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NFIA encodes a member of the NF1 (nuclear factor 1) family of transcription factors. Additionally we are shipping NFIA Antibodies (60) and NFIA Proteins (7) and many more products for this protein.
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NFI-A is involved in the miR (show MLXIP ELISA Kits)-21-induced expression of IL-10 (show IL10 ELISA Kits) in B cells in nasopharyngeal carcinoma; Il-10 (show IL10 ELISA Kits) is capable of suppressing CD8 (show CD8A ELISA Kits)+ T-cell activities.
microRNA-136 targeted and degraded NFIA, which induced the release of microRNA-223, promoting CD11b (show ITGAM ELISA Kits) expression. Direct base pairing occurs between miR (show MLXIP ELISA Kits)-136 and the 3' UTR (show UTS2R ELISA Kits) of NFIA mRNA.
this family also carried a microdeletion affecting solely the NFIA gene, this study substantiates the importance of this gene in craniofacial development.
TGF-beta (show TGFB1 ELISA Kits)-mediated suppression of ANT2 (show SLC25A5 ELISA Kits) through NF1 (show NF1 ELISA Kits)/Smad4 (show SMAD4 ELISA Kits) complexes contributes to oxidative stress and DNA damage during induction of cellular senescence.
RP5-833A20.1/miR (show MLXIP ELISA Kits)-382-5p/NFIA pathway was essential to the regulation of cholesterol homeostasis and inflammatory reactions.
This report presents the first case of an intragenic deletion within the NFIA gene that is still consistent with classic clinical phenotypes present in previously reported cases of chromosome 1p31.3 related deletion.
High nuclear factor IA expression is associated with glioblastomas.
These studies represent the first characterization of miR (show MLXIP ELISA Kits)-223/NFIA axis function in glioma
These data suggest that genetic variants in the NF1A gene region may predispose to seasonal patterned of mania in bipolar disorder.
NFIA expression in white matter lesions of human newborns with neonatal HIE, as well active MS lesions in adults, revealed that it is similarly expressed in oligodendrocyte progenitors and not oligodendrocytes.
Astrocyte induction by Zbtb20 is suppressed by knockdown of NFIA.
NFI-A disrupts myeloid cell differentiation and maturation in septic mice
NFIA and NFIB (show NFIB ELISA Kits) are endogenously expressed in gradients with high caudo-medial to low rostro-lateral expression ... this expression pattern deviates from that of PAX6 (show PAX6 ELISA Kits), suggesting that PAX6 (show PAX6 ELISA Kits) does not drive Nfi (show NFIC ELISA Kits) expression
Nfia cooperates with sonic hedgehog (show SHH ELISA Kits) signaling to initiate medulloblastoma.
Our data thus suggest that NFI (show NFIC ELISA Kits) nucleosomal interaction may contribute to the partitioning of distinct chromatin domains and to epigenetic gene expression regulation
As maturing cerebellar granule neurons become more hyperpolarized, NFATc4 (show NFATC4 ELISA Kits) binding declines leading to onset of NFI (show NFIC ELISA Kits) temporal binding and the NFI (show NFIC ELISA Kits) switch program
NFIA and NFIB (show NFIB ELISA Kits) are expressed in both the developing and the adult olfactory bulb indicative of a regulatory role for these transcription factors in the development of this facet of the olfactory system.
NFIA is expressed in oligodendrocyte progenitors, but not differentiated oligodendrocytes during mouse embryonic development.
NFIA is a transcription factor that plays a crucial role in the onset of gliogenesis; we found that its induction is regulated by the transcription factor Sox9 (show SOX9 ELISA Kits) and that this relationship mediates the initiation of gliogenesis
This gene encodes a member of the NF1 (nuclear factor 1) family of transcription factors. Multiple transcript variants encoding different isoforms have been found for this gene.
nuclear factor I/A
, CCAAT-box-binding transcription factor
, TGGCA-binding protein
, nuclear factor 1 A-type
, nuclear factor 1/A
, nuclear factor I-A