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NOS1AP encodes a cytosolic protein that binds to the signaling molecule, neuronal nitric oxide synthase (nNOS). Additionally we are shipping NOS1AP Antibodies (73) and many more products for this protein.
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Results from genome-wide DNA methylation, functional network analysis and pyrosequencing, show selective CpG sites (NOS1AP, BID, and GABRB1) differentially methylated in smokers and chronic obstructive pulmonary disease patients compared to nonsmokers.
Data indicate that overexpression of nitric oxide synthase 1 adaptor protein short form (CAPON-S) led to the inactivation of the proto-oncogene (show RAB1A Proteins) protein Akt (Akt (show AKT1 Proteins)) signaling pathway.
SNPs in the NOS1AP gene influence QTc interval duration but we have not demonstrated a direct association with the risk of sudden cardiac death
Results suggest that NOS1AP variants are associated with various forms of depression in schizophrenia and are more prevalent in males.
The study suggested that rs3751284 and rs348624 in the NOS1AP gene might be susceptibility loci for sudden unexplained death during daily activities.
report of the association of common NOS1AP polymorphisms with sudden unexplained nocturnal death syndrome in the southern Chinese Han population. These findings suggest that the A allele of rs12567209 and haplotype GCTA may serve as a protective modifier.
The A allele of rs12567209 in NOS1AP may serve as an independent predictor of all-cause death and SCD (show SCD Proteins) in patients with CHF.
This study suggested that NOS1 (show NOS1 Proteins) and NOS1AP were associated with schizophrenia.
We further localized NOS1AP to cardiomyocyte intercalated discs (IDs (show IDS Proteins)) and demonstrate that overexpression of NOS1AP in cardiomyocytes leads to altered cellular electrophysiology.
This is the first study reporting that a variant of the NOS1AP gene is associated with PTSD. Our data also suggest that a genetic variant in NOS1AP may increase the susceptibility to severe depression in patients with PTSD and increased risk for suicide.
Genetic variations at Nos1ap were associated with Oxidative Stress Induced (show SQSTM1 Proteins) Ventricular Arrhythmia and Heart Failure, which may cause Sudden Cardiac Death.
Capon expression is increased in skeletal muscle by position, repair, NOS activity, and dystrophy.
long-term expression of cocaine-induced behavioral sensitization in females (adolescent and adult) is nNOS (show NOS1 Proteins)-independent, unlike previous finding in adult males.
This gene encodes a cytosolic protein that binds to the signaling molecule, neuronal nitric oxide synthase (nNOS). This protein has a C-terminal PDZ-binding domain that mediates interactions with nNOS and an N-terminal phosphotyrosine binding (PTB) domain that binds to the small monomeric G protein, Dexras1. Studies of the related mouse and rat proteins have shown that this protein functions as an adapter protein linking nNOS to specific targets, such as Dexras1 and the synapsins. Alternative splicing results in multiple transcript variants encoding different isoforms.
nitric oxide synthase 1 (neuronal) adaptor protein
, carboxyl-terminal PDZ ligand of neuronal nitric oxide synthase protein
, carboxyl-terminal PDZ ligand of neuronal nitric oxide synthase protein-like
, C-terminal PDZ domain ligand of neuronal nitric oxide synthase (CAPON)
, C-terminal PDZ ligand of neuronal nitric oxide synthase protein
, ligand of neuronal nitric oxide synthase with carboxyl-terminal PDZ domain
, nitric oxide synthase 1 adaptor protein
, C-terminal PDZ domain ligand of neuronal nitric oxide synthase