in patients with old myocardial infarction.
we have validated a novel gene, Myh14, as a negative regulator of ISO-induced left ventricular mass hypertrophy in an in vivo mouse model and demonstrated the up-regulation of immediate early (show JUN ELISA Kits) gene Myc (show MYC ELISA Kits), fetal gene Nppb (show BNP ELISA Kits), and fibrosis gene Lgals3 (show LGALS3 ELISA Kits) in ISO-treated Myh14 deficient hearts compared to controls.
P2X3 (show P2RX3 ELISA Kits) receptors on mouse trigeminal ganglion neurons are subjected to contrasting modulation by inhibitory brain natriuretic peptide (show BNP ELISA Kits) and facilitatory calcitonin gene-related peptide (show CALCA ELISA Kits) that both operate via complex intracellular signaling.
mouse trigeminal neurons endogenous BNP acts on NPR-A (show NPR1 ELISA Kits) receptors to determine constitutive depression of P2X3 (show P2RX3 ELISA Kits) receptor function. Tonic inhibition of P2X3 (show P2RX3 ELISA Kits) receptor activity by BNP/NPR-A (show NPR1 ELISA Kits)/PKG (show PRKG1 ELISA Kits) pathways occurs via two distinct mechanisms: P2X3 (show P2RX3 ELISA Kits) serine phosphorylation and receptor redistribution to non-raft membrane compartments
The findings demonstrated a substantial role of mitochondrial dysfunction in mediating the downregulation of NKCC2 and ENaCalpha in obstructive kidney disease, possibly via iNOS-derived nitric oxide and BNP.
Data (including data from studies in knockout mice) suggest expression of ANP (show NPPA ELISA Kits)/BNP/GC-A (show GUCA1A ELISA Kits) (atrial natriuretic peptide (show NPPA ELISA Kits), brain natriuretic peptide (show BNP ELISA Kits), and guanylyl cyclase-A (show NPR1 ELISA Kits)) is important in embryonic neovascularization and organogenesis.
Goalpha plays a role in ANF sorting during intracellular vectorial transport and with the presence of a mechanism that preserves the molar relationship between cellular ANF and BNP.
The RNA-Seq demonstrates that NPPB (show BNP ELISA Kits) which well activates natriuretic peptide receptor 1 (NPR1 (show NPR1 ELISA Kits)), is well expressed in mouse dorsal root ganglian.
a slow modulatory action by BNP on TRPV1 (show TRPV1 ELISA Kits) and P2X3 (show P2RX3 ELISA Kits) receptors outlining the role of this peptide as a negative regulator of trigeminal sensory neuron excitability to nociceptive stimuli
Natriuretic peptides buffer renin (show REN ELISA Kits)-dependent hypertension.
BNP-NPRA (show NPR1 ELISA Kits) signaling is involved in both itch and pain and does not function upstream of the GRP (show UCMA ELISA Kits)-GRPR (show GRPR ELISA Kits) dedicated neuronal pathway.
This gene is a member of the natriuretic peptide family and encodes a secreted protein which functions as a cardiac hormone. The protein undergoes two cleavage events, one within the cell and a second after secretion into the blood. The protein's biological actions include natriuresis, diuresis, vasorelaxation, inhibition of renin and aldosterone secretion, and a key role in cardiovascular homeostasis. A high concentration of this protein in the bloodstream is indicative of heart failure. Mutations in this gene have been associated with postmenopausal osteoporosis.
gamma-brain natriuretic peptide
, natriuretic peptides B
, brain type natriuretic peptide
, natriuretic peptide precursor B
, natriuretic protein
, brain natriuretic peptide
, Brain natriuretic factor
, natriuretic peptide precursor type B
, Gamma-brain natriuretic peptide